Nitric oxide donors attenuate myocardial stunning in conscious rabbits.

No abstract is available for this paper.

• Publication year

  1999

• Venue

  American Journal of Physiology. Heart and Circulatory Physiology

• Publication date

  1999-12-01

• Fields of study

  Medicine

• Identifiers
  DOI 10.1152/ajpheart.1999.277.6.H2495PMID 29592274
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar

• No claims are published for this paper.

• No concepts are published for this paper.

• Nitroglycerin induces late preconditioning against myocardial stunning via a PKC-dependent pathway.

  1999cited by this paper
• Isoform-selective activation of protein kinase C by nitric oxide in the heart of conscious rabbits: a signaling mechanism for both nitric oxide-induced and ischemia-induced preconditioning.

  1999cited by this paper
• Molecular and cellular mechanisms of myocardial stunning.

  1999cited by this paper
• Nitric oxide donors induce late preconditioning against myocardial stunning and infarction in conscious rabbits via an antioxidant-sensitive mechanism.

  1998influential reference
• The nitric oxide hypothesis of late preconditioning

  1998cited by this paper
• Direct evidence that protein kinase C plays an essential role in the development of late preconditioning against myocardial stunning in conscious rabbits and that epsilon is the isoform involved.

  1998influential reference
• Myocardial protection afforded by nicorandil and ischaemic preconditioning in a rabbit infarct model in vivo.

  1998cited by this paper
• Potentiation by nitric oxide of the ATP‐sensitive K+ current induced by K+ channel openers in guinea‐pig ventricular cells

  1997cited by this paper
• Evidence that late preconditioning against myocardial stunning in conscious rabbits is triggered by the generation of nitric oxide.

  1997cited by this paper
• The protective effect of late preconditioning against myocardial stunning in conscious rabbits is mediated by nitric oxide synthase. Evidence that nitric oxide acts both as a trigger and as a mediator of the late phase of ischemic preconditioning.

  1997cited by this paper
• Intra-coronary administration of L-arginine aggravates myocardial stunning through production of peroxynitrite in dogs.

  1997cited by this paper
• Role of adenosine receptors in late preconditioning against myocardial stunning in conscious rabbits.

  1997cited by this paper
• The early and late phases of ischemic preconditioning: a comparative analysis of their effects on infarct size, myocardial stunning, and arrhythmias in conscious pigs undergoing a 40-minute coronary occlusion.

  1997cited by this paper
• Evidence for an essential role of reactive oxygen species in the genesis of late preconditioning against myocardial stunning in conscious pigs.

  1996cited by this paper
• Time course of late preconditioning against myocardial stunning in conscious pigs.

  1996cited by this paper
• Infarct size-reducing effect of nicorandil is mediated by the KATP channel but not by its nitrate-like properties in dogs.

  1996cited by this paper
• Sustain inhibition of nitric oxide by NG-nitro-l-arginine improves myocardial function following ischemia/reperfusion in isolated perfused rat heart

  1995cited by this paper
• Direct detrimental effects of L-arginine upon ischemia--reperfusion injury to myocardium.

  1995cited by this paper
• Late preconditioning against myocardial stunning. An endogenous protective mechanism that confers resistance to postischemic dysfunction 24 h after brief ischemia in conscious pigs.

  1995cited by this paper
• L-arginine reduces endothelial inflammation and myocardial stunning during ischemia/reperfusion.

  1995cited by this paper
• Comparative study on the effects of intracoronary nicorandil and nitroglycerin in ischaemic, reperfused porcine hearts.

  1995cited by this paper
• Effects of nicorandil and glyceryl trinitrate on infarct size, adenosine release, and neutrophil infiltration in the dog.

  1995cited by this paper
• Recurrent ischemia in the canine heart causes recurrent bursts of free radical production that have a cumulative effect on contractile function. A pathophysiological basis for chronic myocardial "stunning".

