Diagnostic performance of QT interval variables from 24-h electrocardiography in the long QT syndrome.

No abstract is available for this paper.

• Publication year

  1998

• Venue

  European Heart Journal

• Publication date

  Unknown publication date

• Fields of study

  Medicine

• Identifiers
  DOI 10.1053/EUHJ.1997.0730PMID 9503190
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

• Long QT syndrome

  2010cited by this paper
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• Erratum: Long QT syndrome patients with mutations on the SCN5A and HERG genes have differential responses to Na+ channel blockade and to increases in heart rate: Implications for gene-specific therapy (Circulation (1995) 92 (3381-3386))

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• Different circadian behavior of the apex and the end of the T wave.

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• Long QT syndrome patients with mutations of the SCN5A and HERG genes have differential responses to Na+ channel blockade and to increases in heart rate. Implications for gene-specific therapy.

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• A molecular basis for cardiac arrhythmia: HERG mutations cause long QT syndrome.

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• Low level of expression of known deafness genes Kcne1, Kcnj10 or Col4a3 is sufficient to maintain hearing in mice.

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