The Virulence Plasmid of Shigellae: an Archipelago of Pathogenicity Islands?

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• Publication year

  1999

• Venue

  Unknown venue

• Publication date

  Unknown publication date

• Fields of study

  Biology, Medicine

• Identifiers
  DOI 10.1128/9781555818173.CH8
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar

• No claims are published for this paper.

• No concepts are published for this paper.

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  1998cited by this paper
• Induction of type III secretion in Shigella flexneri is associated with differential control of transcription of genes encoding secreted proteins

  1998cited by this paper
• SopA, the outer membrane protease responsible for polar localization of IcsA in Shigella flexneri

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  1997cited by this paper
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  1997cited by this paper
• Use of a novel approach, termed island probing, identifies the Shigella flexneri she pathogenicity island which encodes a homolog of the immunoglobulin A protease-like family of proteins

  1997cited by this paper
• The invasion‐associated type III system of Salmonella typhimurium directs the translocation of Sip proteins into the host cell

  1997cited by this paper
• Disruption of IcsP, the major Shigella protease that cleaves IcsA, accelerates actin‐based motility

  1997cited by this paper
• Modulation of bacterial entry into epithelial cells by association between vinculin and the Shigella IpaA invasin

  1997cited by this paper
• EspP, a novel extracellular serine protease of enterohaemorrhagic Escherichia coli O157:H7 cleaves human coagulation factor V

  1997cited by this paper
• Identification of a virulence locus encoding a second type III secretion system in Salmonella typhimurium.

  1996cited by this paper
• The secreted Ipa complex of Shigella flexneri promotes entry into mammalian cells.

  1996cited by this paper
• A Shigella flexneri invasion plasmid gene, ipgH, with homology to IS629 and sequences encoding bacterial sugar phosphate transport proteins.

  1996cited by this paper
• A bacterial invasin induces macrophage apoptosis by binding directly to ICE.

  1996cited by this paper
• Modulation of Virulence Factor Expression by Pathogen Target Cell Contact

  1996cited by this paper
• Status of YopM and YopN in the Yersinia Yop virulon: YopM of Y.enterocolitica is internalized inside the cytosol of PU5–1.8 macrophages by the YopB, D, N delivery apparatus.

  1996cited by this paper
• Molecular mechanisms of bacterial virulence: type III secretion and pathogenicity islands.

  1996cited by this paper
• Analysis of epithelial cell stress response during infection by Shigella flexneri

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• Identification and characterization of phoN-Sf, a gene on the large plasmid of Shigella flexneri 2a encoding a nonspecific phosphatase

  1996cited by this paper
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  1996cited by this paper
• Molecular genetic bases of Salmonella entry into host cells

  1996cited by this paper
• MxiG, a membrane protein required for secretion of Shigella spp. Ipa invasins: involvement in entry into epithelial cells and in intercellular dissemination

  1995cited by this paper
• Contact of Shigella with host cells triggers release of Ipa invasins and is an essential function of invasiveness.

  1995cited by this paper
• Shigella flexneri surface protein IcsA is sufficient to direct actin-based motility.

  1995cited by this paper
• Functional conservation of the Salmonella and Shigella effectors of entry into epithelial cells

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• Cytoskeletal rearrangements and the functional role of T-plastin during entry of Shigella flexneri into HeLa cells

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• The unrelated surface proteins ActA of Listeria monocytogenes and lcsA of Shigella flexneri are sufficient to confer actin‐based motility on Listeria innocua and Escherichia coli respectively

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• Functional conservation of the secretion and translocation machinery for virulence proteins of yersiniae, salmonellae and shigellae.

  1995cited by this paper
• VIRULENT SHIGELLA CODES FOR A SOLUBLE APYRASE : IDENTIFICATION, CHARACTERIZATION AND CLONING OF THE GENE

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• Extracellular Transport of VirG Protein in Shigella(*)

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• Cleavage of Shigella surface protein VirG occurs at a specific site, but the secretion is not essential for intracellular spreading

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• H-NS regulation of virulence gene expression in enteroinvasive Escherichia coli harboring the virulence plasmid integrated into the host chromosome

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• SepA, the major extracellular protein of Shigella flexneri: autonomous secretion and involvement in tissue invasion

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• Enhanced secretion through the Shigella flexneri Mxi‐Spa translocon leads to assembly of extracellular proteins into macromolecular structures

  1995cited by this paper
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  1995cited by this paper
• Target cell contact triggers expression and polarized transfer of Yersinia YopE cytotoxin into mammalian cells.

