Long term changes in NADPH-diaphorase reactivity in striatal and cortical neurons following experimental perinatal asphyxia: neuroprotective effects of hypothermia.

No abstract is available for this paper.

• Publication year

  1997

• Venue

  International Journal of Neuroscience

• Publication date

  Unknown publication date

• Fields of study

  Biology, Medicine, Chemistry

• Identifiers
  DOI 10.3109/00207459708988460PMID 9134444
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

• Neurology of the newborn.

  2013cited by this paper
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  1996cited by this paper
• Cerebral Cortical Dysplasia Associated with Pediatric Epilepsy. Review of Neuropathologic Features and Proposal for a Grading System

  1995cited by this paper
• Nitric oxide and synaptic function.

  1994cited by this paper
• Long-term effects of perinatal asphyxia on basal ganglia neurotransmitter systems studied with microdialysis in rat.

  1994cited by this paper
• Delayed and prolonged post-ischemic hypothermia is neuroprotective in the gerbil.

  1994cited by this paper
• Neurocircuitry of the basal ganglia studied by monitoring neurotransmitter release

  1994cited by this paper
• Sparing of NADPH-diaphorase striatal neurons in Parkinson's and Alzheimer's diseases.

  1994cited by this paper
• Neurons in rat hippocampus that synthesize nitric oxide

  1993cited by this paper
• Role of nitric oxide in NMDA-evoked release of [3H]-dopamine from striatal slices.

  1992cited by this paper
• Protective effects of brain hypothermia on behavior and histopathology following global cerebral ischemia in rats.

  1992cited by this paper
• Effect of Nitroprusside (Nitric Oxide) on Endogenous Dopamine Release from Rat Striatal Slices

  1992cited by this paper
• Neurons that say NO.

  1992cited by this paper
• Histochemical mapping of nitric oxide synthase in the rat brain.

  1992cited by this paper
• Neonatal asphyxia: vulnerability of basal ganglia, thalamus, and brainstem.

  1991cited by this paper
• Neuronal NADPH diaphorase is a nitric oxide synthase.

  1991cited by this paper
• Asphyctic lesion: proliferation of tyrosine hydroxylase-immunoreactive nerve cell bodies in the rat substantia nigra and functional changes in dopamine neurotransmission.

  1991cited by this paper
• Localization of Mn-superoxide dismutase (Mn-SOD) in cholinergic and somatostatin-containing neurons in the rat neostriatum.

  1991cited by this paper
• Selective sparing of NADPH‐diaphorase–somatostatin–neuropeptide Y neurons in ischemic gerbil striatum

  1990cited by this paper
• Conditions for pharmacologic evaluation in the gerbil model of forebrain ischemia.

  1989cited by this paper
• Postischemic moderate hypothermia inhibits CA1 hippocampal ischemic neuronal injury.

  1989cited by this paper
• Effect of Ischemia on the In Vivo Release of Striatal Dopamine, Glutamate, and γ‐Aminobutyric Acid Studied by Intracerebral Microdialysis

  1988cited by this paper
• Substantia nigra lesion protects against ischemic damage in the striatum.

  1987cited by this paper
• Nitric oxide release accounts for the biological activity of endothelium-derived relaxing factor

  1987cited by this paper
• Small Differences in Intraischemic Brain Temperature Critically Determine the Extent of Ischemic Neuronal Injury

  1987cited by this paper
• Selective sparing of a class of striatal neurons in Huntington's disease.

  1985cited by this paper
• Delayed neuronal death in the gerbil hippocampus following ischemia.

  1982cited by this paper
• Temporal profile of neuronal damage in a model of transient forebrain ischemia

  1982cited by this paper
• Infant resuscitation and prevention of mental retardation.

  1971cited by this paper
• New approaches to preventing brain damage during asphyxia.

  1971cited by this paper
• Hypothermia combined with positive pressure ventilation in resuscitation of the asphyxiated neonate. Clinical observations in 28 infants.

  1969cited by this paper
• A FORMALIN-PERFUSION FIXATION METHOD FOR HISTOPHYSIOLOGICAL STUDY OF THE CENTRAL NERVOUS SYSTEM WITH THE ELECTRON MICROSCOPE.

  1963cited by this paper
• A formalin‐perfusion fixation method for histophysiological study of the central nervous system with the electron microscope

  1963cited by this paper

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  2001cites this paper
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  2000cites this paper
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  2000cites this paper
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  1999cites this paper
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  1998cites this paper
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  1997cites this paper