The response of smooth muscle cells to α‐thrombin depends on its arterial origin: comparison among different species

No abstract is available for this paper.

• Publication year

  1998

• Venue

  European Journal of Clinical Investigation

• Publication date

  1998-04-01

• Fields of study

  Biology, Medicine

• Identifiers
  DOI 10.1046/j.1365-2362.1998.00280.xPMID 9615911
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

• Prolonged thrombin inhibition reduces restenosis after balloon angioplasty in porcine coronary arteries.

  1998cited by this paper
• Mevalonate deprivation impairs IGF-I/insulin signaling in human vascular smooth muscle cells.

  1997cited by this paper
• Thrombin receptor activation elicits rapid protein tyrosine phosphorylation and stimulation of the raf-1/MAP kinase pathway preceding delayed mitogenesis in cultured rat aortic smooth muscle cells: evidence for an obligate autocrine mechanism promoting cell proliferation induced by G-protein-coupled

  1996cited by this paper
• Atherosclerosis-prone branch regions in human aorta: microarchitecture and cell composition of intima.

  1996cited by this paper
• Human and porcine smooth muscle cells share similar proliferation dependence on the mevalonate pathway: implication for in vivo interventions in the porcine model

  1996cited by this paper
• G-protein coupled and tyrosine kinase receptors: evidence that activation of the insulin-like growth factor I receptor is required for thrombin-induced mitogenesis of rat aortic smooth muscle cells.

  1996cited by this paper
• Evidence for Cultured Human Vascular Smooth Muscle Cell Heterogeneity: Isolation of Clonal Cells and Study of their Growth Characteristics

  1996cited by this paper
• Variability in the proliferative responsiveness of cultured human vascular smooth muscle cells to α‐thrombin

  1995cited by this paper
• Regulation of differentiation of vascular smooth muscle cells.

  1995cited by this paper
• A comparison of hirudin with heparin in the prevention of restenosis after coronary angioplasty. Helvetica Investigators.

  1995cited by this paper
• Structural domains of thrombin involved in the induction of mitogenesis in cultured human vascular smooth muscle cells

  1995cited by this paper
• Determinants of Thrombin Receptor Cleavage. RECEPTOR DOMAINS INVOLVED, SPECIFICITY, AND ROLE OF THE P3 ASPARTATE (*)

  1995cited by this paper
• Immediate-early gene expression in response to hypertrophic and proliferative stimuli in pulmonary arterial smooth muscle cells.

  1994cited by this paper
• Endothelial cell function and thrombosis.

  1994cited by this paper
• Endothelial cell function and thrombosis.

  1994cited by this paper
• Antiproliferative activity to vascular smooth muscle cells and receptor binding of heparin-mimicking polyaromatic anionic compounds.

  1994cited by this paper
• Thrombotic mechanisms in atherosclerosis.

  1994cited by this paper
• Local delivery of r-hirudin by a double-balloon perfusion catheter prevents mural thrombosis and minimizes platelet deposition after angioplasty.

  1994cited by this paper
• Inhibition of growth of thrombus on fresh mural thrombus. Targeting optimal therapy.

  1994cited by this paper
• Identification of the sites of phosphorylation in insulin-like growth factor binding protein-1. Regulation of its affinity by phosphorylation of serine 101.

  1993cited by this paper
• Thrombin stimulates proliferation of cultured rat aortic smooth muscle cells by a proteolytically activated receptor.

  1993cited by this paper
• Thrombin-induced mitogenesis of vascular SMC is partially mediated by autocrine production of PDGF-AA.

  1993cited by this paper
• An International Collaborative Study to Investigate Standardisation of Hirudin Potency

  1993cited by this paper
• Kinetics of thrombin receptor cleavage on intact cells. Relation to signaling.

  1993cited by this paper
• The mitogenic effect of thrombin in vascular smooth muscle cells is largely due to basic fibroblast growth factor.

  1993cited by this paper
• Homologous desensitization of HEL cell thrombin receptors. Distinguishable roles for proteolysis and phosphorylation.

  1992cited by this paper
• Thrombin‐induced proliferation and expression of platelet‐derived growth factor‐A chain gene in human vascular smooth muscle cells

  1992cited by this paper
• Characterization of a functional thrombin receptor. Issues and opportunities.

  1992cited by this paper
• HIRUDIN INTERACTIONS WITH THROMBIN

  1992cited by this paper
• Thrombin receptor expression in normal and atherosclerotic human arteries.

  1992cited by this paper
• Thrombin as a multifunctional protein: induction of cell adhesion and proliferation.

  1992cited by this paper
• The pathogenesis of coronary artery disease and the acute coronary syndromes (1).

  1992cited by this paper
• Thrombin-induced events in non-platelet cells are mediated by the unique proteolytic mechanism established for the cloned platelet thrombin receptor

  1992cited by this paper
• The pathogenesis of coronary artery disease and the acute coronary syndromes (2).

  1992cited by this paper
• Inhibition of tyrosine phosphorylation prevents thrombin-induced mitogenesis, but not intracellular free calcium release, in vascular smooth muscle cells.

