B P50 Κ Preferentially Induces Nf- Attenuated by an Autocrine Factor That Transcription in Macrophages Is Tumor Necrosis Factor Alpha

No abstract is available for this paper.

• Publication year

  1998

• Venue

  Unknown venue

• Publication date

  Unknown publication date

• Fields of study

  Not labeled

• Identifiers

  No identifiers available.

• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar

• No claims are published for this paper.

• No concepts are published for this paper.

• Lipopolysaccharide Induction of the Tumor Necrosis Factor-α Promoter in Human Monocytic Cells

  1997cited by this paper
• Regulation of NFKB1 proteins by the candidate oncoprotein BCL‐3: generation of NF‐κB homodimers from the cytoplasmic pool of p50–p105 and nuclear translocation

  1997cited by this paper
• Endotoxin signal transduction in macrophages

  1996cited by this paper
• Conserved kappa B element located downstream of the tumor necrosis factor alpha gene: distinct NF-kappa B binding pattern and enhancer activity in LPS activated murine macrophages.

  1995cited by this paper
• Molecular mechanism in tolerance to lipopolysaccharide.

  1995cited by this paper
• Transcriptional Regulation of the Intercellular Adhesion Molecule-1 Gene by Inflammatory Cytokines in Human Endothelial Cells

  1995cited by this paper
• Interleukin-10 is upregulated in LPS tolerance.

  1995cited by this paper
• Control of tumor necrosis factor gene expression.

  1994cited by this paper
• Kappa B binding activity in a murine macrophage-like cell line. Sequence-specific differences in kappa B binding and transcriptional activation functions.

  1994cited by this paper
• Search for sepsis drugs goes on despite past failures.

  1994cited by this paper
• Lipopolysaccharide induction of tissue factor gene expression in monocytic cells is mediated by binding of c-Rel/p65 heterodimers to a kappa B-like site

  1994cited by this paper
• Lymphokines in autoimmunity--a critical review.

  1994cited by this paper
• Tumor necrosis factor: a pleiotropic cytokine and therapeutic target.

  1994cited by this paper
• Interleukin-10 inhibits interleukin-8 production in human neutrophils.

  1994cited by this paper
• An HMG-like protein that can switch a transcriptional activator to a repressor

  1994cited by this paper
• IL-10 inhibits transcription of cytokine genes in human peripheral blood mononuclear cells.

  1994cited by this paper
• Selective refractoriness of macrophages to endotoxin‐induced production of tumor necrosis factor, elicited by an autocrine mechanism

  1993cited by this paper
• NF-idBSubunit-Specific Regulation ofthe Interleukin-8 Promoter

  1993cited by this paper
• The precursor of NF-κB p50 has IκB-like functions

  1992cited by this paper
• The p65 subunit is responsible for the strong transcription activating potential of NF‐kappa B.

  1991cited by this paper
• IL-10 inhibits cytokine production by activated macrophages.

  1991cited by this paper
• The DNA binding subunit of NF-κB is identical to factor KBF1 and homologous to the rel oncogene product

  1990cited by this paper
• Regulation of monokine gene expression: prostaglandin E2 suppresses tumor necrosis factor but not interleukin-1 alpha or beta-mRNA and cell-associated bioactivity.

  1989cited by this paper
• Dynamics of dibutyryl cyclic AMP- and prostaglandin E2-mediated suppression of lipopolysaccharide-induced tumor necrosis factor alpha gene expression

  1989cited by this paper
• Potential antiinflammatory effects of interleukin 4: suppression of human monocyte tumor necrosis factor alpha, interleukin 1, and prostaglandin E2.

  1989cited by this paper
• Release of tumor necrosis factor-alpha from macrophages. Enhancement and suppression are dose-dependently regulated by prostaglandin E2 and cyclic nucleotides.

  1988cited by this paper
• Luciferase reporter gene vectors for analysis of promoters and enhancers.

  1988cited by this paper
• Multiple nuclear factors interact with the immunoglobulin enhancer sequences.

  1986cited by this paper
• A gel electrophoresis method for quantifying the binding of proteins to specific DNA regions: application to components of the Escherichia coli lactose operon regulatory system

  1981cited by this paper
• Equilibria and kinetics of lac repressor-operator interactions by polyacrylamide gel electrophoresis.

  1981cited by this paper
• Antibody-dependent cytolysis of chicken erythrocytes by an in vitro-established line of mouse peritoneal macrophages.

  1975cited by this paper
• Cleavage of Structural Proteins during the Assembly of the Head of Bacteriophage T4

  1970cited by this paper
• In vivo control of NF-xB activation by IxBa

  year unknowncited by this paper

• The effects of silver nanoparticles biosynthesized using fig and olive extracts on cutaneous leishmaniasis-induced inflammation in female balb/c mice

  2020cites this paper
• Piperidylmethyloxychalcone improves immune-mediated acute liver failure via inhibiting TAK1 activity

  2017cites this paper
• RANKL cytokine enhances TNF-induced osteoclastogenesis independently of TNF receptor associated factor (TRAF) 6 by degrading TRAF3 in osteoclast precursors

  2017cites this paper
• Inhibition of Ubc13-mediated Ubiquitination by GPS2 Regulates Multiple Stages of B Cell Development*

  2016cites this paper
• Transcriptional Repression of the Transforming Growth Factor β (TGF-β) Pseudoreceptor BMP and Activin Membrane-bound Inhibitor (BAMBI) by Nuclear Factor κB (NF-κB) p50 Enhances TGF-β Signaling in Hepatic Stellate Cells*

  2014cites this paper
• TNF-α from inflammatory dendritic cells (DCs) regulates lung IL-17A/IL-5 levels and neutrophilia versus eosinophilia during persistent fungal infection

  2011cites this paper
• Mycobacterium tuberculosis LprG (Rv1411c): A Novel TLR-2 Ligand That Inhibits Human Macrophage Class II MHC Antigen Processing1

  2004cites this paper
• Macrophage Class II MHC Antigen A Novel TLR-2 Ligand That Inhibits Human ) : Rv 1411 c LprG ( Mycobacterium tuberculosis

  2004cites this paper
• Immune protection against septic peritonitis in endotoxin‐primed mice is related to reduced neutrophil apoptosis

  2001cites this paper