The role of calpain in the regulation of ADAM17-dependent GPIbalpha ectodomain shedding.

No abstract is available for this paper.

• Publication year

  2010

• Venue

  Archives of Biochemistry and Biophysics

• Publication date

  2010-03-15

• Fields of study

  Biology, Medicine

• Identifiers
  DOI 10.1016/j.abb.2010.01.003PMID 20060803
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

• Regulation of Mature ADAM17 by Redox Agents for L-Selectin Shedding1

  2009cited by this paper
• Prolonged inhibition of protein kinase A results in metalloproteinase-dependent platelet GPIbalpha shedding.

  2009cited by this paper
• Role of cAMP-dependent protein kinase in the regulation of platelet procoagulant activity.

  2009cited by this paper
• Shear-induced interaction of platelets with von Willebrand factor results in glycoprotein Ibalpha shedding.

  2009cited by this paper
• Isoform-specific contribution of protein kinase C to prion processing.

  2008cited by this paper
• The ADAM metalloproteinases

  2008cited by this paper
• Controlled shedding of platelet glycoprotein (GP)VI and GPIb–IX–V by ADAM family metalloproteinases

  2007cited by this paper
• Platelet receptor proteolysis: a mechanism for downregulating platelet reactivity.

  2007cited by this paper
• Signaling and regulation of the platelet glycoprotein Ib–IX–V complex

  2007cited by this paper
• Aspirin Induces Platelet Receptor Shedding via ADAM17 (TACE)*

  2005cited by this paper
• Platelet GPIb‐IX‐V‐dependent signaling

  2005cited by this paper
• A critical role for 14-3-3zeta protein in regulating the VWF binding function of platelet glycoprotein Ib-IX and its therapeutic implications.

  2005cited by this paper
• Tumor Necrosis Factor- (cid:1) –Converting Enzyme (ADAM17) Mediates GPIb (cid:1) Shedding From Platelets In Vitro and In Vivo

  2004cited by this paper
• Metalloproteinase inhibitors improve the recovery and hemostatic function of in vitro-aged or -injured mouse platelets.

  2003cited by this paper
• The calpain system.

  2003cited by this paper
• Regulation of von Willebrand Factor Binding to the Platelet Glycoprotein Ib-IX by a Membrane Skeleton-dependent Inside-out Signal*

  2001cited by this paper
• Disruption of the Mouse μ-Calpain Gene Reveals an Essential Role in Platelet Function

  2001cited by this paper
• Calpain regulation of integrin alpha IIb beta 3 signaling in human platelets.

  2000cited by this paper
• Calpain regulation of integrin a IIb 3 signaling in human platelets

  2000cited by this paper
• Inhibition of Calpain Blocks Platelet Secretion, Aggregation, and Spreading*

  1999cited by this paper
• Complementary Roles for Receptor Clustering and Conformational Change in the Adhesive and Signaling Functions of Integrin αIIbβ3

  1998cited by this paper
• Cloning of a disintegrin metalloproteinase that processes precursor tumour-necrosis factor-α

  1997cited by this paper
• Calpain Regulation of Cytoskeletal Signaling Complexes in Von Willebrand Factor-stimulated Platelets

  1997cited by this paper
• A metalloproteinase disintegrin that releases tumour-necrosis factor-α from cells

  1997cited by this paper
• A metalloproteinase disintegrin that releases tumour-necrosis factor-alpha from cells.

  1997cited by this paper
• Calpain Cleavage of the Cytoplasmic Domain of the Integrin β2 Subunit (*)

  1995cited by this paper
• Shedding of adhesion receptors from the surface of activated platelets.

  1994cited by this paper
• pp60src is an endogenous substrate for calpain in human blood platelets.

  1993cited by this paper
• Evidence that agonist-induced activation of calpain causes the shedding of procoagulant-containing microvesicles from the membrane of aggregating platelets.

  1991cited by this paper
• von Willebrand factor binding to platelet GpIb initiates signals for platelet activation.

