E-Selectin Polymorphism Associated With Myocardial Infarction Causes Enhanced Leukocyte-Endothelial Interactions Under Flow Conditions

No abstract is available for this paper.

• Publication year

  2003

• Venue

  Arteriosclerosis, Thrombosis and Vascular Biology

• Publication date

  2003-05-01

• Fields of study

  Medicine

• Identifiers
  DOI 10.1161/01.ATV.0000067427.40133.59PMID 12649084
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

• DMA polymorphisms in adhesion molecule genes — a new risk factor for early atherosclerosis

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• Gender- and age-dependent relationships between the E-selectin S128R polymorphism and coronary artery calcification

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• Glycosylation-induced Conformational Modification Positively Regulates Receptor-Receptor Association

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• Adenoviral transduction of human E‐selectin into isolated, perfused, rat aortic segments: an ex vivo model for studying leukocyte‐endothelial interactions

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• E-Selectin-Dependent Signaling Via the Mitogen-Activated Protein Kinase Pathway in Vascular Endothelial Cells1

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• Functional Characterization of Atherosclerosis-Associated Ser128Arg and Leu554Phe E-Selectin Mutations

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