aureus Staphylococcus Virulence Factors in Response and Horizontal Transfer of -Lactam Antibiotics Induce the SOS β

No abstract is available for this paper.

• Publication year

  2013

• Venue

  Unknown venue

• Publication date

  Unknown publication date

• Fields of study

  Biology, Medicine

• Identifiers

  No identifiers available.

• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar

• No claims are published for this paper.

• No concepts are published for this paper.

• Antibiotic‐induced SOS response promotes horizontal dissemination of pathogenicity island‐encoded virulence factors in staphylococci

  2005cited by this paper
• SOS-Independent Induction of dinB Transcription by β-Lactam-Mediated Inhibition of Cell Wall Synthesis in Escherichia coli

  2005cited by this paper
• SOS Response Induction by ß-Lactams and Bacterial Defense Against Antibiotic Lethality

  2004cited by this paper
• SOS response promotes horizontal dissemination of antibiotic resistance genes

  2004cited by this paper
• Mobile genetic elements and bacterial toxinoses: the superantigen-encoding pathogenicity islands of Staphylococcus aureus.

  2003cited by this paper
• Genome-wide transcriptional profiling of the response of Staphylococcus aureus to cell-wall-active antibiotics reveals a cell-wall-stress stimulon.

  2003cited by this paper
• Two‐component system VraSR positively modulates the regulation of cell‐wall biosynthesis pathway in Staphylococcus aureus

  2003cited by this paper
• Expression of the Pseudomonas putida OCT Plasmid Alkane Degradation Pathway Is Modulated by Two Different Global Control Signals: Evidence from Continuous Cultures

  2003cited by this paper
• Sip, an integrase protein with excision, circularization and integration activities, defines a new family of mobile Staphylococcus aureus pathogenicity islands

  2003cited by this paper
• Pathogenicity and resistance islands of staphylococci.

  2001cited by this paper
• Molecular genetics of SaPI1 – a mobile pathogenicity island in Staphylococcus aureus

  2001cited by this paper
• Comparative gene expression profiles following UV exposure in wild-type and SOS-deficient Escherichia coli.

  2001cited by this paper
• Cell wall-active antibiotic induced proteins of Staphylococcus aureus identified using a proteomic approach.

  2001cited by this paper
• Identification of additional genes belonging to the LexA regulon in Escherichia coli

  2000cited by this paper
• Quinolone antibiotics induce Shiga toxin-encoding bacteriophages, toxin production, and death in mice.

  2000cited by this paper
• The gene for toxic shock toxin is carried by a family of mobile pathogenicity islands in Staphylococcus aureus

  1998cited by this paper
• LexA cleavage and other self-processing reactions

  1993cited by this paper
• Induction of the SOS response by new 4-quinolones.

  1987cited by this paper
• Properties of a cryptic high-frequency transducing phage in Staphylococcus aureus.

  1967cited by this paper

• No citing papers are available for this paper.