Blood Glucose Concentration Does Not Affect Outcome in Brain Trauma: A 31P MRS Study

No abstract is available for this paper.

• Publication year

  1997

• Venue

  Journal of Cerebral Blood Flow and Metabolism

• Publication date

  1997-01-01

• Fields of study

  Medicine

• Identifiers
  DOI 10.1097/00004647-199701000-00007PMID 8978386
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

• Estrogen improves biochemical and neurologic outcome following traumatic brain injury in male rats, but not in females.

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• Where are we in the modelling of traumatic brain injury? Models complicated by secondary brain insults

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• Glucose administration after traumatic brain injury improves cerebral metabolism and reduces secondary neuronal injury.

  2013influential citation
• Alcohol Intoxication May Exacerbate the Effects of Blunt Cranial Trauma Through Changes in Brain Free Magnesium Levels

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• Exploring the Impact of Decreased AMPK Pathway Activity on Brain Injury Outcome

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• Effects of l-NAME and 7-NI on NOS Catalytic Activity and Behavioral Outcome After Traumatic Brain Injury in the Rat

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• Poor outcome after hypoxia-ischemia in newborns is associated with physiological abnormalities during early recovery. Possible relevance to secondary brain injury after head trauma in infants.

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• Hyperglycemia Increases Neurological Damage and Behavioral Deficits From Post-Traumatic Secondary Ischeie Insults

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• pH dependency of mu-calpain and m-calpain activity assayed by casein zymography following traumatic brain injury in the rat.

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• Insulin-like growth factor-1 (IGF-1) improves both neurological motor and cognitive outcome following experimental brain injury.

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• Current awareness in NMR in biomedicine

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