Genetic Analysis of Pathways to Parkinson Disease

In this review I outline the arguments as to whether we should consider Parkinson disease one or more than one entity and discuss genetic findings from Mendelian and whole-genome association analysis in that context. I discuss what the demonstration of disease spread implies for our analysis of the genetic and epidemiologic risk factors for disease and outline the surprising fact that we now have genetically identified on the order of half our risk for developing the disease.

• Publication year

  2010

• Venue

  Neuron

• Publication date

  2010-10-21

• Fields of study

  Biology, Medicine

• Identifiers
  DOI 10.1016/j.neuron.2010.10.014PMID 20955928PMCID 2997424
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

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