Alterations of choline acetyltransferase, phosphoinositide hydrolysis, and cytoskeletal proteins in rat brain in response to colchicine administration

No abstract is available for this paper.

• Publication year

  1992

• Venue

  Experimental Brain Research

• Publication date

  1992-06-01

• Fields of study

  Biology, Medicine, Chemistry

• Identifiers
  DOI 10.1007/BF00229873PMID 1322821
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

• Degradation of Microtubule‐Associated Protein 2 and Brain Spectrin by Calpain: A Comparative Study

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  1990cited by this paper
• Neurochemical recovery in the neocortex after colchicine lesions of the nucleus basalis magnocellularis in rats.

  1990cited by this paper
• Autoradiographic imaging of phosphoinositide turnover in the brain.

  1990cited by this paper
• Cholinergic cell loss and cognitive impairments following intraventricular or intradentate injection of colchicine.

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  1989cited by this paper
• Intrahippocampal colchicine injection results in spectrin proteolysis.

  1989cited by this paper
• Colchicine-induced alterations in receptor-stimulated phosphoinositide hydrolysis in the rat hippocampus.

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• Stimulation of NMDA receptors induces proteolysis of spectrin in hippocampus.

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• Colchicine as an investigative tool in neurobiology.

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• Colchicine Neurotoxicity Demonstrates the Cholinergic Projection from the Supracommissural Septum to the Habenula and the Nucleus Interpeduncularis in the Rat

  1984cited by this paper
• Neurotoxicity of colchicine and other tubulin-binding agents: a selective vulnerability of certain neurons to the disruption of microtubules.

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• Lithium amplifies agonist-dependent phosphatidylinositol responses in brain and salivary glands.

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