Jaks, STATs, cytokine signal transduction, and immunoregulation: are we there yet?

No abstract is available for this paper.

• Publication year

  1997

• Venue

  Immunity

• Publication date

  1997-07-01

• Fields of study

  Biology, Medicine

• Identifiers
  DOI 10.1016/S1074-7613(00)80505-1PMID 9252115
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

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• STAM, signal transducing adaptor molecule, is associated with Janus kinases and involved in signaling for cell growth and c-myc induction.

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• Peripheral Expression of Jak3 Is Required to Maintain T Lymphocyte Function

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• Targeted disruption of the mouse Stat3 gene leads to early embryonic lethality.

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• Interleukin-2 induces tyrosine phosphorylation of SHP-2 through IL-2 receptor β chain

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• Mutation in the Jak kinase JH2 domain hyperactivates Drosophila and mammalian Jak-Stat pathways

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• A JAK-STAT pathway regulates wing vein formation in Drosophila.

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• Activation and association of Stat3 with Src in v-Src-transformed cell lines

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• IL-12-Deficient Mice Are Defective in IFNγ Production and Type 1 Cytokine Responses

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• P210 and P190BCR/ABL Induce the Tyrosine Phosphorylation and DNA Binding Activity of Multiple Specific STAT Family Members*

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• Jak-STAT signaling induced by the v-abl oncogene.

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