Loss of C. elegans BBS-7 and BBS-8 protein function results in cilia defects and compromised intraflagellar transport.

O. Blacque,Michael J. Reardon,Chunmei Li,Jonathan McCarthy,M. Mahjoub,S. J. Ansley,José L. Badano,A. Mah,P. Beales,W. Davidson,R. Johnsen,M. Audeh,R. Plasterk,D. Baillie,N. Katsanis,L. Quarmby,S. R. Wicks,M. Leroux

Published 2004 in Genes & Development

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