D. S. Boeldt

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Aliases
D S Boeldt, Derek S Boeldt, Derek S. Boeldt
Publication record
39 papers · 687 citations · H-index 15

PAPERS

Immune Cells and Inflammatory mediators cause endothelial dysfunction in a vascular microphysiological system
202318 citationsauthor #5
Mononuclear Cells Negatively Regulate Endothelial Ca^2+ Signaling
20231 citationsauthor #5
Src kinase partially mediates cytokine-induced endothelial dysfunction.
20230 citationsauthor #6
The Wide Reach of Fatty Acids and their Metabolites.
20222 citationsauthor #1
Differential Distribution of Tryptophan-Metabolites in Fetal and Maternal Circulations During Normotensive and Preeclamptic Pregnancies
202117 citationsauthor #6
Fatty acids in normal and pathological pregnancies.
202117 citationsauthor #4
Cytokine concentrations direct endothelial function in pregnancy and preeclampsia.
202010 citationsauthor #7
Adenoviral transduction of EGFR into pregnancy-adapted uterine artery endothelial cells remaps growth factor induction of endothelial dysfunction.
202022 citationsauthor #2
Maternal disease and gasotransmitters.
202017 citationsauthor #3
Conjugated linoleic acid improves endothelial Ca2+ signaling by blocking growth factor and cytokine-mediated Cx43 phosphorylation.
20206 citationsauthor #8
TNF-alpha inhibits pregnancy-adapted Ca2+ signaling in uterine artery endothelial cells.
201914 citationsauthor #2
Positive versus negative effects of VEGF165 on Ca2+ signaling and NO production in human endothelial cells.
201720 citationsauthor #1
Pregnancy‐adapted uterine artery endothelial cell Ca2+ signaling and its relationship with membrane potential
20173 citationsauthor #2
960: Modeling preeclampsia endothelial dysfunction in a HUVEC model with VEGF, TNFα and IL-6 with rescue by CLA
20170 citationsauthor #2
Vascular adaptation in pregnancy and endothelial dysfunction in preeclampsia.
2017259 citationsauthor #1
Vascular Biology and Microcirculation Positive versus negative effects of VEGF 165 on Ca 2 (cid:2) signaling and NO production in human endothelial cells
20160 citationsauthor #1
Changes in Ca2+ Signaling and Nitric Oxide Output by Human Umbilical Vein Endothelium in Diabetic and Gestational Diabetic Pregnancies1
201510 citationsauthor #3
Phosphorylation of Ser-279/282 and Tyr-265 positions on Cx43 as possible mediators of VEGF-165 inhibition of pregnancy-adapted Ca2+ burst function in ovine uterine artery endothelial cells.
201520 citationsauthor #1
Altered VEGF-stimulated Ca2+ signaling in part underlies pregnancy-adapted eNOS activity in UAEC.
201423 citationsauthor #1
700: Pregnancy-enhanced changes in membrane potential are not driven by pregnancy-enhanced Ca2+ signaling in uterine artery endothelial cells (UAEC)
20140 citationsauthor #2
Pregnancy programming and preeclampsia: identifying a human endothelial model to study pregnancy-adapted endothelial function and endothelial adaptive failure in preeclamptic subjects.
201416 citationsauthor #1
259: CLA as a Src inhibitor: an innovative approach to rescue endothelial function in preeclampsia
20140 citationsauthor #2
72: 10:12 Conjugated linoleic acid (CLA) isomer rescues HUVEC cell dysfunction in a preeclamptic in-vitro model
20140 citationsauthor #2
264: 10:12 CLA as an endothelial rescue agent: a novel dietary approach to preeclampsia therapy
20140 citationsauthor #1
292: Modeling preeclamptic endothelial failure in HUVEC-CS
20140 citationsauthor #2
The loss of sustained Ca(2+) signaling underlies suppressed endothelial nitric oxide production in preeclamptic pregnancies: implications for new therapy.
201311 citationsauthor #2
Endothelial Nitric Oxide Production in Preeclamptic Pregnancies
201325 citationsauthor #2
Regulating Specific Growth Factor Signaling Using Immobilized Branched Ligands
201216 citationsauthor #4
766: Dysfunctional vascular adaptations in human umbilical vein endothelial cells (HUVEC) in preeclamptic pregnancies
20120 citationsauthor #2
Artery Endothelium: Relative roles in pregnancy adaptation and reversal by
20110 citationsauthor #2
eNOS activation and NO function: pregnancy adaptive programming of capacitative entry responses alters nitric oxide (NO) output in vascular endothelium--new insights into eNOS regulation through adaptive cell signaling.
201151 citationsauthor #1
President’s Plenary Session
20110 citationsauthor #5
eNOS activation and NO function: Pregnancy adaptive programming of capacitative entry responses alters nitric oxide (NO) output in vascular endothelium–new insights into eNOS regulation through adaptive cell signaling
20110 citationsauthor #1
252: TNF alpha has a direct inhibitory effect on Ca2+ responses necessary for eNOS activation in primary HUVEC
20110 citationsauthor #3
[Ca2+]i signaling vs. eNOS expression as determinants of NO output in uterine artery endothelium: relative roles in pregnancy adaptation and reversal by VEGF165.
201134 citationsauthor #2
255: Alterations of calcium (Ca2+) signaling in human umbilical vein endothelial cells (HUVEC) suggesting a unique vascular adaptation in pre-existing diabetes mellitus (DM) versus gestational diabetes mellitus (GDM) pregnancies
20110 citationsauthor #2
Pregnancy Induced Reprogramming of Endothelial Function in Response to ATP: Evidence for Post Receptor Ca2+ Signaling Plasticity
201013 citationsauthor #2
Pregnancy Enhances Sustained Ca2+ Bursts and Endothelial Nitric Oxide Synthase Activation in Ovine Uterine Artery Endothelial Cells Through Increased Connexin 43 Function1
201065 citationsauthor #2
Signaling vs eNOS Expression as Determinants of NO Output In Uterine 1 Artery Endothelium : Relative roles in pregnancy adaptation and reversal by 2 VEGF 165 . 3 4 5
20100 citationsauthor #2

TIMELINE

2010 78 citations

2011 85 citations

2012 16 citations

2013 36 citations

2014 39 citations

2015 30 citations

2016 0 citations

2017 282 citations

2019 14 citations

2020 55 citations

2021 34 citations

2022 2 citations

2023 19 citations

D. S. Boeldt

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