Non-lipid-related effects of statins

No abstract is available for this paper.

• Publication year

  2000

• Venue

  Annals medicus

• Publication date

  2000-01-01

• Fields of study

  Biology, Medicine

• Identifiers
  DOI 10.3109/07853890008998823PMID 10821323
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

• Reduced platelet aggregation after fluvastatin therapy is associated with altered platelet lipid composition and drug binding to the platelets.

  2003cited by this paper
• Lipid metabolism.

  2002cited by this paper
• Farnesyltransferase inhibitors.

  2001cited by this paper
• Improvement of nitric oxide-dependent vasodilatation by HMG-CoA reductase inhibitors through attenuation of endothelial superoxide anion formation.

  2000cited by this paper
• Inhibition of thrombin generation by simvastatin and lack of additive effects of aspirin in patients with marked hypercholesterolemia.

  1999cited by this paper
• Alendronate mechanism of action: geranylgeraniol, an intermediate in the mevalonate pathway, prevents inhibition of osteoclast formation, bone resorption, and kinase activation in vitro.

  1999cited by this paper
• Lowering LDL cholesterol: questions from recent meta-analyses and subset analyses of clinical trial DataIssues from the Interdisciplinary Council on Reducing the Risk for Coronary Heart Disease, ninth Council meeting.

  1999cited by this paper
• A randomized placebo-controlled trial of fluvastatin for prevention of restenosis after successful coronary balloon angioplasty; final results of the fluvastatin angiographic restenosis (FLARE) trial.

  1999cited by this paper
• Atherosclerosis--an inflammatory disease.

  1999cited by this paper
• Fluvastatin reduces endothelin secretion of cultured human umbilical vein endothelial cells

  1999cited by this paper
• Statins and monocytes.

  1999cited by this paper
• Improvement in coronary flow reserve determined by positron emission tomography after 6 months of cholesterol-lowering therapy in patients with early stages of coronary atherosclerosis.

  1999cited by this paper
• Lovastatin-Induced Proliferation Inhibition and Apoptosis in C 6 Glial Cells 1

  1999cited by this paper
• Lovastatin decreases the receptor-mediated degradation of acetylated and oxidized LDLs in human blood monocytes during the early stage of differentiation into macrophages.

  1999cited by this paper
• The effect of fluvastatin on cardiac events in patients with symptomatic coronary artery disease during one year of treatment.

  1999cited by this paper
• Pravastatin sodium activates endothelial nitric oxide synthase independent of its cholesterol-lowering actions.

  1999cited by this paper
• Inhibition by simvastatin, but not pravastatin, of glucose‐induced cytosolic Ca2+ signalling and insulin secretion due to blockade of L‐type Ca2+ channels in rat islet β‐cells

  1999cited by this paper
• Lovastatin-induced proliferation inhibition and apoptosis in C6 glial cells.

  1999cited by this paper
• Protective effect of fluvastatin sodium (XU-62-320), a 3-hydroxy-3-methylglutaryl coenzyme A (HMG-CoA) reductase inhibitor, on oxidative modification of human low-density lipoprotein in vitro.

  1999cited by this paper
• Pravastatin therapy in hyperlipidemia: effects on thrombus formation and the systemic hemostatic profile.

  1999cited by this paper
• Stimulation of bone formation in vitro and in rodents by statins.

  1999cited by this paper
• Delayed response of myocardial flow reserve to lipid-lowering therapy with fluvastatin.

  1999cited by this paper
• Effect of cerivastatin sodium, a new inhibitor of HMG‐CoA reductase, on plasma lipid levels, progression of atherosclerosis, and the lesional composition in the plaques of WHHL rabbits

  1999cited by this paper
• New insights into the pharmacodynamic and pharmacokinetic properties of statins.

  1999cited by this paper
• Aggressive lipid-lowering therapy compared with angioplasty in stable coronary artery disease. Atorvastatin versus Revascularization Treatment Investigators.

  1999cited by this paper
• [HMG-CoA reductase inhibitors].

  1998cited by this paper
• Effects of the 3-hydroxy-3-methylglutaryl-CoA reductase inhibitors, atorvastatin and simvastatin, on the expression of endothelin-1 and endothelial nitric oxide synthase in vascular endothelial cells.

  1998cited by this paper
• HMG-CoA reductase inhibitors reduce MMP-9 secretion by macrophages.

  1998cited by this paper
• Atorvastatin and gemfibrozil metabolites, but not the parent drugs, are potent antioxidants against lipoprotein oxidation.

  1998cited by this paper
• Interactions of platelets, macrophages, and lipoproteins in hypercholesterolemia: antiatherogenic effects of HMG-CoA reductase inhibitor therapy.

  1998cited by this paper
• A phase I-II trial of lovastatin for anaplastic astrocytoma and glioblastoma multiforme.

