Modulation of DNA Repair and Glutathione Levels in Human Keratinocytes by Micromolar Arsenite

No abstract is available for this paper.

• Publication year

  1999

• Venue

  Unknown venue

• Publication date

  Unknown publication date

• Fields of study

  Biology, Medicine, Chemistry, Environmental Science

• Identifiers
  DOI 10.1016/B978-008043648-7/50028-5
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar

• No claims are published for this paper.

• No concepts are published for this paper.

• The Stress Inducer Arsenite Activates Mitogen-activated Protein Kinases Extracellular Signal-regulated Kinases 1 and 2 via a MAPK Kinase 6/p38-dependent Pathway*

  1998cited by this paper
• Arsenic toxicity is enzyme specific and its affects on ligation are not caused by the direct inhibition of DNA repair enzymes.

  1998cited by this paper
• Arsenic toxicity is enzyme specific and arsenic inhibition of DNA repair is not caused by direct inhibition of repair enzymes

  1998cited by this paper
• Arsenite retards DNA break rejoining by inhibiting DNA ligation.

  1997cited by this paper
• Differentiation-dependent p53 regulation of nucleotide excision repair in keratinocytes.

  1997cited by this paper
• Reconstitution of human base excision repair with purified proteins.

  1997cited by this paper
• Modulation of the arsenite-induced expression of stress proteins by reducing agents.

  1997cited by this paper
• Arsenic can mediate skin neoplasia by chronic stimulation of keratinocyte-derived growth factors.

  1997cited by this paper
• Interaction of arsenic(III) with nucleotide excision repair in UV-irradiated human fibroblasts.

  1997cited by this paper
• Arsenic induces overexpression of growth factors in human keratinocytes.

  1996cited by this paper
• The tumor promoter arsenite stimulates AP‐1 activity by inhibiting a JNK phosphatase.

  1996cited by this paper
• Differential activation of ERK, JNK/SAPK and P38/CSBP/RK map kinase family members during the cellular response to arsenite.

  1996cited by this paper
• Reversible protein phosphorylation modulates nucleotide excision repair of damaged DNA by human cell extracts.

  1996cited by this paper
• Arsenic induces oxidant stress and NF-kappa B activation in cultured aortic endothelial cells.

  1996cited by this paper
• How phosphorylation regulates the activity of p53.

  1996cited by this paper
• Chromium‐ and nickel‐induced cytotoxicity in normal and transformed human keratinocytes: an investigation of pharmacological approaches to the prevention of Cr(VI)‐induced cytotoxicity

  1996cited by this paper
• Effect of arsenic and cadmium on the persistence of mutagen‐induced DNA lesions in human cells

  1996cited by this paper
• Induction of the mammalian stress response gene GADD153 by oxidative stress: role of AP-1 element.

  1996cited by this paper
• Specific Interaction of DNA Polymerase β and DNA Ligase I in a Multiprotein Base Excision Repair Complex from Bovine Testis*

  1996cited by this paper
• DNA repair in eukaryotes.

  1996cited by this paper
• DNA Polymerase Conducts the Gap-filling Step in Uracil-initiated Base Excision Repair in a Bovine Testis Nuclear Extract (*)

  1995cited by this paper
• In vitro inhibition of glutathione reductase by arsenotriglutathione.

  1995cited by this paper
• Enhancement of glutathione content in glutathione synthetase-deficient fibroblasts from a patient with 5-oxoprolinuria via metabolic cooperation with normal fibroblasts.

  1994cited by this paper
• Pathways of heat shock protein 28 phosphorylation by TNF in human fibroblasts.

  1994cited by this paper
• Analysis of HSF-1 phosphorylation in A549 cells treated with a variety of stresses.

  1994cited by this paper
• Arsenite enhances DNA double-strand breaks and cell killing of methyl methanesulfonate-treated cells by inhibiting the excision of alkali-labile sites.

  1993cited by this paper
• Reactions of arsenic(III) and arsenic(V) species with glutathione.

