O-17: Overexpression of a constitutively active form of Phosphatidylinositol 3 kinase is sufficient to Promote GLUT 4 translocation in transfected rat adipose cells

No abstract is available for this paper.

• Publication year

  2009

• Venue

  Experimental and Clinical Endocrinology & Diabetes

• Publication date

  2009-07-15

• Fields of study

  Biology, Medicine

• Identifiers
  DOI 10.1055/s-0029-1211508
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar

• No claims are published for this paper.

• No concepts are published for this paper.

• Ras-dependent induction of cellular responses by constitutively active phosphatidylinositol-3 kinase.

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• Roles of 1-phosphatidylinositol 3-kinase and ras in regulating translocation of GLUT4 in transfected rat adipose cells

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• Phosphatidylinositol 3-kinase activation is required for insulin stimulation of pp70 S6 kinase, DNA synthesis, and glucose transporter translocation

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• Essential role of phosphatidylinositol 3-kinase in insulin-induced glucose transport and antilipolysis in rat adipocytes. Studies with a selective inhibitor wortmannin.

  1994cited by this paper
• Transfection of DNA into isolated rat adipose cells by electroporation: evaluation of promoter activity in transfected adipose cells which are highly responsive to insulin after one day in culture.

  1993cited by this paper

• No citing papers are available for this paper.