  1998cited by this paper
• 3-Hydroxy-3-methylglutaryl coenzyme a reductase and isoprenylation inhibitors induce apoptosis of vascular smooth muscle cells in culture.

  1998cited by this paper
• Upregulation of endothelial nitric oxide synthase by HMG CoA reductase inhibitors.

  1998cited by this paper
• Pravastatin has cholesterol-lowering independent effects on the artery wall of atherosclerotic monkeys.

  1998cited by this paper
• Primary prevention of acute coronary events with lovastatin in men and women with average cholesterol levels: results of AFCAPS/TexCAPS. Air Force/Texas Coronary Atherosclerosis Prevention Study.

  1998cited by this paper
• Evidence for a role of osteopontin in macrophage infiltration in response to pathological stimuli in vivo.

  1998cited by this paper
• Antiatherothrombotic properties of statins: implications for cardiovascular event reduction.

  1998cited by this paper
• Stroke protection by 3-hydroxy-3-methylglutaryl (HMG)-CoA reductase inhibitors mediated by endothelial nitric oxide synthase.

  1998cited by this paper
• Lovastatin modulates in vivo and in vitro the plasminogen activator/plasmin system of rat proximal tubular cells: role of geranylgeranylation and Rho proteins.

  1998cited by this paper
• Prevention of cardiovascular events and death with pravastatin in patients with coronary heart disease and a broad range of initial cholesterol levels.

  1998cited by this paper
• HMG-CoA reductase inhibition by atorvastatin reduces neointimal inflammation in a rabbit model of atherosclerosis.

  1998cited by this paper
• 3-Hydroxy-3-methylglutaryl coenzyme A reductase inhibitors increase fibrinolytic activity in rat aortic endothelial cells. Role of geranylgeranylation and Rho proteins.

  1998cited by this paper
• Differential effects of lovastatin on the trafficking of endogenous and lipoprotein-derived cholesterol in human monocyte-derived macrophages.

  1998cited by this paper
• A comparison of the effect of the 3-hydroxy-3-methylglutaryl coenzyme A (HMG-CoA) reductase inhibitors simvastatin, lovastatin and pravastatin on leukaemic and normal bone marrow progenitors.

  1997cited by this paper
• Pharmacological control of the mevalonate pathway: effect on arterial smooth muscle cell proliferation.

  1997cited by this paper
• Induction of cytokine expression in leukocytes by binding of thrombin-stimulated platelets.

  1997cited by this paper
• Suppression of neointimal thickening by a newly developed HMG‐CoA reductase inhibitor, BAYw6228, and its inhibitory effect on vascular smooth muscle cell growth

  1997cited by this paper
• Proapoptotic effect of atorvastatin on stimulated rabbit smooth muscle cells.

  1997cited by this paper
• Inhibition of 3-Hydroxy-3-methylglutaryl (HMG)-CoA Reductase Blocks Hypoxia-mediated Down-regulation of Endothelial Nitric Oxide Synthase*

  1997cited by this paper
• Vastatins inhibit tissue factor in cultured human macrophages. A novel mechanism of protection against atherothrombosis.

  1997cited by this paper
• Simvastatin, an HMG-coenzyme A reductase inhibitor, improves endothelial function within 1 month.

  1997cited by this paper
• HMG-CoA reductase inhibitors suppress macrophage growth induced by oxidized low density lipoprotein.

  1997cited by this paper
• Effects of fluvastatin on leukocyte-endothelial cell adhesion in hypercholesterolemic rats.

  1997cited by this paper
• Direct vascular effects of HMG-CoA reductase inhibitors.

  1997cited by this paper
• Effect of pravastatin on angiographic restenosis after coronary balloon angioplasty. The PREDICT Trial Investigators. Prevention of Restenosis by Elisor after Transluminal Coronary Angioplasty.

  1997cited by this paper
• HMG-CoA reductase inhibitors decrease CD11b expression and CD11b-dependent adhesion of monocytes to endothelium and reduce increased adhesiveness of monocytes isolated from patients with hypercholesterolemia.

  1997cited by this paper
• Effects of fluvastatin on coronary atherosclerosis in patients with mild to moderate cholesterol elevations (Lipoprotein and Coronary Atherosclerosis Study [LCAS]).

  1997cited by this paper
• Reduced susceptibility of low density lipoprotein (LDL) to lipid peroxidation after fluvastatin therapy is associated with the hypocholesterolemic effect of the drug and its binding to the LDL.

  1997cited by this paper
• Infection, inflammation, and infarction: does acute endothelial dysfunction provide a link?

  1997cited by this paper
• Mevalonate-dependent inhibition of transendothelial migration and chemotaxis of human peripheral blood neutrophils by pravastatin.

  1997cited by this paper
• Antioxidants and atherosclerotic heart disease.

