The demyelination and altered motor performance following electrolytic lesion in the ventrolateral white matter of spinal cord in male rats: benefit of post-injury administration of estradiol

No abstract is available for this paper.

• Publication year

  2016

• Venue

  Physiology and Pharmacology

• Publication date

  2016-08-10

• Fields of study

  Biology, Medicine

• Identifiers

  No identifiers available.

• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar

• No claims are published for this paper.

• No concepts are published for this paper.

• Role of Microglia and Astrocyte in Central Pain Syndrome Following Electrolytic Lesion at the Spinothalamic Tract in Rats

  2013cited by this paper
• Electromagnetic Field Stimulation Potentiates Endogenous Myelin Repair by Recruiting Subventricular Neural Stem Cells in an Experimental Model of White Matter Demyelination

  2012cited by this paper
• Analysis of Structural and Molecular Events Associated with Adult Rat Optic Chiasm and Nerves Demyelination and Remyelination; Possible Role for 3rd Ventricle Proliferating Cells

  2011cited by this paper
• Spinal cord injury.

  2011cited by this paper
• Effect of Endogenous Androgens on 17β-Estradiol-Mediated Protection after Spinal Cord Injury in Male Rats

  2010influential reference
• IGF-1 Can Either Protect Against or Increase LPS-Induced Damage in the Developing Rat Brain

  2010cited by this paper
• Neuroprotective efficacy of estrogen in experimental spinal cord injury in rats

  2010cited by this paper
• Postinjury estrogen treatment of chronic spinal cord injury improves locomotor function in rats

  2010cited by this paper
• Chronic spinal hemisection in rats induces a progressive decline in transmission in uninjured fibers to motoneurons.

  2009cited by this paper
• Inflammation and its role in neuroprotection, axonal regeneration and functional recovery after spinal cord injury.

  2008cited by this paper
• No Evidence for Chronic Demyelination in Spared Axons after Spinal Cord Injury in a Mouse

  2008cited by this paper
• Prominent Role of the Spinal Central Pattern Generator in the Recovery of Locomotion after Partial Spinal Cord Injuries

  2008cited by this paper
• Effect of 17beta-estradiol on functional outcome, release of cytokines, astrocyte reactivity and inflammatory spreading after spinal cord injury in male rats.

  2008cited by this paper
• Behavioral models of Parkinson's disease in rodents: A new look at an old problem

  2006cited by this paper
• Gender-related differences in recovery of locomotor function after spinal cord injury in mice

  2006cited by this paper
• Estrogen treatment of spinal cord injury attenuates calpain activation and apoptosis

  2006cited by this paper
• Estrogen reduces the severity of autonomic dysfunction in spinal cord-injured male mice.

  2006cited by this paper
• A modified technique for high-resolution staining of myelin.

  2006cited by this paper
• Estradiol and raloxifene protect cultured SN4741 neurons against oxidative stress.

  2005cited by this paper
• Macrophages and neurodegeneration.

  2005cited by this paper
• Estrogen attenuated markers of inflammation and decreased lesion volume in acute spinal cord injury in rats

  2005cited by this paper
• Demyelinated neocortical lesions in marmoset autoimmune encephalomyelitis mimic those in multiple sclerosis.

  2005cited by this paper
• Spinal cord injury is accompanied by chronic progressive demyelination

  2005cited by this paper
• Functional and electrophysiological changes after graded traumatic spinal cord injury in adult rat.

  2005cited by this paper
• Degenerative and regenerative mechanisms governing spinal cord injury.

  2004cited by this paper
• Systemic administration of 17beta-estradiol reduces apoptotic cell death and improves functional recovery following traumatic spinal cord injury in rats.

  2004cited by this paper
• The Pathology of Human Spinal Cord Injury: Defining the Problems

  2004cited by this paper
• Course of motor recovery following ventrolateral spinal cord injury in the rat.

