Mitochondrial permeability transition in CNS trauma: Cause or effect of neuronal cell death?

No abstract is available for this paper.

• Publication year

  2005

• Venue

  Journal of Neuroscience Research

• Publication date

  Unknown publication date

• Fields of study

  Biology, Medicine

• Identifiers
  DOI 10.1002/jnr.20292PMID 15573402
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

• Growth and senescence of Medicago truncatula cultured cells are associated with characteristic mitochondrial morphology.

  2006cited by this paper
• Brain‐derived respiring mitochondria exhibit homogeneous, complete and cyclosporin‐sensitive permeability transition

  2004cited by this paper
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  2004cited by this paper
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• Zn2+ Induces Permeability Transition Pore Opening and Release of Pro-apoptotic Peptides from Neuronal Mitochondria*

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  1997cited by this paper
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  1996cited by this paper
• Complexes between kinases, mitochondrial porin and adenylate translocator in rat brain resemble the permeability transition pore

  1996cited by this paper
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  1996cited by this paper
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  1995cited by this paper
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  1990cited by this paper
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