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Hypomagnesaemia and new onset Diabetes Mellitus after Kidney transplantation

Bernadett Borda,A. Nemes,C. Lengyel,Csilla Keresztes,A. Ottlakan,F. Rárosi,G. Lázár

Published 2018 in Unknown venue

ABSTRACT

Background: The leading causes of death in patients who died with a functioning allograft are cardiovascular diseases, which account for almost 40 percent of all deaths in this population. Patients and methods: We studied prospectively basic data, hypomagnesaemia HbA1C, renal functions (serum creatinine, eGFR, and urea), and urinalysis. Then oral glucose tolerance test was performed as well, and HOMA index was calculated. Results: The ratio of patients changed at Minute 120 of the test: there were 22 (37%) patients in the normal group, 26 (43%) patients in the IFG/IGT group, and 12 (20%) patients in the NODAT group. Regarding basic data, body mass index (p=0.854) did not influence the development of diabetes mellitus significantly. The results of laboratory tests showed significant difference in serum Mg level. HbA1C (p=0.009) and HOMA index (p=0.09) were significantly different. Renal function parameters, such as serum creatinine (p=0.001) and eGFR (p=0.0001), were also significantly different. Conclusion: In our clinical study, given the frequency of NODAT and its relationship with cardiovascular risk, correcting hypomagnesemia soon after transplantation could translate into a significant decrease in vascular disease, which today is the primary cause of death in kidney transplant recipients. *Correspondence to: Bernadett Borda MD, PhD, Szeged, 6728 Semmelweis u. 8. Hungary, Tel: +36/62-54-54-62; E-mail: borda.bernadett@med.u-szeged.hu

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