Lack of REDD1 reduces whole body glucose and insulin tolerance, and impairs skeletal muscle insulin signaling.

No abstract is available for this paper.

• Publication year

  2014

• Venue

  Biochemical and Biophysical Research Communications - BBRC

• Publication date

  2014-10-31

• Fields of study

  Biology, Medicine

• Identifiers
  DOI 10.1016/j.bbrc.2014.10.032PMID 25445588
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

• Insulin receptor signaling in normal and insulin-resistant states.

  2014cited by this paper
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  2014influential reference
• Regulated in DNA damage and development 1 (REDD1) promotes cell survival during serum deprivation by sustaining repression of signaling through the mechanistic target of rapamycin in complex 1 (mTORC1).

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• mTOR signaling in growth control and disease.

  2012influential reference
• Relative muscle mass is inversely associated with insulin resistance and prediabetes. Findings from the third National Health and Nutrition Examination Survey.

  2011cited by this paper
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  2010cited by this paper
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  2009cited by this paper
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  2007cited by this paper
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  2006cited by this paper
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  2005cited by this paper
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  2002cited by this paper
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  2002cited by this paper
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  1998cited by this paper
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  2020cites this paper
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  2020cites this paper
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  2019cites this paper
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  2019influential citation
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  2018cites this paper
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  2017cites this paper
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  2016cites this paper
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  2016influential citation
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  2016cites this paper
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  2015cites this paper
• Title: Aberrant REDD1-mTORC1 responses to insulin in skeletal muscle from type 2 diabetics

  2015cites this paper
• Aberrant REDD1-mTORC1 responses to insulin in skeletal muscle from Type 2 diabetics.

  2015cites this paper