Pathologically elevated cyclic hydrostatic pressure induces CD95-mediated apoptotic cell death in vascular endothelial cells.

No abstract is available for this paper.

• Publication year

  2005

• Venue

  American Journal of Physiology - Cell Physiology

• Publication date

  2005-08-01

• Fields of study

  Biology, Medicine

• Identifiers
  DOI 10.1152/AJPCELL.00107.2004PMID 15772124
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

• Differential responsiveness of vascular endothelial cells to different types of fluid mechanical shear stress

  2007cited by this paper
• Non-apoptotic Fas signaling.

  2003cited by this paper
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  2003cited by this paper
• Activation of the JNK signaling pathway: Breaking the brake on apoptosis

  2003cited by this paper
• Physiologic cyclic stretch inhibits apoptosis in vascular endothelium

  2003cited by this paper
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  2002cited by this paper
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  2002cited by this paper
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  2002cited by this paper
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  2002cited by this paper
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  2002cited by this paper
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  2002cited by this paper
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  2002cited by this paper
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  2001cited by this paper
• Endothelial cell apoptosis: biochemical characteristics and potential implications for atherosclerosis.

  2001cited by this paper
• Atherosclerosis : Vascular biology

  2000cited by this paper
• Relation between endothelial cell apoptosis and blood flow direction in human atherosclerotic plaques.

  2000cited by this paper
• [Vascular remodeling].

  2000cited by this paper
• Vascular endothelial cells and smooth muscle cells differ in expression of Fas and Fas ligand and in sensitivity to Fas ligand-induced cell death: implications for vascular disease and therapy.

  2000influential reference
• The cardiac Fas (APO-1/CD95) Receptor/Fas ligand system : relation to diastolic wall stress in volume-overload hypertrophy in vivo and activation of the transcription factor AP-1 in cardiac myocytes.

  2000cited by this paper
• c-Jun-Dependent CD95-L Expression Is a Rate-Limiting Step in the Induction of Apoptosis by Alkylating Agents

  2000cited by this paper
• Autoamplification of apoptosis following ligation of CD95-L, TRAIL and TNF-α

  2000cited by this paper
• Endothelial cell response to different mechanical forces.

  2000cited by this paper
• Fas ligand promotes cell survival of immature human bone marrow CD34+CD38- hematopoietic progenitor cells by suppressing apoptosis.

  1999cited by this paper
• CREB: a stimulus-induced transcription factor activated by a diverse array of extracellular signals.

  1999cited by this paper
• Is extravasation a Fas-regulated process?

  1999cited by this paper
• Fluid shear stress stimulates phosphorylation of Akt in human endothelial cells: involvement in suppression of apoptosis.

  1998cited by this paper
• A regulatory element in the CD95 (APO‐1/Fas) ligand promoter is essential for responsiveness to TCR‐mediated activation

  1998cited by this paper
• Ambient pulsatile pressure modulates endothelial cell proliferation.

  1998cited by this paper
• Colon carcinoma cells use different mechanisms to escape CD95-mediated apoptosis.

  1998cited by this paper
• Changes in the Amplitude of Cyclic Load Biphasically Modulate Endothelial Cell DNA Synthesis and Division

  1997cited by this paper
• Lack of hemodynamic forces triggers apoptosis in vascular endothelial cells.

  1997cited by this paper
• Oxidized low-density lipoprotein induces apoptosis of human endothelial cells by activation of CPP32-like proteases. A mechanistic clue to the 'response to injury' hypothesis.

  1997cited by this paper
• Fluid Shear Stress‐Mediated Signal Transduction: How Do Endothelial Cells Transduce Mechanical Force into Biological Responses?

  1997cited by this paper
• A rapid and simple method for measuring thymocyte apoptosis by propidium iodide staining and flow cytometry.

  1991cited by this paper
• Preparation of extracts from higher eukaryotes.

  1990cited by this paper
• Textbook of Pharmacology

  1952cited by this paper

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  2008cites this paper
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  2008cites this paper
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  2007influential citation