Inhibition of glycogen synthase kinase-3β prevents sympathetic hyperinnervation in infarcted rats

No abstract is available for this paper.

• Publication year

  2015

• Venue

  Experimental biology and medicine

• Publication date

  2015-01-08

• Fields of study

  Medicine

• Identifiers
  DOI 10.1177/1535370214564746PMID 25576342PMCID PMC4935399
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

• Cardiac Fibroblast Glycogen Synthase Kinase-3&bgr; Regulates Ventricular Remodeling and Dysfunction in Ischemic Heart

  2014cited by this paper
• Cardiac Fibroblast GSK-3 Regulates Ventricular Remodeling and Dysfunction in Ischemic Heart

  2014cited by this paper
• Both PKA and Epac Pathways Mediate N-Acetylcysteine-Induced Connexin43 Preservation in Rats with Myocardial Infarction

  2013cited by this paper
• Cardiac sympathetic hyperinnervation in deoxycorticosterone acetate-salt hypertensive rats.

  2012cited by this paper
• Functional Implications of Glycogen Synthase Kinase-3-Mediated Tau Phosphorylation

  2011influential reference
• Enhanced catecholamine release in mice expressing PKB/SGK-resistant GSK3

  2011cited by this paper
• Role of GSK3 Signaling in Neuronal Morphogenesis

  2011cited by this paper
• MEK/ERKs signaling is essential for lithium‐induced neurite outgrowth in N2a cells

  2011influential reference
• Inhibition of glycogen synthase kinase 3β induces dermal fibrosis by activation of the canonical Wnt pathway

  2011cited by this paper
• Glycogen Synthase Kinase-3&bgr; Regulates Post–Myocardial Infarction Remodeling and Stress-Induced Cardiomyocyte Proliferation In Vivo

  2010influential reference
• Cardiac fibroblasts are essential for the adaptive response of the murine heart to pressure overload.

  2010cited by this paper
• NFAT/Fas signaling mediates the neuronal apoptosis and motor side effects of GSK-3 inhibition in a mouse model of lithium therapy.

  2010cited by this paper
• Loss of tau elicits axonal degeneration in a mouse model of Alzheimer's disease.

  2010cited by this paper
• Tau Potentiates Nerve Growth Factor-induced Mitogen-activated Protein Kinase Signaling and Neurite Initiation without a Requirement for Microtubule Binding*

  2010influential reference
• Tau phosphorylation: the therapeutic challenge for neurodegenerative disease.

  2009cited by this paper
• Supernatant from Bifidobacterium Differentially Modulates Transduction Signaling Pathways for Biological Functions of Human Dendritic Cells

  2008cited by this paper
• Inhibition of glycogen synthase kinase-3.

  2008cited by this paper
• Anti-inflammatory effects of lithium gluconate on keratinocytes: a possible explanation for efficiency in seborrhoeic dermatitis

  2008cited by this paper
• GSK3 alpha and GSK3 beta are necessary for axon formation

  2007cited by this paper
• Glycogen Synthase Kinase-3α Reduces Cardiac Growth and Pressure Overload-induced Cardiac Hypertrophy by Inhibition of Extracellular Signal-regulated Kinases*

  2007cited by this paper
• Essential roles for GSK-3s and GSK-3-primed substrates in neurotrophin-induced and hippocampal axon growth.

  2006cited by this paper
• Glycogen Synthase Kinase 3- and Extracellular Signal-Regulated Kinase-Dependent Phosphorylation of Paxillin Regulates Cytoskeletal Rearrangement

  2006cited by this paper
• TLR2 is Required for the Altered Transcription of p75NGF Receptors in Gram Positive Infection

  2006cited by this paper
• Brain‐derived neurotrophic factor induces a rapid dephosphorylation of tau protein through a PI‐3Kinase signalling mechanism

  2005cited by this paper
• Both the Establishment and the Maintenance of Neuronal Polarity Require Active Mechanisms: Critical Roles of GSK-3β and Its Upstream Regulators

  2005cited by this paper
• Both the Establishment and the Maintenance of Neuronal Polarity Require Active Mechanisms: Critical Roles of GSK-3 and Its Upstream Regulators

  2005cited by this paper
• Both the establishment and the maintenance of neuronal polarity require active mechanisms: critical roles of GSK-3beta and its upstream regulators.

  2005cited by this paper
• Low-dose lithium combined with captopril prevents stroke and improves survival in salt-loaded, stroke-prone spontaneously hypertensive rats

  2005cited by this paper
• Glycogen Synthase Kinase-3β Haploinsufficiency Mimics the Behavioral and Molecular Effects of Lithium

  2004influential reference
• Endothelin-1 regulates cardiac sympathetic innervation in the rodent heart by controlling nerve growth factor expression.

  2004cited by this paper
• A Jekyll and Hyde kinase: roles for Cdk5 in brain development and disease.