  1995cited by this paper
• Nitric oxide synthase protects the heart against ischemia-reperfusion injury in rabbits.

  1995cited by this paper
• Endogenous nitric oxide (NO) protects against ischaemia-reperfusion injury in the rabbit.

  1995cited by this paper
• Inhibition of nitric oxide synthesis reduces infarct size by an adenosine-dependent mechanism.

  1995cited by this paper
• Sustained inhibition of nitric oxide by NG-nitro-L-arginine improves myocardial function following ischemia/reperfusion in isolated perfused rat heart.

  1995cited by this paper
• Preconditioning protects ischemic rabbit heart by protein kinase C activation.

  1994cited by this paper
• Evidence that translocation of protein kinase C is a key event during ischemic preconditioning of rabbit myocardium.

  1994cited by this paper
• Attenuation of Myocardial Stunning by the ACE Inhibitor Ramiprilat Through a Signal Cascade of Bradykinin and Prostaglandins but not Nitric Oxide

  1994cited by this paper
• Inhibition of Endothelium‐Derived Relaxing Factor Enhances Myocardial Stunning in Conscious Dogs

  1993cited by this paper
• Preconditioning Improves Energy Metabolism During Reperfusion but Does Not Attenuate Myocardial Stunning in Porcine Hearts

  1993cited by this paper
• EFFECT OF NICORANDIL ON POST‐ISCHAEMIC CONTRACTILE DYSFUNCTION IN THE HEART: ROLES OF ITS ATP‐SENSITIVE K’ CHANNEL OPENING PROPERTY AND NITRATE PROPERTY

  1993cited by this paper
• Control of cardiac muscle cell function by an endogenous nitric oxide signaling system.

  1993cited by this paper
• Nitric oxide regulates cardiac Ca2+ current. Involvement of cGMP-inhibited and cGMP-stimulated phosphodiesterases through guanylyl cyclase activation.

  1993cited by this paper
• Inhibition of nitric oxide limits infarct size in the in situ rabbit heart.

  1993cited by this paper
• The Role of L‐Arginine in Ameliorating Reperfusion Injury After Myocardial Ischemia in the Cat

  1992cited by this paper
• Intracoronary L-arginine during reperfusion improves endothelial function and reduces infarct size.

  1992cited by this paper
• Preconditioning Does Not Attenuate Myocardial Stunning

  1992cited by this paper
• Postischemic myocardial "stunning". Identification of major differences between the open-chest and the conscious dog and evaluation of the oxygen radical hypothesis in the conscious dog.

  1991cited by this paper
• Comparative Effects of Nicorandil, Nitroglycerin, Nicotinic Acid, and SG‐86 on the Metabolic Status and Functional Recovery of the Ischemic‐Reperfused Myocardium

  1987cited by this paper
• Beta-adrenergic stimulation reverses postischemic myocardial dysfunction without producing subsequent functional deterioration.

  1985cited by this paper
• Interaction between Cyclic Adenosine Monophosphate and Cyclic Guanosine Monophosphate in Guinea Pig Ventricular Myocardium

  1975cited by this paper
• Nuclear Factor- (cid:107) B Plays an Essential Role in the Late Phase of Ischemic Preconditioning in Conscious Rabbits

  year unknowncited by this paper

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  2009cites this paper
• Sulfaphenazole Protects Heart Against Ischemia–Reperfusion Injury and Cardiac Dysfunction by Overexpression of iNOS, Leading to Enhancement of Nitric Oxide Bioavailability and Tissue Oxygenation

  2009cites this paper
• Role of Oxygen in Postischemic Myocardial Injury

  2007cites this paper
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  2007cites this paper
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  2007cites this paper
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  2007cites this paper
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  2007cites this paper
• The role of ATP sensitive K+ channels and of nitric oxide synthase on myocardial ischemia/reperfusion-induced apoptosis.

  2006cites this paper
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  2006cites this paper
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  2004cites this paper
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  2002cites this paper
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  1999cites this paper