  1992cited by this paper
• Effect of Chemical Modification of α-Thrombin on Its Reaction with Platelets and Nucleated Cells

  1992cited by this paper
• Molecular cloning of a functional thrombin receptor reveals a novel proteolytic mechanism of receptor activation.

  1991cited by this paper
• Dissociation between activation of growth-related genes and mitogenic responses of neonatal vascular smooth muscle cells.

  1991cited by this paper
• Thrombin-stimulated events in cultured vascular smooth-muscle cells.

  1991cited by this paper
• Thrombin regulation of platelet interaction with damaged vessel wall and isolated collagen type I at arterial flow conditions in a porcine model: effects of hirudins, heparin, and calcium chelation.

  1991cited by this paper
• Effectiveness of Recombinant Desulphatohirudin in Reducing Restenosis After Balloon Angioplasty of Atherosclerotic Femoral Arteries in Rabbits

  1991cited by this paper
• Time course of smooth muscle cell proliferation in the intima and media of arteries following experimental angioplasty.

  1990cited by this paper
• Thrombin immobilized to extracellular matrix is a potent mitogen for vascular smooth muscle cells: nonenzymatic mode of action.

  1990cited by this paper
• Thrombin signal transduction mechanisms in rat vascular smooth muscle cells. Calcium and protein kinase C-dependent and -independent pathways.

  1990cited by this paper
• Syndromes of accelerated atherosclerosis: role of vascular injury and smooth muscle cell proliferation.

  1990cited by this paper
• Clot-bound thrombin is protected from inhibition by heparin-antithrombin III but is susceptible to inactivation by antithrombin III-independent inhibitors.

  1990cited by this paper
• Effects of thrombin inhibition on the development of acute platelet-thrombus deposition during angioplasty in pigs. Heparin versus recombinant hirudin, a specific thrombin inhibitor.

  1989cited by this paper
• Isolation and characterization of the c-fos(rat) cDNA and analysis of post-translational modification in vitro.

  1987cited by this paper
• Thrombin activation of the Na+/H+ exchanger in vascular smooth muscle cells. Evidence for a kinase C-independent pathway which is Ca2+-dependent and pertussis toxin-sensitive.

  1987cited by this paper
• Thrombin stimulates c-sis gene expression in microvascular endothelial cells.

  1986cited by this paper
• Thrombin-Elicited Contractile Responses of Aortic Smooth Muscle

  1985cited by this paper
• Cloning and characterization of different human sequences related to the onc gene (v-myc) of avian myelocytomatosis virus (MC29).

  1982cited by this paper
• Cloning and characterization of ribosomal RNA genes from wheat and barley.

  1979cited by this paper
• A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye binding.

  1976cited by this paper

• Viscoelastic properties of multi-layered cellularized vascular tissues fabricated from collagen gel.

  2018cites this paper
• Genética y biología molecular en cardiología (II) Biología celular y molecular de las lesiones ateroscleróticas

  2016cites this paper
• Implicación del Low Density Lipoprotein Receptor-Related Protein 1 (LRP1) en el remodelado vascular y cardiaco

  2015cites this paper
• Thrombin and protease-activated receptors (PARs) in atherothrombosis

  2008cites this paper
• Neuron‐derived orphan receptor‐1 (NOR‐1) is induced by thrombin and mediates vascular endothelial cell growth

  2007cites this paper
• Preparation and characterization of a scaffold for vascular tissue engineering by direct-assembling of collagen and cells in a cylindrical geometry.

  2007cites this paper
• Serpins, the vasculature, and viral therapeutics.

  2006cites this paper
• Biological performances of collagen-based scaffolds for vascular tissue engineering.

  2005cites this paper
• DEVELOPMENT AND VALIDATION OF A COLLAGEN-BASED SCAFFOLD FOR VASCULAR TISSUE ENGINEERING

  2005cites this paper
• Intracellular lipid accumulation, low‐density lipoprotein receptor‐related protein expression, and cell survival in vascular smooth muscle cells derived from normal and atherosclerotic human coronaries

  2004cites this paper
• [Neovascularization in human coronary arteries with lesions of different severity].

  2003influential citation
• Neovascularización en arterias coronarias humanas con distintos grados de lesión

  2003cites this paper
• Einfluss von Thrombin und Thrombinrezeptor-aktivierenden Peptiden (TRAP) auf die Mitogenese von Gefäßmuskelzellen: Untersuchungen zur Signaltransduktion

  2002cites this paper
• [Cellular and molecular biology of atherosclerotic lesions].

  2001cites this paper
• Atherosclerosis and Thrombosis: Lessons from Animal Models

  2001cites this paper
• Inhibition of cellular action of thrombin by N3-cyclopropyl-7-[[4-(1-methylethyl)phenyl]methyl]-7H-pyrrolo[3, 2-f]quinazoline-1,3-diamine (SCH 79797), a nonpeptide thrombin receptor antagonist.

  2000cites this paper