  2004cited by this paper
• Adverse syndromes associated with lithium

  2004cited by this paper
• Glycogen synthase kinase-3beta haploinsufficiency mimics the behavioral and molecular effects of lithium.

  2004cited by this paper
• Lithium Blocks the c-Jun Stress Response and Protects Neurons via Its Action on Glycogen Synthase Kinase 3

  2003cited by this paper
• Inhibition of glycogen synthase kinase 3β in sensory neurons in culture alters filopodia dynamics and microtubule distribution in growth cones

  2003cited by this paper
• Lithium treatment alters brain concentrations of nerve growth factor, brain-derived neurotrophic factor and glial cell line-derived neurotrophic factor in a rat model of depression.

  2003cited by this paper
• Molecular targets of lithium action.

  2003cited by this paper
• Inhibition of glycogen synthase kinase 3beta in sensory neurons in culture alters filopodia dynamics and microtubule distribution in growth cones.

  2003cited by this paper
• CRMP-2 binds to tubulin heterodimers to promote microtubule assembly

  2002cited by this paper
• Aberrant Tau Phosphorylation by Glycogen Synthase Kinase-3β and JNK3 Induces Oligomeric Tau Fibrils in COS-7 Cells*

  2002cited by this paper
• Growth retardation and increased apoptosis in mice with homozygous disruption of the Akt1 gene.

  2001cited by this paper
• Selective small‐molecule inhibitors of glycogen synthase kinase‐3 activity protect primary neurones from death

  2001cited by this paper
• Significance of timing of angiotensin AT1 receptor blockade in rats with myocardial infarction-induced heart failure.

  2001cited by this paper
• Relationship between regional cardiac hyperinnervation and ventricular arrhythmia.

  2000cited by this paper
• Selective small molecule inhibitors of glycogen synthase kinase-3 modulate glycogen metabolism and gene transcription.

  2000cited by this paper
• Extracellular-signal-regulated kinase signalling in neurons.

  1999cited by this paper
• Signal transduction underlying growth cone guidance by diffusible factors.

  1999cited by this paper
• Nerve and Epidermal Growth Factor Induce Protein Synthesis and eIF2B Activation in PC12 Cells*

  1998cited by this paper
• Nerve Growth Factor Modulates Synaptic Transmission between Sympathetic Neurons and Cardiac Myocytes

  1997cited by this paper
• Lithium Reduces Tau Phosphorylation by Inhibition of Glycogen Synthase Kinase-3*

  1997cited by this paper
• The effect of chronic lithium treatment on the calibre of axons and nerve fibres in the rat sural nerve.

  1997cited by this paper
• Dosing strategies and time course of response to antimanic drugs.

  1996cited by this paper
• GSK3: a SHAGGY frog story.

  1996cited by this paper
• Lithium inhibits glycogen synthase kinase-3 activity and mimics wingless signalling in intact cells.

  1996influential reference
• Interaction of tau with the neural plasma membrane mediated by tau's amino-terminal projection domain

  1995cited by this paper
• Markedly different effects on ventricular remodeling result in a decrease in inducibility of ventricular arrhythmias.

  1994cited by this paper
• Localization and Developmental Changes of τ Protein Kinase I/Glycogen Synthase Kinase‐3β in Rat Brain

  1994cited by this paper
• Ventricular remodeling after myocardial infarction.

  1993cited by this paper
• Sympathetic nerves in adult rats regenerate normally and restore pilomotor function during an anti‐NGF treatment that prevents their collateral sprouting

  1992cited by this paper
• Ventricular Remodeling After Myocardial Infarction: Experimental Observations and Clinical Implications

  1990cited by this paper
• Nerve growth factor-induced rapid reorganization of microfilaments in PC12 cells: possible roles of different second messenger systems.

  1990cited by this paper
• Effect of transmural versus nontransmural myocardial infarction on inducibility of ventricular arrhythmias during sympathetic stimulation in dogs.

  1988cited by this paper
• Lithium ion inhibits nerve growth factor-induced neurite outgrowth and phosphorylation of nerve growth factor-modulated microtubule-associated proteins

  1985cited by this paper

• Recent advances in small molecule ATR kinase inhibitors as anticancer agents.

  2026cites this paper
• Lithium in cardiovascular health and disease: Pharmacological and clinical implications.

  2025cites this paper
• The Role of Calcium-Independent Phospholipase A2 in the Molecular Mechanisms of Schizophrenia

  2025cites this paper
• Cardiac sympathetic hyperinnervation after myocardial infarction: a systematic review and qualitative analysis

  2023cites this paper
• Crosstalk between GSK-3β-actuated molecular cascades and myocardial physiology

  2020cites this paper
• Lithium interacts with cardiac remodeling: the fundamental value in the pharmacotherapy of bipolar disorder

  2019cites this paper