  2004cited by this paper
• Projections and termination of the corticospinal tract in rodents

  2004cited by this paper
• Inflammation and apoptosis: linked therapeutic targets in spinal cord injury.

  2004cited by this paper
• Estrogen attenuates oxidative stress-induced apoptosis in C6 glial cells.

  2003cited by this paper
• Cell death in models of spinal cord injury.

  2002cited by this paper
• Functional Redundancy of Ventral Spinal Locomotor Pathways

  2002cited by this paper
• Sexual dimorphism in the spontaneous recovery from spinal cord injury: a gender gap in beneficial autoimmunity?

  2002influential reference
• Recent advances in pathophysiology and treatment of spinal cord injury.

  2002cited by this paper
• Chronically injured supraspinal neurons exhibit only modest axonal dieback in response to a cervical hemisection lesion.

  2001cited by this paper
• Oligodendroglial apoptosis occurs along degenerating axons and is associated with FAS and p75 expression following spinal cord injury in the rat.

  2001cited by this paper
• Spontaneous corticospinal axonal plasticity and functional recovery after adult central nervous system injury

  2001cited by this paper
• Biology of oligodendrocyte and myelin in the mammalian central nervous system.

  2001cited by this paper
• Essential role of oligodendrocytes in the formation and maintenance of central nervous system nodal regions.

  2001influential reference
• Schwann Cells Are Removed from the Spinal Cord after Effecting Recovery from Paraplegia

  2000cited by this paper
• Neuroanatomical substrates of functional recovery after experimental spinal cord injury: implications of basic science research for human spinal cord injury.

  2000cited by this paper
• Nerve Growth Factor in Glia and Inflammatory Cells of the Injured Rat Spinal Cord

  2000cited by this paper
• Review of Current Evidence for Apoptosis After Spinal Cord Injury

  2000cited by this paper
• The re-expression of the homeodomain transcription factor Gtx during remyelination of experimentally induced demyelinating lesions in young and old rat brain.

  2000cited by this paper
• Death of oligodendrocytes and microglial phagocytosis of myelin precede immigration of Schwann cells into the spinal cord

  1999cited by this paper
• Apoptosis of oligodendrocytes occurs for long distances away from the primary injury after compression trauma to rat spinal cord

  1999cited by this paper
• Apoptosis after traumatic human spinal cord injury.

  1998cited by this paper
• Neuronal and Glial Apoptosis after Traumatic Spinal Cord Injury

  1997cited by this paper
• Apoptosis of microglia and oligodendrocytes after spinal cord contusion in rats

  1997cited by this paper
• The Pathobiology of the Oligodendrocyte

  1997cited by this paper
• Apoptosis and delayed degeneration after spinal cord injury in rats and monkeys

  1997cited by this paper
• Observations on the pathology of several types of human spinal cord injury, with emphasis on the astrocyte response to penetrating injuries.

  1997cited by this paper
• Induction and its spread of apoptosis in rat spinal cord after mechanical trauma.

  1996cited by this paper
• Demonstration of steroid hormone receptors and steroid action in primary cultures of rat glial cells.

  1992cited by this paper
• Cell death and control of cell survival in the oligodendrocyte lineage.

  1992cited by this paper
• Demyelination in spinal cord injury.

  1989cited by this paper
• Cellular morphology of chronic spinal cord injury in the cat: analysis of myelinated axons by line-sampling.

  1983cited by this paper
• Localization of a descending pathway in the spinal cord which is necessary for controlled treadmill locomotion.

  1980cited by this paper
• Rubrospinal projections in the rat

  1974cited by this paper
• Demyelination and remyelination after acute spinal cord compression.

  1973cited by this paper
• A note on a simple apparatus for detecting neurological deficit in rats and mice.

  1957cited by this paper

• Impact of commonly administered drugs on the progression of spinal cord injury: a systematic review

  2024cites this paper
• The neuroprotective effects of estrogen and estrogenic compounds in spinal cord injury

  2022cites this paper