Autism‐relevant behaviors are minimally impacted by conditional deletion of Pten in oxytocinergic neurons

No abstract is available for this paper.

• Publication year

  2016

• Venue

  Autism Research

• Publication date

  2016-05-25

• Fields of study

  Biology, Medicine

• Identifiers
  DOI 10.1002/aur.1641PMID 27220363PMCID PMC6467524
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

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  2014cited by this paper
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• Molecular and Phenotypic Abnormalities in Individuals with Germline Heterozygous PTEN Mutations and Autism

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  2013influential reference
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  2012influential reference
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  2012cited by this paper
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  2012influential reference
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• Excessive activation of mTOR in postnatally generated granule cells is sufficient to cause epilepsy.

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  2011cited by this paper
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  2010cited by this paper
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  2010cited by this paper
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  2005influential reference
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  2005influential reference
• Pten Loss Causes Hypertrophy and Increased Proliferation of Astrocytes In vivo

  2004influential reference
• Anxiety and Stress Responses in Female Oxytocin Deficient Mice

  2004influential reference
• Female oxytocin-deficient mice display enhanced anxiety-related behavior.

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  2003cited by this paper
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  2003cited by this paper
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  2002cited by this paper
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  2001influential reference
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  2001cited by this paper
• Deletion of Pten in mouse brain causes seizures, ataxia and defects in soma size resembling Lhermitte-Duclos disease

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• Negative Regulation of Neural Stem/Progenitor Cell Proliferation by the Pten Tumor Suppressor Gene in Vivo

  2001influential reference
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  2000cited by this paper
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  2000cited by this paper
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  1999cited by this paper
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  1986cited by this paper
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  1984cited by this paper
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  1963cited by this paper

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• Environmental Enrichment Rescues Social Behavioral Deficits and Synaptic Abnormalities in Pten Haploinsufficient Mice

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