Effect of Active Fraction of Cerebral on Expression of Caspase-3 and β-Amyloid Precursor Protein during Therapy of Hemorrhagic Stroke in the Acute and Delayed Periods

No abstract is available for this paper.

• Publication year

  2005

• Venue

  Bulletin of experimental biology and medicine

• Publication date

  2005-02-01

• Fields of study

  Biology, Medicine

• Identifiers
  DOI 10.1007/s10517-005-0249-7PMID 16027808
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

• Hemoglobin-Induced Cytotoxicity in Rat Cerebral Cortical Neurons: Caspase Activation and Oxidative Stress

  2002cited by this paper
• Molecular Cloning: A Laboratory Manual

  2001cited by this paper
• Alterations of Extracellular Calcium Elicit Selective Modes of Cell Death and Protease Activation in SH‐SY5Y Human Neuroblastoma Cells

  1999cited by this paper
• Increased Production of Amyloid Precursor Protein Provides a Substrate for Caspase-3 in Dying Motoneurons

  1998cited by this paper
• Caspase-3 Is Required for DNA Fragmentation and Morphological Changes Associated with Apoptosis*

  1998cited by this paper
• Increased expression of amyloid precursor protein and amyloid precursor-like protein 2 during trophic factor withdrawal-induced death of neuronal PC12 cells.

  1998cited by this paper
• The β-Amyloid Precursor Protein of Alzheimer’s Disease Enhances Neuron Viability and Modulates Neuronal Polarity

  1997cited by this paper
• Ca2+ channel blockers verapamil and nifedipine inhibit apoptosis induced by 25-hydroxycholesterol in human aortic smooth muscle cells.

  1997cited by this paper
• The regulation of amyloid β protein precursor secretion and its modulatory role in cell adhesion

  1989cited by this paper
• A rapid method for detecting specific RNA transcripts by hybridization to DNA probes in solution.

  1983cited by this paper

• No citing papers are available for this paper.