  1991cited by this paper
• Activation of calpain I and hydrolysis of calpain substrates (actin-binding protein, glycoprotein Ib, and talin) are not a function of thrombin-induced platelet aggregation.

  1991cited by this paper
• The role of calpain in stimulus-response coupling: evidence that calpain mediates agonist-induced expression of procoagulant activity in platelets.

  1990cited by this paper
• THE ROLE OF CALPAIN IN STIMULUS-RESPONSE COUPLING : EVIDENCE THAT CALPAIN MEDIATES AGONIST-INDUCED EXPRESSION OF PROCOAGULANT ACTIVITY IN PLATELETS

  1990cited by this paper
• Glycocalicin binding to von Willebrand factor adsorbed onto collagen-coated or polystyrene surfaces.

  1989cited by this paper
• Calpain I remains intact and intracellular during platelet activation. Immunochemical measurements with monoclonal and polyclonal antibodies.

  1987cited by this paper
• Cleavage site of calcium-dependent protease in human platelet membrane glycoprotein Ib.

  1986cited by this paper
• Identification of two proteins (actin-binding protein and P235) that are hydrolyzed by endogenous Ca2+-dependent protease during platelet aggregation.

  1985cited by this paper
• On the association of glycoprotein Ib and actin-binding protein in human platelets

  1985cited by this paper
• Calcium-dependent proteolysis occurs during platelet aggregation.

  1983cited by this paper
• The action of calcium-dependent protease on platelet surface glycoproteins.

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• Platelet glycocalicin. Interaction with thrombin and role as thrombin receptor of the platelet surface.

  1978cited by this paper

• Effects of Artificial Lung and Roller Pump on Platelet Morphology and Activation During Simulated Extracorporeal Circulation

  2025cites this paper
• Phenotyping of human platelets in response to platelet agonists and inhibitors using multiparameter flow cytometry and unbiased high-dimensional analysis

  2025cites this paper
• The Reduction of Tau Hyperphosphorylation by Cornel Iridoid Glycosides Is Mediated by Their Influence on Calpain Activity

  2022cites this paper
• Actin polymerization regulates glycoprotein Ibα shedding

  2021influential citation
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• Protein kinase C signaling dysfunction in von Willebrand disease ( p . V 1316 M ) type 2 B platelets

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• Protein kinase C signaling dysfunction in von Willebrand disease (p.V1316M) type 2B platelets.

  2018cites this paper
• Platelet heterogeneity in activation-induced glycoprotein shedding: functional effects.

  2018cites this paper
• Selective Inhibition of ADAM17 Efficiently Mediates Glycoprotein Ibα Retention During Ex Vivo Generation of Human Induced Pluripotent Stem Cell‐Derived Platelets

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• Selective Inhibition of ADAM17 Efficiently Mediates Glycoprotein Ibα Retention During Ex Vivo Generation of Human Induced Pluripotent Stem Cell-Derived Platelets.

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• Mitochondria-Derived Reactive Oxygen Species Play an Important Role in Doxorubicin-Induced Platelet Apoptosis

  2015cites this paper
• The role of mitochondrial permeability transition pore in regulating the shedding of the platelet GPIbα ectodomain

  2014cites this paper
• Platelet hyperreactivity explains the bleeding abnormality and macrothrombocytopenia in a murine model of sitosterolemia.

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• The Role of Mitochondria-Derived Reactive Oxygen Species in Hyperthermia-Induced Platelet Apoptosis

  2013cites this paper
• The role of intraplatelet reactive oxygen species in the regulation of platelet glycoprotein Ibα ectodomain shedding.

  2013cites this paper
• Cisplatin induces platelet apoptosis through the ERK signaling pathway.

  2012cites this paper
• Physiologic and pathophysiologic role of calpain: implications for the occurrence of atrial fibrillation.

  2012cites this paper
• Platelet receptor shedding.

  2012cites this paper
• Calpain Activator Dibucaine Induces Platelet Apoptosis

  2011cites this paper