Overproduction of NOX-derived ROS in AML promotes proliferation and is associated with defective oxidative stress signaling.

No abstract is available for this paper.

• Publication year

  2013

• Venue

  Blood

• Publication date

  2013-11-07

• Fields of study

  Biology, Medicine

• Identifiers
  DOI 10.1182/blood-2013-04-491944PMID 24089327
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

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