MÉCANISMES D'ACTION DE LA CONTRACTION MUSCULAIRE SUR LE TRANSPORT DU GLUCOSE DANS LE MUSCLE SQUELETTIQUE DE RAT

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• Publication year

  2003

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• Fields of study

  Biology, Medicine, Chemistry

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  Open on Semantic Scholar

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• Activation of AMPK is essential for AICAR-induced glucose uptake by skeletal muscle but not adipocytes.

  2002cited by this paper
• Activation of the ERK Pathway and Atypical Protein Kinase C Isoforms in Exercise- and Aminoimidazole-4-carboxamide- 1-β-d-riboside (AICAR)-stimulated Glucose Transport*

  2002cited by this paper
• Identification of a novel glucose transporter-like protein-GLUT-12.

  2002cited by this paper
• Exercise-induced transcription of the muscle glucose transporter (GLUT 4) gene.

  2002cited by this paper
• Effects of endurance training on activity and expression of AMP-activated protein kinase isoforms in rat muscles.

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• Effect of fiber type and nutritional state on AICAR- and contraction-stimulated glucose transport in rat muscle.

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• Contraction Regulation of Akt in Rat Skeletal Muscle*

  2002cited by this paper
• Sustained exposure of L6 myotubes to high glucose and insulin decreases insulin-stimulated GLUT4 translocation but upregulates GLUT4 activity.

  2002cited by this paper
• Effect of AICAR treatment on glycogen metabolism in skeletal muscle.

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• Insulin signaling after exercise in insulin receptor substrate-2-deficient mice.

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• Contrasting effects of exercise and NOS inhibition on tissue-specific fatty acid and glucose uptake in mice.

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• Endosomes: multipurpose designs for integrating housekeeping and specialized tasks

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• Role of PKC isoforms in glucose transport in 3T3-L1 adipocytes: insignificance of atypical PKC.

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• A Dominant-negative p38 MAPK Mutant and Novel Selective Inhibitors of p38 MAPK Reduce Insulin-stimulated Glucose Uptake in 3T3-L1 Adipocytes without Affecting GLUT4 Translocation*

  2002cited by this paper
• Progressive increase in human skeletal muscle AMPKalpha2 activity and ACC phosphorylation during exercise.

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• Glycogen-dependent effects of 5-aminoimidazole-4-carboxamide (AICA)-riboside on AMP-activated protein kinase and glycogen synthase activities in rat skeletal muscle.

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• Chronic treatment with 5-aminoimidazole-4-carboxamide-1-beta-D-ribofuranoside increases insulin-stimulated glucose uptake and GLUT4 translocation in rat skeletal muscles in a fiber type-specific manner.

  2001cited by this paper
• An activating mutation in the gamma1 subunit of the AMP-activated protein kinase.

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• A role for AMP-activated protein kinase in contraction- and hypoxia-regulated glucose transport in skeletal muscle.

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• AMP-activated protein kinase (AMPK) is activated in muscle of subjects with type 2 diabetes during exercise.

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• Stimulation of Glucose Transport by AMP-activated Protein Kinase via Activation of p38 Mitogen-activated Protein Kinase*

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• The antihyperglycemic drug alpha-lipoic acid stimulates glucose uptake via both GLUT4 translocation and GLUT4 activation: potential role of p38 mitogen-activated protein kinase in GLUT4 activation.

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• An activating mutation in the γ1 subunit of the AMP‐activated protein kinase

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• Lipid raft microdomain compartmentalization of TC10 is required for insulin signaling and GLUT4 translocation

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• Insulin signalling and the regulation of glucose and lipid metabolism

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• Nitric oxide increases glucose uptake through a mechanism that is distinct from the insulin and contraction pathways in rat skeletal muscle.

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• Intracellular signalling mechanisms regulating glucose transport in insulin-sensitive tissues (review).

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• AMP-activated protein kinase activity and glucose uptake in rat skeletal muscle.

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• The Endosomal Compartment Is an Insulin-Sensitive Recruitment Site for GLUT4 and GLUT1 Glucose Transporters in Cardiac Myocytes.

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• Differential Effects of Phosphatidylinositol 3-Kinase Inhibition on Intracellular Signals Regulating GLUT4 Translocation and Glucose Transport*

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• Nitric oxide synthases: structure, function and inhibition.

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• GLUT4 translocation precedes the stimulation of glucose uptake by insulin in muscle cells: potential activation of GLUT4 via p38 mitogen-activated protein kinase.

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• Physiology of nitric oxide in skeletal muscle.

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• Molecular cloning of a novel member of the GLUT family of transporters, SLC2a10 (GLUT10), localized on chromosome 20q13.1: a candidate gene for NIDDM susceptibility.

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• Amino acid and insulin signaling via the mTOR/p70 S6 kinase pathway. A negative feedback mechanism leading to insulin resistance in skeletal muscle cells.

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• Insulin Accelerates Inter-endosomal GLUT4 Traffic via Phosphatidylinositol 3-Kinase and Protein Kinase B*

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• Expression of a prenylation-deficient Rab4 inhibits the GLUT4 translocation induced by active phosphatidylinositol 3-kinase and protein kinase B.

