Smad3 and Smad3 Phosphoisoforms Are Prognostic Markers of Gastric Carcinoma

No abstract is available for this paper.

• Publication year

  2013

• Venue

  Digestive Diseases and Sciences

• Publication date

  2013-04-01

• Fields of study

  Medicine

• Identifiers
  DOI 10.1007/s10620-012-2470-3PMID 23179147
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

• Decreased Levels of Active SMAD2 Correlate with Poor Prognosis in Gastric Cancer

  2012cited by this paper
• Smad3 and its phosphoisoforms are prognostic predictors of hepatocellular carcinoma after curative hepatectomy.

  2012cited by this paper
• Phosphorylated Smad2 in Advanced Stage Gastric Carcinoma

  2010cited by this paper
• Inhibition of c‐Jun NH2‐terminal kinase switches Smad3 signaling from oncogenesis to tumor‐ suppression in rat hepatocellular carcinoma

  2009cited by this paper
• Smad2 and Smad3 phosphorylated at both linker and COOH-terminal regions transmit malignant TGF-beta signal in later stages of human colorectal cancer.

  2009cited by this paper
• Transforming Growth Factor-β-inducible Phosphorylation of Smad3*

  2009cited by this paper
• Hepatitis B virus X protein shifts human hepatic transforming growth factor (TGF)‐β signaling from tumor suppression to oncogenesis in early chronic hepatitis B

  2009cited by this paper
• A Novel Transforming Growth Factor β Receptor Kinase Inhibitor, A-77, Prevents the Peritoneal Dissemination of Scirrhous Gastric Carcinoma

  2008cited by this paper
• How the Smads regulate transcription.

  2008cited by this paper
• TGFbeta in Cancer.

  2008cited by this paper
• Sonic hedgehog signaling promotes motility and invasiveness of gastric cancer cells through TGF-beta-mediated activation of the ALK5-Smad 3 pathway.

  2007cited by this paper
• Reversible Smad-dependent signaling between tumor suppression and oncogenesis.

  2007cited by this paper
• Chronic inflammation associated with hepatitis C virus infection perturbs hepatic transforming growth factor β signaling, promoting cirrhosis and hepatocellular carcinoma

  2007cited by this paper
• Smad3 phosphoisoform-mediated signaling during sporadic human colorectal carcinogenesis.

  2006cited by this paper
• Causes of cancer in the world: comparative risk assessment of nine behavioural and environmental risk factors.

  2005cited by this paper
• Acceleration of Smad2 and Smad3 phosphorylation via c-Jun NH(2)-terminal kinase during human colorectal carcinogenesis.

  2005cited by this paper
• Prognostic significance of the expression of Smad4 and Smad7 in human gastric carcinomas.

  2004cited by this paper
• Loss of the Smad3 expression increases susceptibility to tumorigenicity in human gastric cancer

  2004cited by this paper
• Smad-dependent and Smad-independent pathways in TGF-beta family signalling.

  2003cited by this paper
• Smad-dependent and Smad-independent pathways in TGF-β family signalling

  2003cited by this paper
• AJCC Cancer Staging Manual

  2002cited by this paper
• Frequency of Smad gene mutations in human cancers.

  1997cited by this paper
• Comparison of the prognostic significance between the number of metastatic lymph nodes and nodal stage based on their location in patients with gastric cancer.

  1993cited by this paper
• Adenocarcinoma of the stomach with invasion limited to the muscularis propria.

  1991cited by this paper

• Machine learning-based bulk RNA analysis reveals a prognostic signature of 13 cell death patterns and potential therapeutic target of SMAD3 in acute myeloid leukemia

  2025cites this paper
• Drug Repurposing of Selected Antibiotics: An Emerging Approach in Cancer Drug Discovery

  2024cites this paper
• Prognostic Significance of TLR2, SMAD3 and Localization-dependent SATB1 in Stage I and II Non–Small-Cell Lung Cancer Patients

  2021cites this paper
• Knockdown of SMAD3 inhibits the growth and enhances the radiosensitivity of lung adenocarcinoma via p21 in vitro and in vivo

  2020cites this paper
• SUV39H1-Mediated DNMT1 is Participated in the Epigenetic Regulation of Smad3 in Cervical Cancer.

  2020cites this paper
• Crosstalk of protein kinase C ε with Smad2/3 promotes tumor cell proliferation in prostate cancer cells by enhancing aerobic glycolysis

  2018cites this paper
• The Effects of Smad3 on Adrenocorticotropic Hormone-Secreting Pituitary Adenoma Development, Cell Proliferation, Apoptosis, and Hormone Secretion.

  2018cites this paper
• A small-molecule inhibitor of SMAD3 attenuates resistance to anti-HER2 drugs in HER2-positive breast cancer cells

  2017cites this paper
• Negative immune factors might predominate local tumor immune status and promote carcinogenesis in cervical carcinoma

  2017cites this paper
• Bleomycin Suppresses the Proliferation and the Mobility of Human Gastric Cancer Cells Through the Smad Signaling Pathway

  2016cites this paper
• Deregulated E2F5/p38/SMAD3 Circuitry Reinforces the Pro‐Tumorigenic Switch of TGFβ Signaling in Prostate Cancer

  2016cites this paper
• The prognostic significance of Smad3, Smad4, Smad3 phosphoisoform expression in esophageal squamous cell carcinoma

  2014cites this paper