  1993cited by this paper
• Glutathione as a cellular defence against arsenite toxicity in cultured Chinese hamster ovary cells.

  1993cited by this paper
• Metal carcinogenesis: mechanistic implications.

  1992cited by this paper
• Effects of sodium arsenite on the cytoskeleton and cellular glutathione levels in cultured cells.

  1992cited by this paper
• Differential susceptibility to carcinogen‐induced amplification of sv40 and dhfr sequences in sv40‐transformed human keratinocytes

  1992cited by this paper
• Activation of mammalian DNA ligase I through phosphorylation by casein kinase II.

  1992cited by this paper
• Inhibition of DNA ligase activity by arsenite: a possible mechanism of its comutagenesis.

  1989cited by this paper
• Inhibition of human excision DNA repair by inorganic arsenic and the co-mutagenic effect in V79 Chinese hamster cells.

  1986cited by this paper
• [62] Glutathione transferases from human liver

  1985cited by this paper
• The carcinogenicity of arsenic.

  1981cited by this paper
• Absence of arsenite mutagenicity in E coli and Chinese hamster cells.

  1980cited by this paper
• Arsenite inhibits β-oxidation in isolated rat liver mitochondria

  1979cited by this paper
• Effects of arsenite on DNA repair in Escherichia coli

  1977cited by this paper
• Inhibition of mitochondrial energy-linked functions by arsenate. Evidence for a nonhydrolytic mode of inhibitor action.

  1971cited by this paper
• Enzymic method for quantitative determination of nanogram amounts of total and oxidized glutathione: applications to mammalian blood and other tissues.

  1969cited by this paper

• Mycoremediation of Heavy Metal/Metalloid-Contaminated Soil: Current Understanding and Future Prospects

  2016cites this paper
• Metallothionein does not sequester arsenic(III) ions in condition of acute arsenic toxicity.

  2016cites this paper
• Arsenic Binding to Proteins

  2013cites this paper
• Arsenic Biotransformation as a Cancer Promoting Factor by Inducing DNA Damage and Disruption of Repair Mechanisms

  2011cites this paper
• Mechanisms of malignant transformation of human urothelial cells by monomethylarsonous acid

  2011cites this paper
• Persistence of DNA damage following exposure of human bladder cells to chronic monomethylarsonous acid.

  2009cites this paper
• Modulation of DNA polymerase beta-dependent base excision repair in cultured human cells after low dose exposure to arsenite.

  2008cites this paper
• Comparison of the effects of arsenic and cadmium on benzo(a)pyrene-induced micronuclei in mouse bone-marrow.

  2007cites this paper
• Elevation of Cellular BPDE Uptake by Human Cells: A Possible Factor Contributing to Co-Carcinogenicity by Arsenite

  2006cites this paper
• Arsenic in Drinking Water

  2005cites this paper
• Arsenic, mode of action at biologically plausible low doses: what are the implications for low dose cancer risk?

  2005cites this paper
• Alteration of GSH level, gene expression and cell transformation in NIH3T3 cells by chronic exposure to low dose of arsenic

  2003cites this paper
• The metabolism of inorganic arsenic oxides, gallium arsenide, and arsine: a toxicochemical review.

  2003cites this paper
• Regulation of redox and DNA repair genes by arsenic: Low dose protection against oxidative stress?

  2003cites this paper
• Effect of arsenic on transcription factor AP-1 and NF-kappaB DNA binding activity and related gene expression.

  2002cites this paper
• An arsenic(III)‐oxidizing bacterial population: selection, characterization, and performance in reactors

  2002cites this paper
• Upregulation of glutathione-related genes and enzyme activities in cultured human cells by sublethal concentrations of inorganic arsenic.

  2002cites this paper
• Selected mechanisms of genotoxic effects of inorganic arsenic compounds.

  2001cites this paper
• Mode of Action Studies for Assessing Carcinogenic Risks Posed by Inorganic Arsenic

  1999cites this paper