Supporting evidence for using Perispinal Etanercept to inhibit TNF&agr; when treating neuropathologies including dementia, chronic stroke, neuropathic pain or traumatic brain injury: Role of TNF in neuropathologies and in particular in Alzheimer's disease (Part II)

No abstract is available for this paper.

• Publication year

  2015

• Venue

  Healthy Aging Research

• Publication date

  Unknown publication date

• Fields of study

  Medicine

• Identifiers
  DOI 10.12715/har.2015.4.14
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar

• No claims are published for this paper.

• No concepts are published for this paper.

• Dysfunctionally phosphorylated type 1 insulin receptor substrate in neural‐derived blood exosomes of preclinical Alzheimer's disease

  2015cited by this paper
• TNF and TNF-receptors: From mediators of cell death and inflammation to therapeutic giants - past, present and future.

  2014cited by this paper
• Plasma phospholipids identify antecedent memory impairment in older adults

  2014cited by this paper
• Studies of selective TNF inhibitors in the treatment of brain injury from stroke and trauma: a review of the evidence to date

  2014cited by this paper
• Immediate Neurological Recovery Following Perispinal Etanercept Years After Brain Injury

  2014cited by this paper
• TNF-α mediates PKR-dependent memory impairment and brain IRS-1 inhibition induced by Alzheimer's β-amyloid oligomers in mice and monkeys.

  2013cited by this paper
• Soluble oligomers and fibrillar species of amyloid β-peptide differentially affect cognitive functions and hippocampal inflammatory response.

  2012cited by this paper
• Clinical and biomarker changes in dominantly inherited Alzheimer's disease.

  2012cited by this paper
• Deciphering the physiology underlying the rapid clinical effects of perispinal etanercept in Alzheimer's disease.

  2012cited by this paper
• Amyloid in neurodegenerative diseases: friend or foe?

  2011cited by this paper
• Rapid Improvement of Chronic Stroke Deficits after Perispinal Etanercept

  2011cited by this paper
• Effects of acute stress and GluN2B-containing NMDA receptor antagonism on object and object-place recognition memory.

  2010cited by this paper
• The roles of TNF in brain dysfunction and disease.

  2010cited by this paper
• Tumour necrosis factor alpha induces rapid reduction in AMPA receptor‐mediated calcium entry in motor neurones by increasing cell surface expression of the GluR2 subunit: relevance to neurodegeneration

  2010cited by this paper
• Alzheimer’s Disease Amyloid β-Protein and Synaptic Function

  2010cited by this paper
• Inhibition of soluble TNF signaling in a mouse model of Alzheimer's disease prevents pre-plaque amyloid-associated neuropathology.

  2009cited by this paper
• Amyloid deposits: Protection against toxic protein species?

  2009cited by this paper
• Rapid intracerebroventricular delivery of Cu-DOTA-etanercept after peripheral administration demonstrated by PET imaging

  2009cited by this paper
• GluN2B subunit-containing NMDA receptor antagonists prevent Aβ-mediated synaptic plasticity disruption in vivo

  2009cited by this paper
• Rapid Tumor Necrosis Factor α-Induced Exocytosis of Glutamate Receptor 2-Lacking AMPA Receptors to Extrasynaptic Plasma Membrane Potentiates Excitotoxicity

  2008cited by this paper
• Rapid improvement in verbal fluency and aphasia following perispinal etanercept in Alzheimer's disease

  2008cited by this paper
• Perispinal etanercept produces rapid improvement in primary progressive aphasia: identification of a novel, rapidly reversible TNF-mediated pathophysiologic mechanism.

  2008cited by this paper
• Rapid cognitive improvement in Alzheimer's disease following perispinal etanercept administration

  2008cited by this paper
• Synaptic memory mechanisms: Alzheimer's disease amyloid beta-peptide-induced dysfunction.

  2007cited by this paper
• Deletion of tumor necrosis factor death receptor inhibits amyloid β generation and prevents learning and memory deficits in Alzheimer's mice

  2007cited by this paper
• Interferon-gamma and tumor necrosis factor-alpha regulate amyloid-beta plaque deposition and beta-secretase expression in Swedish mutant APP transgenic mice.

  2007cited by this paper
• Deletion of tumor necrosis factor death receptor inhibits amyloid beta generation and prevents learning and memory deficits in Alzheimer's mice.

  2007cited by this paper
• Synaptic scaling mediated by glial TNF-alpha.

