Sensitivity of spinal reflexes to TRH and 5-HT in 5,6-dihydroxytryptamine-treated rats.

No abstract is available for this paper.

• Publication year

  1987

• Venue

  European Journal of Pharmacology

• Publication date

  1987-07-23

• Fields of study

  Biology, Medicine, Chemistry

• Identifiers
  DOI 10.1016/0014-2999(87)90589-9PMID 3117574
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

• THE DIRECT DEMONSTRATION OF A BARRIER MECHANISM IN THE BRAIN CAPILLARIES.

  2009cited by this paper
• Substance P in the lumbar spinal cord of the rat affects the motor response to 5-HTP and TRH.

  1986cited by this paper
• Potentiation of motoneurone excitability by combined administration of 5-HT agonist and TRH analogue.

  1985cited by this paper
• Up-regulation of thyrotropin-releasing hormone (TRH) receptors in rat spinal cord after codepletion of serotonin and TRH.

  1985cited by this paper
• Cardio-respiratory actions of substance P, TRH and 5-HT in the nucleus tractus solitarius of rats: evidence for functional interactions of neuropeptides and amine neurotransmitters.

  1985cited by this paper
• Denervation supersensitivity to 5-hydroxytryptamine in the rat spinal cord is not due to the absence of 5-hydroxytryptamine.

  1985cited by this paper
• Supersensitivity to the respiratory stimulatory effect of TRH in 5,7-dihydroxytryptamine-treated rats.

  1984cited by this paper
• Properties of the depolarization induced by TRH in the isolated frog spinal cord.

  1984cited by this paper
• Codepletion of serotonin and TRH induces apparent supersensitivity of spinal TRH receptors.

  1983cited by this paper
• The effects of monoamine neurotoxins on peptides in the rat spinal cord.

  1982cited by this paper
• Localization and release of 5-hydroxytryptamine thyrotrophin releasing hormone and substance P in rat ventral spinal cord.

  1982cited by this paper
• Ventral root depolarization and spinal reflex augmentation by a TRH analog in rat spinal cord.

  1982cited by this paper
• Altered neuronal responsiveness to biogenic amines in rat cerebral cortex after serotonin denervation or depletion.

  1982cited by this paper
• Immunohistochemical support for three putative transmitters in one neuron: coexistence of 5-hydroxytryptamine, substance P- and thyrotropin releasing hormone-like immunoreactivity in medullary neurons projecting to the spinal cord.

  1981cited by this paper
• Non-serotonergic depression of spinal monosynaptic reflex transmission by 5-hydroxytryptophan.

  1981cited by this paper
• Similar motor effects of 5-HT and TRH in rats following chronic spinal transection and 5.7-dihydroxytryptamine injection.

  1981cited by this paper
• The action of thyrotropin-releasing hormone, substance P and related peptides on frog spinal motoneurons.

  1978cited by this paper
• Coupling of peptides to albumin with difluorodinitrobenzene.

  1976cited by this paper
• Behavioral evidence for supersensitivity following destruction of central serotonergic nerve terminals by 5,7-dihydroxytryptamine.

  1976cited by this paper
• Coupling of peptides to albumin with difluorodinitrobenzene.

  1976cited by this paper
• DECARBOXYLATION OF EXOGENOUS l‐5‐HYDROXYTRYPTOPHAN AFTER DESTRUCTION OF THE CEREBRAL RAPHE SYSTEM

  1974cited by this paper
• Functional regeneration of 5-hydroxytryptamine nerve terminals in the rat spinal cord following 5, 6-dihydroxytryptamine induced degeneration.

  1974cited by this paper
• Axonal degeneration and regeneration of bulbo-spinal indolamine neurons after 5,6-dihydroxytryptamine treatment.

  1973cited by this paper
• Effects of 5,6-dihydroxytryptamine on nerve terminal serotonin and serotonin uptake in the rat brain.

  1973cited by this paper
• THE EFFECTS OF 5-HYDROXYTRYPTOPHAN AND l-TRYPTOPHAN ON SPINAL SYNAPTIC ACTIVITY

  1966cited by this paper

• Contribution of 5‐HT to locomotion – the paradox of Pet‐1−/− mice

  2011cites this paper
• Spinal cord injury-specific depression of monosynaptic spinal reflex transmission by l-5-hydroxytryptophan results from loss of the 5-HT uptake system and not 5-HT receptor supersensitivity.

  2006cites this paper
• Endogenously released 5-hydroxytryptamine depresses the spinal monosynaptic reflex via 5-HT1D receptors.

  2004cites this paper
• Serotonergic depression of spinal monosynaptic transmission is mediated by 5-HT1B receptors.

  2003cites this paper
• The role of serotonin in reflex modulation and locomotor rhythm production in the mammalian spinal cord.

  2000cites this paper
• [Induction of noradrenergic supersensitivity following cordotomy in neonatal rat spinal motoneurons].

  1999cites this paper
• Effects of 8-OH-DPAT, a 5-HT1A receptor agonist, and DOI, a 5-HT2A/2C agonist, on monosynaptic transmission in spinalized rats.

  1996cites this paper
• Thyrotropin-releasing hormone reverses the supersensitively depressed monosynaptic transmission by serotonin in 5,7-dihydroxytryptamine-treated neonatal rats in vitro.

  1994cites this paper
• Descending inhibition in the neonate rat spinal cord is mediated by 5-hydroxytryptamine.

  1993cites this paper
• Depression of the monosynaptic reflex by apomorphine or bromocriptine is not mediated by D1/D2 receptors.

  1993cites this paper
• FAST and SLOW ipsilateral and contralateral spinal reflexes in the neonate rat are modulated by 5-HT.

  1992cites this paper
• Stimulatory and inhibitory effects of serotonergic hallucinogens on spinal mono- and polysynaptic reflex pathways in the rat.

  1992cites this paper
• Functional supersensitivity ofα1-adrenergic system in spinal ventral horn is due to absence of an uptake system and not to postsynaptic change

  1991cites this paper
• Effects of 5-hydroxytryptamine on cat spinal motoneurons.

  1991cites this paper
• 2-phenylethylamine and methamphetamine enhance the spinal monosynaptic reflex by releasing noradrenaline from the terminals of descending fibers.

  1991cites this paper
• [Pharmacology of neural mechanisms of the motor system].

  1991cites this paper
• The most desirable conformation of phenylethylamine (PEA) moiety stimulating noradrenergic neurons: effects of PEA, methamphetamine, phenelzine, methylphenidate, nomifensine and mazindol on rat spinal reflexes.

  1989cites this paper
• Enhancement of spinal monosynaptic reflexes with phenylethylamine and related drugs through descending noradrenergic neurons.

  1989cites this paper
• Chronic thyrotropin-releasing hormone decreases the affinity and increases the number of its own receptor in the spinal cord.

  1989cites this paper
• Structure-activity relationships of phenylethylamine analogs in their serotonergic depressant effects on the spinal monosynaptic reflex in rats.

  1989cites this paper
• A comparison of 5-hydroxytryptophan effects on rat lumbar spinal cord serotonin release and monosynaptic response amplitude.

  1989cites this paper
• Functional significance of subtypes of 5-HT receptors in the rat spinal reflex pathway.

  1988cites this paper
• Inhibitory effect of phencyclidine on rat spinal reflexes.

  1988cites this paper
• Thyrotropin releasing hormone, cholecystokinin and endogenous opioids in the modulation of spinal reflexes in the rabbit.

  1988cites this paper
• The spinal reflex of chronic spinal rats is supersensitive to 5-HTP but not to TRH or 5-HT agonists.

  1988cites this paper