The involvement of FANCM, FANCI, and checkpoint proteins in the interstrand DNA crosslink repair pathway is conserved in C. elegans.

No abstract is available for this paper.

• Publication year

  2010

• Venue

  DNA Repair

• Publication date

  2010-04-04

• Fields of study

  Biology, Medicine

• Identifiers
  DOI 10.1016/j.dnarep.2009.12.018PMID 20075016
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

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  2009cited by this paper
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  2009cited by this paper
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  2009cited by this paper
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  2009cited by this paper
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  2009cited by this paper
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  2009cited by this paper
• The FANCM Ortholog Fml1 Promotes Recombination at Stalled Replication Forks and Limits Crossing Over during DNA Double-Strand Break Repair

  2008cited by this paper
• FANCM and FAAP24 function in ATR-mediated checkpoint signaling independently of the Fanconi anemia core complex.

  2008cited by this paper
• The Fanconi anemia protein FANCM can promote branch migration of Holliday junctions and replication forks.

  2008cited by this paper
• Mechanistic insight into site-restricted monoubiquitination of FANCD2 by Ube2t, FANCL, and FANCI.

  2008cited by this paper
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  2008cited by this paper
• FANCM of the Fanconi anemia core complex is required for both monoubiquitination and DNA repair.

  2008cited by this paper
• Loss of CHK1 function impedes DNA damage-induced FANCD2 monoubiquitination but normalizes the abnormal G2 arrest in Fanconi anemia.

  2008cited by this paper
• FANCI phosphorylation functions as a molecular switch to turn on the Fanconi anemia pathway

  2008cited by this paper
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  2008cited by this paper
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  2008cited by this paper
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  2007cited by this paper
• DOG-1 Is the Caenorhabditis elegans BRIP1/FANCJ Homologue and Functions in Interstrand Cross-Link Repair

  2007cited by this paper
• The Fanconi anemia pathway and ubiquitin

  2007cited by this paper
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  2007cited by this paper
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  2007cited by this paper
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  2007cited by this paper
• Emergence of a DNA-damage response network consisting of Fanconi anaemia and BRCA proteins

  2007cited by this paper
• Developmental stage- and DNA damage-specific functions of C. elegans FANCD2.

  2007cited by this paper
• Dicer‐related drh‐3 gene functions in germ‐line development by maintenance of chromosomal integrity in Caenorhabditis elegans

  2007cited by this paper
• Fanconi anemia is associated with a defect in the BRCA2 partner PALB2

  2007cited by this paper
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  2007cited by this paper
• Identification of FAAP24, a Fanconi anemia core complex protein that interacts with FANCM.

  2007cited by this paper
• C. elegans FANCD2 responds to replication stress and functions in interstrand cross-link repair.

  2006cited by this paper
• Functional proteomics reveals the biochemical niche of C. elegans DCR-1 in multiple small-RNA-mediated pathways.

  2006cited by this paper
• Control of BRCA2 cellular and clinical functions by a nuclear partner, PALB2.

  2006cited by this paper
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  2006cited by this paper
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  2005cited by this paper
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  2005cited by this paper
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  2005cited by this paper
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  2005cited by this paper
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  2005cited by this paper
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  2005cited by this paper
• The vertebrate Hef ortholog is a component of the Fanconi anemia tumor-suppressor pathway

  2005cited by this paper
• BACH1 is critical for homologous recombination and appears to be the Fanconi anemia gene product FANCJ.

  2005cited by this paper
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  2005cited by this paper
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  2004cited by this paper
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  2004cited by this paper
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  2004cited by this paper
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  2004cited by this paper
• Functional Interaction of Monoubiquitinated FANCD2 and BRCA2/FANCD1 in Chromatin

  2004cited by this paper
• A Multiprotein Nuclear Complex Connects Fanconi Anemia and Bloom Syndrome

  2003cited by this paper
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  2003cited by this paper
• Sensing DNA Damage Through ATRIP Recognition of RPA-ssDNA Complexes

  2003cited by this paper
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  2003cited by this paper
• The dsRNA binding protein RDE-4 interacts with RDE-1, DCR-1, and a DExH-box helicase to direct RNAi in C. elegans.

  2002cited by this paper
• S-phase-specific interaction of the Fanconi anemia protein, FANCD2, with BRCA1 and RAD51.

  2002cited by this paper
• Attenuation of the formation of DNA-repair foci containing RAD51 in Fanconi anaemia.

  2002cited by this paper
• The emerging genetic and molecular basis of Fanconi anaemia

  2001cited by this paper
• Interaction of the Fanconi anemia proteins and BRCA1 in a common pathway.

  2001cited by this paper
• Repair of DNA interstrand cross-links.

  2001cited by this paper
• Cell cycle checkpoint signaling through the ATM and ATR kinases.

  2001cited by this paper
• Chk1 is an essential kinase that is regulated by Atr and required for the G(2)/M DNA damage checkpoint.

  2000cited by this paper

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  2025cites this paper
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  2024cites this paper
• Clinical and translational advances in ovarian cancer therapy

  2023cites this paper
• Conserved function of Drosophila Fancd2 monoubiquitination in response to double-strand DNA breaks

  2022cites this paper
• Histone demethylase AMX-1 is necessary for proper sensitivity to interstrand crosslink DNA damage

  2021cites this paper
• A Replication stress biomarker is associated with response to gemcitabine versus combined gemcitabine and ATR inhibitor therapy in ovarian cancer

  2021cites this paper
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  2020cites this paper
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  2020cites this paper
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  2020cites this paper
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  2019cites this paper
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  2019cites this paper
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  2018cites this paper
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  2018influential citation
• Inhibition of non-homologous end joining in Fanconi Anemia cells results in rescue of survival after interstrand crosslinks but sensitization to replication associated double-strand breaks.

  2018cites this paper
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  2017cites this paper
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  2017cites this paper
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  2016cites this paper
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  2015cites this paper
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  2015cites this paper
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  2014cites this paper
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  2014cites this paper
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  2013cites this paper
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  2013cites this paper
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  2012cites this paper
• Disrupted Signaling through the Fanconi Anemia Pathway Leads to Dysfunctional Hematopoietic Stem Cell Biology: Underlying Mechanisms and Potential Therapeutic Strategies

  2012cites this paper
• Preventing nonhomologous end joining suppresses DNA repair defects of Fanconi anemia.

  2010cites this paper