p53 Signaling and cell cycle checkpoints.

No abstract is available for this paper.

• Publication year

  2001

• Venue

  Chemical Research in Toxicology

• Publication date

  2001-02-07

• Fields of study

  Biology, Medicine, Chemistry

• Identifiers
  DOI 10.1021/TX000199TPMID 11258974
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

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• Tumor Suppressor p53 Is Required To Modulate BRCA1 Expression

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• p53-dependent G2 arrest associated with a decrease in cyclins A2 and B1 levels in a human carcinoma cell line

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• Pidd, a new death-domain–containing protein, is induced by p53 and promotes apoptosis

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• p53 transcriptional activity is essential for p53‐dependent apoptosis following DNA damage

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• SUMO-1 modification of Mdm2 prevents its self-ubiquitination and increases Mdm2 ability to ubiquitinate p53.

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• Ataxia telangiectasia-mutated phosphorylates Chk2 in vivo and in vitro.

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• Chk1 is an essential kinase that is regulated by Atr and required for the G(2)/M DNA damage checkpoint.

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• Phosphorylation of murine p53, but not human p53, by MAP kinase in vitro and in cultured cells highlights species-dependent variation in post-translational modification

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• mdm2: a bridge over the two tumour suppressors, p53 and Rb

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• p53 and chemosensitivity.

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• The cellular response to p53: the decision between life and death

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