A low nicotine concentration augments vesicle motion and exocytosis triggered by K(+) depolarisation of chromaffin cells.

No abstract is available for this paper.

• Publication year

  2008

• Venue

  European Journal of Pharmacology

• Publication date

  2008-11-19

• Fields of study

  Medicine, Chemistry

• Identifiers
  DOI 10.1016/j.ejphar.2008.09.003PMID 18831972
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

• A two-step model for acetylcholine control of exocytosis via nicotinic receptors.

  2008cited by this paper
• Single-Vesicle Catecholamine Release Has Greater Quantal Content and Faster Kinetics in Chromaffin Cells from Hypertensive, as Compared with Normotensive, Rats

  2008cited by this paper
• Ouabain augments and maintains the catecholamine release responses evoked by repetitive pulses of potassium, caffeine or histamine in perifused bovine chromaffin cells.

  2007cited by this paper
• Depolarization preconditioning produces cytoprotection against veratridine-induced chromaffin cell death.

  2006cited by this paper
• Sensitivity and tolerance to nicotine in smokers and nonsmokers

  2006cited by this paper
• New Roles of Myosin II during Vesicle Transport and Fusion in Chromaffin Cells*

  2004cited by this paper
• Modulation of presynaptic store calcium induces release of glutamate and postsynaptic firing.

  2003cited by this paper
• Calcium regulates exocytosis at the level of single vesicles

  2003cited by this paper
• Acetylcholine and potassium elicit different patterns of exocytosis in chromaffin cells when the intracellular calcium handling is disturbed

  2002cited by this paper
• Restriction of Secretory Granule Motion near the Plasma Membrane of Chromaffin Cells

  2001cited by this paper
• Vesicle pools and Ca2+ microdomains: new tools for understanding their roles in neurotransmitter release.

  1998cited by this paper
• Caffeine and cardiac arrhythmias. An experimental study in dogs with review of literature.

  1997cited by this paper
• Cardiac Arrhythmias Following Intravenous Nicotine: Experimental Study in Dogs

  1997cited by this paper
• Neuronal Nicotinic Acetylcholine Receptors on Bovine Chromaffin Cells: Cloning, Expression, and Genomic Organization of Receptor Subunits

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  2019cites this paper
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  2018cites this paper
• The quantal catecholamine release from mouse chromaffin cells challenged with repeated ACh pulses is regulated by the mitochondrial Na+/Ca2+ exchanger

  2017cites this paper
• Regulation by L channels of Ca2+-evoked secretory responses in ouabain-treated chromaffin cells

  2016cites this paper
• Aortic Remodelling in Chronic Nicotine-Administered Rat

  2014cites this paper
• Cytosolic organelles shape calcium signals and exo-endocytotic responses of chromaffin cells.

  2012cites this paper
• La Escuela de Farmacología de Madrid de D. Teófilo Hernando a Instituto de I+D del medicamento de la Universidad Autónoma de Madrid

  2012cites this paper
• Ouabain enhances exocytosis through the regulation of calcium handling by the endoplasmic reticulum of chromaffin cells.

  2011cites this paper
• Electrophysiological and morphological features underlying neurotransmission efficacy at the splanchnic nerve-chromaffin cell synapse of bovine adrenal medulla.

  2010cites this paper
• Is nicotine a key player or spectator in the induction and progression of cardiovascular disorders?

  2009cites this paper
• Preconditioning stimuli that augment chromaffin cell secretion.

  2009cites this paper