Assessment of the role of activator protein-1 on transcription of the mouse steroidogenic acute regulatory protein gene.

No abstract is available for this paper.

• Publication year

  2004

• Venue

  Molecular Endocrinology

• Publication date

  2004-03-01

• Fields of study

  Biology, Medicine

• Identifiers
  DOI 10.1210/ME.2003-0223PMID 14673133
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

• Role of the Fos family members, c-Fos, Fra-1 and Fra-2, in the regulation of cell motility

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• Sp1 and SF-1 interact and cooperate in the regulation of human steroidogenic acute regulatory protein gene expression.

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• Printed in U.S.A. Copyright © 2001 by The Endocrine Society Assessment of Mechanisms of Thyroid Hormone Action in Mouse Leydig Cells: Regulation of the Steroidogenic Acute Regulatory Protein, Steroidogenesis, and Luteinizing Hormone Receptor Function*

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• CCAAT/Enhancer-binding Proteins Regulate Expression of the Human Steroidogenic Acute Regulatory Protein (StAR) Gene*

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• Homologous regulation of the gonadotropin-releasing hormone receptor gene is partially mediated by protein kinase C activation of an activator protein-1 element.

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• CHOP/GADD153 and methionyl-tRNA synthetase (MetRS) genes overlap in a conserved region that controls mRNA stability.

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• SF-1 (Steroidogenic Factor-1) and C/EBPβ (CCAAT/Enhancer Binding Protein-β) Cooperate to Regulate the Murine StAR (Steroidogenic Acute Regulatory) Promoter

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• Transcriptional Regulation of the Rat Steroidogenic Acute Regulatory Protein Gene by Steroidogenic Factor 1* * This work was supported by NIH Grant R01-HD35163 (to M.P.M.).

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