Sustained activation of p53 in confluent nucleotide excision repair-deficient cells resistant to ultraviolet-induced apoptosis.

No abstract is available for this paper.

• Publication year

  2008

• Venue

  DNA Repair

• Publication date

  2008-06-01

• Fields of study

  Biology, Medicine

• Identifiers
  DOI 10.1016/j.dnarep.2008.03.003PMID 18440285
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

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  2008cited by this paper
• The enigmatic effects of caffeine in cell cycle and cancer.

  2007influential reference
• RPA and ATR link transcriptional stress to p53

  2007cited by this paper
• Perturbed gap-filling synthesis in nucleotide excision repair causes histone H2AX phosphorylation in human quiescent cells

  2007cited by this paper
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• UV-induced apoptosis in XPG-deficient fibroblasts involves activation of CD95 and caspases but not p53.

  2007cited by this paper
• Involvement of DNA replication in ultraviolet-induced apoptosis of mammalian cells

  2006cited by this paper
• Regulation of the p53 transcriptional activity

  2006cited by this paper
• Cell apoptosis: requirement of H2AX in DNA ladder formation, but not for the activation of caspase-3.

  2006cited by this paper
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  2005cited by this paper
• Oxidative stress-induced apoptosis in dividing fibroblasts involves activation of p38 MAP kinase and over-expression of Bax: Resistance of quiescent cells to oxidative stress

  2004cited by this paper
• Regulation of DNA replication by ATR: signaling in response to DNA intermediates.

  2004cited by this paper
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  2004cited by this paper
• The eukaryotic nucleotide excision repair pathway.

  2003cited by this paper
• Effect of cell confluence on ultraviolet light apoptotic responses in DNA repair deficient cells.

  2003influential reference
• Subpathways of nucleotide excision repair and their regulation

  2002cited by this paper
• The genetics of the hereditary xeroderma pigmentosum syndrome.

  2002cited by this paper
• Ultraviolet light-induced apoptosis is associated with S-phase in primary human fibroblasts.

  2002cited by this paper
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  2002cited by this paper
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  2002cited by this paper
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  2001cited by this paper
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  2001cited by this paper
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  2001cited by this paper
• p53 Signaling and cell cycle checkpoints.

  2001influential reference
• Photorepair prevents ultraviolet-induced apoptosis in human cells expressing the marsupial photolyase gene.

  2000cited by this paper
• p21(WAF1/Cip1): more than a break to the cell cycle?

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  2000cited by this paper
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  2000cited by this paper
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  2000cited by this paper
• A role for ATR in the DNA damage-induced phosphorylation of p53.

  1999cited by this paper
• Inhibition of ATM and ATR kinase activities by the radiosensitizing agent, caffeine.

  1999cited by this paper
• The regulation of apoptotic cell death.

  1999cited by this paper
• Apoptosis in Proliferating, Senescent, and Immortalized Keratinocytes*

  1999cited by this paper
• Expression of the p48 xeroderma pigmentosum gene is p53-dependent and is involved in global genomic repair.

  1999cited by this paper
• Signal transduction pathways that regulate cell survival and cell death

  1998cited by this paper
• Blockage of RNA polymerase as a possible trigger for u.v. light-induced apoptosis.

  1996cited by this paper
• Li-Fraumeni syndrome fibroblasts homozygous for p53 mutations are deficient in global DNA repair but exhibit normal transcription-coupled repair and enhanced UV resistance.

  1995cited by this paper
• U.v.-induced nuclear accumulation of p53 is evoked through DNA damage of actively transcribed genes independent of the cell cycle.

  1994cited by this paper
• Transcript cleavage by RNA polymerase II arrested by a cyclobutane pyrimidine dimer in the DNA template.

  1994cited by this paper
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  1993cited by this paper
• A mammalian cell cycle checkpoint pathway utilizing p53 and GADD45 is defective in ataxia-telangiectasia.

  1992cited by this paper
• EFFECTS OF UV, SUNLIGHT AND X‐RAY RADIATION ON QUIESCENT HUMAN CELLS IN CULTURE

  1986cited by this paper
• Failure of RNA synthesis to recover after UV irradiation: an early defect in cells from individuals with Cockayne's syndrome and xeroderma pigmentosum.

  1982cited by this paper
• THE EFFECT OF ULTRAVIOLET LIGHT ON ARRESTED HUMAN DIPLOID CELL POPULATIONS

  1977cited by this paper
• Control of nucleic acid synthesis in human diploid cells undergoing contact inhibition.

  1968cited by this paper

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