  2006cited by this paper
• Synaptic scaling mediated by glial TNF-α

  2006cited by this paper
• TNF alpha potentiates glutamate neurotoxicity by inhibiting glutamate uptake in organotypic brain slice cultures: neuroprotection by NF kappa B inhibition.

  2005cited by this paper
• Beta-amyloid inhibition of long-term potentiation is mediated via tumor necrosis factor.

  2005cited by this paper
• Differential Regulation of AMPA Receptor and GABA Receptor Trafficking by Tumor Necrosis Factor-α

  2005cited by this paper
• TNFα potentiates glutamate neurotoxicity by inhibiting glutamate uptake in organotypic brain slice cultures: neuroprotection by NFκB inhibition

  2005cited by this paper
• β‐amyloid inhibition of long‐term potentiation is mediated via tumor necrosis factor

  2005cited by this paper
• β-Amyloid-Mediated Inhibition of NMDA Receptor-Dependent Long-Term Potentiation Induction Involves Activation of Microglia and Stimulation of Inducible Nitric Oxide Synthase and Superoxide

  2004cited by this paper
• Soluble Arctic amyloid β protein inhibits hippocampal long‐term potentiation in vivo

  2004cited by this paper
• Block of Long-Term Potentiation by Naturally Secreted and Synthetic Amyloid β-Peptide in Hippocampal Slices Is Mediated via Activation of the Kinases c-Jun N-Terminal Kinase, Cyclin-Dependent Kinase 5, and p38 Mitogen-Activated Protein Kinase as well as Metabotropic Glutamate Receptor Type 5

  2004cited by this paper
• NMDA receptor regulation by amyloid-β does not account for its inhibition of LTP in rat hippocampus

  2003cited by this paper
• Role of cytokines in the gene expression of amyloid β–protein precursor: Identification of a 5'-UTR-Binding nuclear factor and its implications in Alzheimer's disease

  2003cited by this paper
• A Proximal Gene Promoter Region for the β‐Amyloid Precursor Protein Provides a Link between Development, Apoptosis, and Alzheimer's Disease

  2003cited by this paper
• Naturally secreted oligomers of amyloid β protein potently inhibit hippocampal long-term potentiation in vivo

  2002cited by this paper
• Regulation of Promoter Activity of the APP Gene by Cytokines and Growth Factors

  2002cited by this paper
• Amyloid-beta oligomers: their production, toxicity and therapeutic inhibition.

  2002cited by this paper
• LIGHT, a new member of the TNF superfamily.

  2002cited by this paper
• Amyloid β-peptide inhibition of the PKA/CREB pathway and long-term potentiation: Reversibility by drugs that enhance cAMP signaling

  2002cited by this paper
• Neurodegenerative and Neuroprotective Effects of Tumor Necrosis Factor (TNF) in Retinal Ischemia: Opposite Roles of TNF Receptor 1 and TNF Receptor 2

  2002cited by this paper
• Amyloid-β oligomers: their production, toxicity and therapeutic inhibition

  2001cited by this paper
• TNF‐α stimulates caspase‐3 activation and apoptotic cell death in primary septo‐hippocampal cultures

  2001cited by this paper
• The role of TNF and its receptors in Alzheimer's disease.

  2001cited by this paper
• Cytokines and the central nervous system.

  2001cited by this paper
• Generation of Aggregated β-Amyloid in the Rat Hippocampus Impairs Synaptic Transmission and Plasticity and Causes Memory Deficits

  2001cited by this paper
• Use-Dependent Effects of Amyloidogenic Fragments of β-Amyloid Precursor Protein on Synaptic Plasticity in Rat Hippocampus In Vivo

  2001cited by this paper
• Impairment of hippocampal long-term potentiation by Alzheimer amyloid beta-peptides.

  2000cited by this paper
• Impairment of hippocampal long‐term potentiation by Alzheimer amyloid β‐peptides

  2000cited by this paper
• Cell mediators of inflammation in the Alzheimer disease brain.

  2000cited by this paper
• Dual role of tumor necrosis factor alpha in brain injury.

  1999cited by this paper
• Impairments of long-term potentiation in hippocampal slices of beta-amyloid-infused rats.

  1999cited by this paper
• Diffusible, nonfibrillar ligands derived from Abeta1-42 are potent central nervous system neurotoxins.

  1998cited by this paper
• Block of LTP in rat hippocampus in vivo by beta-amyloid precursor protein fragments.

  1997cited by this paper
• Long-term potentiation and memory.

  1987cited by this paper

• No citing papers are available for this paper.