  1997cited by this paper
• Mevalonate modulation of cell proliferation and apoptosis

  1997cited by this paper
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  1997cited by this paper
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  1996cited by this paper
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  1996cited by this paper
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  1996cited by this paper
• Oral L-arginine improves endothelium-dependent dilation in hypercholesterolemic young adults.

  1996cited by this paper
• Inhibitory effect of fluvastatin, an HMG-CoA reductase inhibitor, on the expression of adhesion molecules on human monocyte cell line.

  1996cited by this paper
• The effect of pravastatin on coronary events after myocardial infarction in patients with average cholesterol levels. Cholesterol and Recurrent Events Trial investigators.

  1996cited by this paper
• Farnesyl analogues inhibit vasoconstriction in animal and human arteries.

  1996cited by this paper
• Inhibitory effect of fluvastatin at doses insufficient to lower serum lipids on the catheter-induced thickening of intima in rabbit femoral artery.

  1996cited by this paper
• Contrary effects of lightly and strongly oxidized LDL with potent promotion of growth versus apoptosis on arterial smooth muscle cells, macrophages, and fibroblasts.

  1996cited by this paper
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  1996cited by this paper
• Lipid Metabolism as a Target for Brain Cancer Therapy: Synergistic Activity of Lovastatin and Sodium Phenylacetate Against Human Glioma Cells

  1996cited by this paper
• Inhibitor of proliferation of arterial smooth-muscle cells by fluvastatin.

  1996cited by this paper
• Non-lipid-related effects of 3-hydroxy-3-methylglutaryl coenzyme A reductase inhibitors.

  1996cited by this paper
• Lovastatin reduces expression of the combined adhesion and scavenger receptor CD36 in human monocytic cells.

  1996cited by this paper
• Intracellular Alkalinization Suppresses Lovastatin-induced Apoptosis in HL-60 Cells through the Inactivation of a pH-dependent Endonuclease (*)

  1995cited by this paper
• Improvement of myocardial perfusion by short-term fluvastatin therapy in coronary artery disease.

  1995cited by this paper
• Regulation of bovine endothelial constitutive nitric oxide synthase by oxygen.

  1995cited by this paper
• Matrix metalloproteinases and cardiovascular disease.

  1995cited by this paper
• Apoptosis participates in cellularity regulation during rat aortic intimal thickening.

  1995cited by this paper
• In vivo enhanced antitumor activity of carmustine [N,N'-bis(2-chloroethyl)-N-nitrosourea] by simvastatin.

  1995cited by this paper
• Evidence for apoptosis in human atherogenesis and in a rat vascular injury model.

  1995cited by this paper
• Reduction of serum cholesterol levels alters lesional composition of atherosclerotic plaques. Effect of pravastatin sodium on atherosclerosis in mature WHHL rabbits.

  1995cited by this paper
• Prevention of coronary heart disease with pravastatin in men with hypercholesterolemia. West of Scotland Coronary Prevention Study Group.

  1995cited by this paper
• Platelet cytosolic Ca2+ and membrane dynamics in patients with primary hypercholesterolemia. Effects of pravastatin.

  1995cited by this paper
• Pathogenesis of atherosclerosis and the role of 3-hydroxy-3-methylglutaryl coenzyme A reductase inhibitors.

  1995cited by this paper
• Inhibition of isoprenoid biosynthesis and arterial smooth-muscle cell proliferation.

  1995cited by this paper
• Prevention of coronary heart disease with pravastatin in men with hypercholesterolemia

  1995cited by this paper
• Effects of lipid lowering by pravastatin on progression and regression of coronary artery disease in symptomatic men with normal to moderately elevated serum cholesterol levels. The Regression Growth Evaluation Statin Study (REGRESS).

  1995cited by this paper
• Apoptosis in human atherosclerosis and restenosis.

  1995cited by this paper
• HMG-CoA reductase inhibitors reduce acetyl LDL endocytosis in mouse peritoneal macrophages.

  1995cited by this paper
• Coronary plaque disruption.

  1995cited by this paper
• Mevalonate availability affects human and rat resistance vessel function.

  1995cited by this paper
• Antiatherosclerotic activity of inhibitors of 3-hydroxy-3-methylglutaryl coenzyme A reductase in cholesterol-fed rabbits: a biochemical and morphological evaluation.

  1994cited by this paper
• Lack of effect of lovastatin on restenosis after coronary angioplasty. Lovastatin Restenosis Trial Study Group.

  1994cited by this paper
• Pathogenesis of atherosclerosis and the role of drug intervention

  1994cited by this paper
• Inhibition of Isoprenoid Biosynthesis and Arterial Smooth‐Muscle Cell Proliferation

  1994cited by this paper
• HMG CoA reductase inhibitors.

  1994cited by this paper

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