Mitophagy: mechanisms, pathophysiological roles, and analysis

No abstract is available for this paper.

• Publication year

  2012

• Venue

  Biological chemistry

• Publication date

  2012-07-01

• Fields of study

  Biology, Medicine

• Identifiers
  DOI 10.1515/hsz-2012-0119PMID 22944659PMCID PMC3630798
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

• p53-TIGAR axis attenuates mitophagy to exacerbate cardiac damage after ischemia.

  2012cited by this paper
• p53-TIGAR axis attenuates mitophagy to exacerbate cardiac damage after ischemia

  2012cited by this paper
• Role of PINK1 binding to the TOM complex and alternate intracellular membranes in recruitment and activation of the E3 ligase Parkin.

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• Activation of autophagy protects against acetaminophen‐induced hepatotoxicity

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• Structures containing Atg9A and the ULK1 complex independently target depolarized mitochondria at initial stages of Parkin-mediated mitophagy

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• Mitochondrial outer-membrane protein FUNDC1 mediates hypoxia-induced mitophagy in mammalian cells

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• ER Tubules Mark Sites of Mitochondrial Division

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  2011cited by this paper
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• Phosphorylation of Serine 114 on Atg32 mediates mitophagy

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• Parkin Mediates Proteasome-dependent Protein Degradation and Rupture of the Outer Mitochondrial Membrane*♦

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• Two MAPK-signaling pathways are required for mitophagy in Saccharomyces cerevisiae

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• TBK1 Mediates Crosstalk Between the Innate Immune Response and Autophagy

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• During autophagy mitochondria elongate, are spared from degradation and sustain cell viability

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• Postfertilization Autophagy of Sperm Organelles Prevents Paternal Mitochondrial DNA Transmission

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• High-mobility group box 1 is essential for mitochondrial quality control.

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• The emerging role of autophagy in alcoholic liver disease

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• Parkin Interacts with Ambra1 to Induce Mitophagy

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• Mitochondria and the Autophagy–Inflammation–Cell Death Axis in Organismal Aging

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• Nix is a selective autophagy receptor for mitochondrial clearance

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• Can autophagy promote longevity?

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• Mitofusin 1 and mitofusin 2 are ubiquitinated in a PINK1/parkin-dependent manner upon induction of mitophagy.

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• Endogenous HMGB1 regulates autophagy

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• Proteasome and p97 mediate mitophagy and degradation of mitofusins induced by Parkin

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• Mitochondrial membrane potential regulates PINK1 import and proteolytic destabilization by PARL

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• p62/SQSTM1 is required for Parkin-induced mitochondrial clustering but not mitophagy; VDAC1 is dispensable for both

  2010cited by this paper
• The autophagosomal protein LGG-2 acts synergistically with LGG-1 in dauer formation and longevity in C. elegans

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• Nix Is Critical to Two Distinct Phases of Mitophagy, Reactive Oxygen Species-mediated Autophagy Induction and Parkin-Ubiquitin-p62-mediated Mitochondrial Priming*

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• Mff is an essential factor for mitochondrial recruitment of Drp1 during mitochondrial fission in mammalian cells

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• p62/SQSTM1 cooperates with Parkin for perinuclear clustering of depolarized mitochondria

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• Nix directly binds to GABARAP: A possible crosstalk between apoptosis and autophagy

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  2009cited by this paper
• Hypoxia-Induced Autophagy Is Mediated through Hypoxia-Inducible Factor Induction of BNIP3 and BNIP3L via Their BH3 Domains

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• Glutathione Participates in the Regulation of Mitophagy in Yeast*

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  2009cited by this paper
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  2009cited by this paper
• Mitochondria-anchored receptor Atg32 mediates degradation of mitochondria via selective autophagy.

  2009influential reference
• Dynamics and diversity in autophagy mechanisms: lessons from yeast

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• Coenzyme Q deficiency triggers mitochondria degradation by mitophagy

  2009cited by this paper
• Viruses and autophagy

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  2009influential reference
• Mitophagy.

  2009cited by this paper
• Parkin is recruited selectively to impaired mitochondria and promotes their autophagy

  2008influential reference
• Restoration of chaperone-mediated autophagy in aging liver improves cellular maintenance and hepatic function

  2008cited by this paper
• High levels of Fis1, a pro-fission mitochondrial protein, trigger autophagy.

  2008influential reference
• The kinase domain of mitochondrial PINK1 faces the cytoplasm

  2008influential reference
• Akt inhibition promotes autophagy and sensitizes PTEN-null tumors to lysosomotropic agents

  2008cited by this paper
• Hypoxia induces autophagic cell death in apoptosis-competent cells through a mechanism involving BNIP3

  2008cited by this paper
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  2008influential reference
• PINK1 controls mitochondrial localization of Parkin through direct phosphorylation.

  2008cited by this paper
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  2008influential reference
• Fission and selective fusion govern mitochondrial segregation and elimination by autophagy

  2008cited by this paper
• Mature ribosomes are selectively degraded upon starvation by an autophagy pathway requiring the Ubp3p/Bre5p ubiquitin protease

  2008cited by this paper
• Sorting, recognition and activation of the misfolded protein degradation pathways through macroautophagy and the proteasome

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• Mitochondrial Autophagy Is an HIF-1-dependent Adaptive Metabolic Response to Hypoxia*

  2008influential reference
• Autophagy protects renal tubular cells against cyclosporine toxicity

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  2008cited by this paper
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  2007cited by this paper
• FoxO3 controls autophagy in skeletal muscle in vivo.

  2007cited by this paper
• Homeostatic levels of p62 control cytoplasmic inclusion body formation in autophagy-deficient mice.

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  2007influential reference
• Hsp90 Inhibition Decreases Mitochondrial Protein Turnover

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• Inhibition of mitochondrial complex IV leads to secondary loss complex II-III activity: implications for the pathogenesis and treatment of mitochondrial encephalomyopathies.

  2007cited by this paper
• Aup 1 p , a Yeast Mitochondrial Protein Phosphatase Homolog , Is Required for Efficient Stationary Phase Mitophagy and Cell Survival *

  2007cited by this paper
• Selective degradation of mitochondria by mitophagy.

  2007influential reference

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