Expression of adenylyl cyclase-4 (AC-4) in Y1 and forskolin-resistant adrenal cells.

No abstract is available for this paper.

• Publication year

  2004

• Venue

  Molecular and Cellular Endocrinology

• Publication date

  2004-02-27

• Fields of study

  Biology, Medicine

• Identifiers
  DOI 10.1016/J.MCE.2003.11.019PMID 15026181
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

• Forskolin-resistant Y1 adrenal cell mutants are deficient in adenylyl cyclase type 4.

  2004cited by this paper
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  1995cited by this paper
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  1991cited by this paper
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• Forskolin-resistant Y1 mutants harbor defects associated with the guanyl nucleotide-binding regulatory protein, Gs.

  1987cited by this paper
• Isolation of ACTH-resistant Y1 adrenal tumor cells.

  1985cited by this paper
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  1985cited by this paper
• Isolation of forskolin-resistant adrenal cells defective in the adenylate cyclase system.

  1984cited by this paper
• Analysis of the mutation to forskolin-resistance in Y1 adrenocortical tumor cells.

  1984cited by this paper
• Cyclic nucleotides in hormonal regulation of adrenocortical function.

  1980cited by this paper
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  1980cited by this paper
• Isolation of biologically active ribonucleic acid from sources enriched in ribonuclease.

  1979cited by this paper
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  1979cited by this paper
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• Phenotypically variant adrenal tumor cell cultures with biochemical lesions in the ACTH‐stimulated steroidogenic pathway

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• Retention of differentiated function in clonal animal cell lines, particularly hormone-secreting cultures.

  1968cited by this paper
• Clonal analysis of differentiated function in animal cell cultures. I. Possible correlated maintenance of differentiated function and the diploid karyotype.

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