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• Energy-sensing and signaling by AMP-activated protein kinase in skeletal muscle.

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• VAMP3 Null Mice Display Normal Constitutive, Insulin- and Exercise-Regulated Vesicle Trafficking

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• Effects of concentric and eccentric contractions on phosphorylation of MAPKerk1/2 and MAPKp38 in isolated rat skeletal muscle

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• Insulin-regulated release from the endosomal recycling compartment is regulated by budding of specialized vesicles.

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• Acetylcholine receptors and neuronal nitric oxide synthase distribution at the neuromuscular junction of regenerated muscle fibers

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• The insulin-sensitive GLUT4 storage compartment is a postendocytic and heterogeneous population recruited by acute exercise.

  2001cited by this paper
• Insulin-stimulated GLUT4 translocation requires the CAP-dependent activation of TC10

  2001cited by this paper
• Acute exposure to AICAR increases glucose transport in mouse EDL and soleus muscle.

  2001cited by this paper
• Static stretch increases c-Jun NH2-terminal kinase activity and p38 phosphorylation in rat skeletal muscle.

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• Insulin-stimulated Activation of eNOS Is Independent of Ca2+ but Requires Phosphorylation by Akt at Ser1179 *

  2001cited by this paper
• Moving the insulin-regulated glucose transporter GLUT4 into and out of storage.

  2001cited by this paper
• Intracellular signaling specificity in skeletal muscle in response to different modes of exercise.

  2001cited by this paper
• AMP-activated protein kinase activation prevents denervation-induced decline in gastrocnemius GLUT-4.

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• Coordinated Control of Endothelial Nitric-oxide Synthase Phosphorylation by Protein Kinase C and the cAMP-dependent Protein Kinase*

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• MKK6/3 and p38 MAPK Pathway Activation Is Not Necessary for Insulin-induced Glucose Uptake but Regulates Glucose Transporter Expression*

  2001cited by this paper
• Targeted disruption of inducible nitric oxide synthase protects against obesity-linked insulin resistance in muscle

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• Mechanism of impaired nitric oxide synthase activity in skeletal muscle of streptozotocin-induced diabetic rats

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• Marathon running transiently increases c‐Jun NH2‐terminal kinase and p38γ activities in human skeletal muscle

  2000cited by this paper
• Expression of a Prenylation-Deficient Rab4 Interferes with Propagation of Insulin Signaling through Insulin Receptor Substrate-11.

  2000cited by this paper
• Contraction inhibits insulin-stimulated insulin receptor substrate-1/2-associated phosphoinositide 3-kinase activity, but not protein kinase B activation or glucose uptake, in rat muscle.

  2000cited by this paper
• A mutation in PRKAG3 associated with excess glycogen content in pig skeletal muscle.

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• Modulation of GLUT4 and GLUT1 recycling by insulin in rat adipocytes: kinetic analysis based on the involvement of multiple intracellular compartments.

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• Nitric oxide: a new player in the modulation of energy metabolism

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• Rab11 is associated with GLUT4-containing vesicles and redistributes in response to insulin

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• GLUTX1, a Novel Mammalian Glucose Transporter Expressed in the Central Nervous System and Insulin-sensitive Tissues*

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• Exercise induces isoform-specific increase in 5'AMP-activated protein kinase activity in human skeletal muscle.

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• Effects of adenoviral gene transfer of wild-type, constitutively active, and kinase-defective protein kinase C-lambda on insulin-stimulated glucose transport in L6 myotubes.

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• Expression of a prenylation-deficient Rab4 interferes with propagation of insulin signaling through insulin receptor substrate-1.

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• Characterization of Insulin-Responsive GLUT4 Storage Vesicles Isolated from 3T3-L1 Adipocytes

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• Nitric oxide‐mediated metabolic regulation during exercise: effects of training in health and cardiovascular disease

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• Effect of stimulation frequency on contraction-induced glucose transport in rat skeletal muscle.

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• Mice with gene disruption of both endothelial and neuronal nitric oxide synthase exhibit insulin resistance.

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• CAP defines a second signalling pathway required for insulin-stimulated glucose transport

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• VAMP2, but not VAMP3/cellubrevin, mediates insulin-dependent incorporation of GLUT4 into the plasma membrane of L6 myoblasts.

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• Stimulation of MAPK cascades by insulin and osmotic shock: lack of an involvement of p38 mitogen-activated protein kinase in glucose transport in 3T3-L1 adipocytes.

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• Activation of AMP-Activated Protein Kinase as a Unifying Coupling Mechanism

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• Increased expression of GLUT-4 and hexokinase in rat epitrochlearis muscles exposed to AICAR in vitro.

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• AMPK signaling in contracting human skeletal muscle: acetyl-CoA carboxylase and NO synthase phosphorylation.

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• Characterization of AMP-activated protein kinase gamma-subunit isoforms and their role in AMP binding.

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• GLUT8, a Novel Member of the Sugar Transport Facilitator Family with Glucose Transport Activity*

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• Acute exercise increases nitric oxide synthase activity in skeletal muscle.

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