Two conserved domains in PCIF1 mediate interaction with pancreatic transcription factor PDX‐1

No abstract is available for this paper.

• Publication year

  2006

• Venue

  FEBS Letters

• Publication date

  2006-12-11

• Fields of study

  Biology, Medicine

• Identifiers
  DOI 10.1016/j.febslet.2006.11.021PMID 17126328
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

• Minireview: transcriptional regulation in pancreatic development.

  2005cited by this paper
• The nuclear receptor corepressor deacetylase activating domain is essential for repression by thyroid hormone receptor.

  2005cited by this paper
• Pdx-1 Interacts with Histone Deacetylases Hdac1 and Hdac-2 on Low Levels of Glucose

  2004cited by this paper
• Glucose regulation of insulin gene expression requires the recruitment of p300 by the beta-cell-specific transcription factor Pdx-1.

  2004cited by this paper
• Insulin promoter factor-1 mutations and diabetes in Trinidad: identification of a novel diabetes-associated mutation (E224K) in an Indo-Trinidadian family.

  2004influential reference
• NLS-dependent nuclear localization of p120ctn is necessary to relieve Kaiso-mediated transcriptional repression

  2004cited by this paper
• Identification of PCIF1, a POZ Domain Protein That Inhibits PDX-1 (MODY4) Transcriptional Activity

  2004cited by this paper
• The Pancreatic Duodenal Homeobox-1 Protein (Pdx-1) Interacts with Histone Deacetylases Hdac-1 and Hdac-2 on Low Levels of Glucose*

  2004cited by this paper
• The role of pdx1 and HNF6 in proliferation and differentiation of endocrine precursors

  2004cited by this paper
• Nuclear speckles: a model for nuclear organelles

  2003cited by this paper
• MacroH2A1.2 binds the nuclear protein Spop.

  2002cited by this paper
• Functional consequences of mutations in the MODY4 gene (IPF1) and coexistence with MODY3 mutations

  2001cited by this paper
• Regulation of the Insulin Gene by Glucose: Stimulation of Trans-Activation Potency of Human PDX-1 N-Terminal Domain

  1999cited by this paper
• Defective mutations in the insulin promoter factor-1 (IPF-1) gene in late-onset type 2 diabetes mellitus.

  1999cited by this paper
• Identification of a novel nuclear speckle-type protein,SPOP(新規核speckle型タンパク質,SPOPの同定)

  1998cited by this paper
• Components of the SMRT Corepressor Complex Exhibit Distinctive Interactions with the POZ Domain Oncoproteins PLZF, PLZF-RARα, and BCL-6*

  1998cited by this paper
• Conservation of PDX-1 structure, function, and expression in zebrafish.

  1998cited by this paper
• The BCL-6 POZ domain and other POZ domains interact with the co-repressors N-CoR and SMRT

  1998influential reference
• Insulin promoter factor-1 gene mutation linked to early-onset type 2 diabetes mellitus directs expression of a dominant negative isoprotein.

  1998cited by this paper
• Histone deacetylase associated with mSin3A mediates repression by the acute promyelocytic leukemia-associated PLZF protein

  1998influential reference
• Functional characterization of the transactivation properties of the PDX-1 homeodomain protein

  1997cited by this paper
• Identification of a novel nuclear speckle‐type protein, SPOP

  1997cited by this paper
• Functional regions of the homeodomain protein IDX-1 required for transactivation of the rat somatostatin gene.

  1996cited by this paper
• IDX‐1: a new homeodomain transcription factor expressed in rat pancreatic islets and duodenum that transactivates the somatostatin gene.

  1994cited by this paper
• Insulin expression in pancreatic islet cells relies on cooperative interactions between the helix loop helix factor E47 and the homeobox factor STF-1.

  1994cited by this paper

• E3 ligase substrate adaptor SPOP fine-tunes the UPR of pancreatic β cells

  2024cites this paper
• PCIF1, the only methyltransferase of N6,2-O-dimethyladenosine

  2023cites this paper
• Journal Pre-proof insights into glucose-dependent transcriptional regulation by PDX1

  2022cites this paper
• Biophysical insights into glucose-dependent transcriptional regulation by PDX1

  2022cites this paper
• m6Am methyltransferase PCIF1 is essential for aggressiveness of gastric cancer cells by inhibiting TM9SF1 mRNA translation

  2022cites this paper
• SPOP in Cancer: Phenomena, Mechanisms and Its Role in Therapeutic Implications

  2022cites this paper
• Novel insights into the SPOP E3 ubiquitin ligase: From the regulation of molecular mechanisms to tumorigenesis.

  2022cites this paper
• Cell Cycle Regulation of the Pdx1 Transcription Factor in Developing Pancreas and Insulin-Producing β-Cells

  2021cites this paper
• Intrinsically disordered substrates dictate SPOP subnuclear localization and ubiquitination activity

  2021cites this paper
• The Effect of 12 Weeks Aerobic Training on PDX-1 and GLUT2 Gene Expression in the Pancreatic Tissue of Type 2 Diabetic Rats

  2020cites this paper
• SPOP inhibits mice pancreatic stellate cell activation by promoting FADD degradation in cerulein-induced chronic pancreatitis.

  2019cites this paper
• Protein Kinase CK2—A Putative Target for the Therapy of Diabetes Mellitus?

  2019influential citation
• The Structure of the SPOP-Pdx1 Interface Reveals Insights into the Phosphorylation-Dependent Binding Regulation.

  2019cites this paper
• Functional analysis of Cullin 3 E3 ligases in tumorigenesis.

  2018cites this paper
• Comprehensive Maturity Onset Diabetes of the Young (MODY) Gene Screening in Pregnant Women with Diabetes in India

  2017cites this paper
• Structural and functional characterization of the transcription factor Pdx1 and its interactions with DNA and proteins

  2015cites this paper
• Differential expression of speckled POZ protein, SPOP: Putative regulation by miR-145

  2014cites this paper
• Generating NMR chemical shift assignments of intrinsically disordered proteins using carbon-detected NMR methods.

  2014cites this paper
• Neutralizing IL-1 β induces β -cell survival by maintaining PDX1 nuclear localization

  2011cites this paper
• Induction of Pancreatic β-Cell–Like Cells from CD44+/CD105+ Human Amniotic Fluids via Epigenetic Regulation of the Pancreatic and Duodenal Homeobox Factor 1 Promoter

  2011cites this paper
• Neutralizing interleukin 1β (IL-1β) induces β-cell survival by maintaining PDX1 protein nuclear localization.

  2011cites this paper
• Neutralizing Interleukin-1β (IL-1β) Induces β-Cell Survival by Maintaining PDX1 Protein Nuclear Localization*

  2011cites this paper
• Pcif1 modulates Pdx1 protein stability and pancreatic β cell function and survival in mice.

  2010cites this paper
• The diabetes gene Pdx1 regulates the transcriptional network of pancreatic endocrine progenitor cells in mice.

  2009cites this paper
• The homeodomain-interacting protein kinase 2 regulates insulin promoter factor-1/pancreatic duodenal homeobox-1 transcriptional activity.

  2009cites this paper
• Glucose regulation of insulin gene expression in pancreatic beta-cells.

  2008cites this paper
• Coordinated Activation of the Nuclear Ubiquitin Ligase Cul3-SPOP by the Generation of Phosphatidylinositol 5-Phosphate*

  2008cites this paper