A cis‐eQTL genetic variant in PLK4 confers high risk of hepatocellular carcinoma

The overexpression and knockdown of PLK4 were both reported to generate aneuploidy. Thus, we aimed to investigate whether genetic variants in PLK4 contribute to the development of hepatocellular carcinoma (HCC).

• Publication year

  2019

• Venue

  Cancer Medicine

• Publication date

  2019-09-06

• Fields of study

  Biology, Medicine

• Identifiers
  DOI 10.1002/cam4.2487PMID 31489978PMCID 6797585
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

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