Replication of ICP0-Null Mutant Herpes Simplex Virus Type 1 Is Restricted by both PML and Sp100

No abstract is available for this paper.

• Publication year

  2007

• Venue

  Journal of Virology

• Publication date

  2007-12-26

• Fields of study

  Biology, Medicine

• Identifiers
  DOI 10.1128/JVI.02308-07PMID 18160441PMCID PMC2258993
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

• Nuclear Domain 10 Components Promyelocytic Leukemia Protein and hDaxx Independently Contribute to an Intrinsic Antiviral Defense against Human Cytomegalovirus Infection

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• Repressor element-1 silencing transcription factor/neuronal restrictive silencer factor (REST/NRSF) can regulate HSV-1 immediate-early transcription via histone modification

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• PML Contributes to a Cellular Mechanism of Repression of Herpes Simplex Virus Type 1 Infection That Is Inactivated by ICP0

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• Human Daxx-mediated Repression of Human Cytomegalovirus Gene Expression Correlates with a Repressive Chromatin Structure around the Major Immediate Early Promoter*

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  2006influential reference
• Evidence for a Role of the Cellular ND10 Protein PML in Mediating Intrinsic Immunity against Human Cytomegalovirus Infections

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• ICP0 and the US3 protein kinase of herpes simplex virus 1 independently block histone deacetylation to enable gene expression.

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• Mediation of Epstein–Barr virus EBNA‐LP transcriptional coactivation by Sp100

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  2005cited by this paper
• Histone deacetylase inhibitors induce reactivation of herpes simplex virus type 1 in a latency-associated transcript-independent manner in neuronal cells

  2005cited by this paper
• Components of the REST/CoREST/histone deacetylase repressor complex are disrupted, modified, and translocated in HSV-1-infected cells.

  2005cited by this paper
• SP100B is a repressor of gene expression

  2005cited by this paper
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• Formation of Nuclear Foci of the Herpes Simplex Virus Type 1 Regulatory Protein ICP4 at Early Times of Infection: Localization, Dynamics, Recruitment of ICP27, and Evidence for the De Novo Induction of ND10-Like Complexes

  2004cited by this paper
• Specific Histone Tail Modification and Not DNA Methylation Is a Determinant of Herpes Simplex Virus Type 1 Latent Gene Expression

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• Functional Interaction between Class II Histone Deacetylases and ICP0 of Herpes Simplex Virus Type 1

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  2004cited by this paper
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• Infection of a human hepatoma cell line by hepatitis B virus

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  2001cited by this paper
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  2000cited by this paper
• Role of SUMO-1-modified PML in nuclear body formation.

  2000cited by this paper
• ICP0 Induces the Accumulation of Colocalizing Conjugated Ubiquitin

  2000cited by this paper
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  2000cited by this paper
• The Nuclear Dot Protein Sp100, Characterization of Domains Necessary for Dimerization, Subcellular Localization, and Modification by Small Ubiquitin-like Modifiers*

  1999cited by this paper
• Splice variants of the nuclear dot-associated Sp100 protein contain homologies to HMG-1 and a human nuclear phosphoprotein-box motif.

  1999cited by this paper
• Herpes virus induced proteasome-dependent degradation of the nuclear bodies-associated PML and Sp100 proteins

  1999cited by this paper
• Human Daxx regulates Fas‐induced apoptosis from nuclear PML oncogenic domains (PODs)

  1999cited by this paper
• Pml Is Critical for Nd10 Formation and Recruits the Pml-Interacting Protein Daxx to This Nuclear Structure When Modified by Sumo-1

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• Copyright © 1997, American Society for Microbiology Experimental Investigation of Herpes Simplex Virus Latency†

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