  1994cited by this paper
• Extracellular association and cytoplasmic partitioning of the IpaB and IpaC invasins of S. flexneri.

  1994cited by this paper
• Polymorphonuclear leukocyte transmigration promotes invasion of colonic epithelial monolayer by Shigella flexneri.

  1994cited by this paper
• Translocation of a hybrid YopE‐adenylate cyclase from Yersinia enterocolitica into HeLa cells

  1994cited by this paper
• Genetic and molecular analyses of the interaction between the flagellum‐specific sigma and anti‐sigma factors in Salmonella typhimurium.

  1994cited by this paper
• The secretion of the Shigella flexneri Ipa invasins is activated by epithelial cells and controlled by IpaB and IpaD.

  1994cited by this paper
• Sensing structural intermediates in bacterial flagellar assembly by export of a negative regulator.

  1993cited by this paper
• Characterization of the Shigella flexneri ipgD and ipgF genes, which are located in the proximal part of the mxi locus

  1993cited by this paper
• Site of transcriptional activation of virB on the large plasmid of Shigella flexneri 2a by VirF, a member of the AraC family of transcriptional activators.

  1993cited by this paper
• Transcriptional control of the invasion regulatory gene virB of Shigella flexneri: activation by virF and repression by H-NS

  1993cited by this paper
• Eight genes in region 5 that form an operon are essential for invasion of epithelial cells by Shigella flexneri 2a

  1993cited by this paper
• Nonpolar mutagenesis of the ipa genes defines IpaB, IpaC, and IpaD as effectors of Shigella flexneri entry into epithelial cells

  1993cited by this paper
• Unipolar localization and ATPase activity of IcsA, a Shigella flexneri protein involved in intracellular movement

  1993cited by this paper
• MxiD, an outer membrane protein necessary for the secretion of the Shigella flexneri Ipa invasins

  1993cited by this paper
• icsB: a Shigella flexneri virulence gene necessary for the lysis of protrusions during intercellular spread

  1992cited by this paper
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  1992cited by this paper
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  1992cited by this paper
• Pathogenesis of Shigellosis

  1992cited by this paper
• Shigella flexneri enters human colonic Caco-2 epithelial cells through the basolateral pole

  1992cited by this paper
• Molecular and cellular biology of Shigella flexneri invasiveness: from cell assay systems to shigellosis.

  1992cited by this paper
• Unipolar reorganization of F-actin layer at bacterial division and bundling of actin filaments by plastin correlate with movement of Shigella flexneri within HeLa cells

  1992cited by this paper
• Shigella flexneri induces apoptosis in infected macrophages

  1992cited by this paper
• Identification and characterization of virK, a virulence‐associated large plasmid gene essential for intercellular spreading of Shigella flexneri

  1992cited by this paper
• MxiJ, a lipoprotein involved in secretion of Shigella Ipa invasins, is homologous to YscJ, a secretion factor of the Yersinia Yop proteins

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• Surface presentation of Shigella flexneri invasion plasmid antigens requires the products of the spa locus

  1992cited by this paper
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  1991cited by this paper
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  1991cited by this paper
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• Temperature‐regulated expression of invasion genes in Shigella flexneri is controlled through the transcriptional activation of the virB gene on the large plasmid

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  1990cited by this paper
• Molecular characterization of a trans-acting, positive effector (ipaR) of invasion plasmid antigen synthesis in Shigella flexneri serotype 5.

  1990cited by this paper
• Histone-like protein H1 (H-NS), DNA supercoiling, and gene expression in bacteria.

  1990cited by this paper
• Sequence and molecular characterization of a multicopy invasion plasmid antigen gene, ipaH, of Shigella flexneri

  1990cited by this paper
• DNA supercoiling and environmental regulation of virulence gene expression in Shigella flexneri

  1990cited by this paper
• Nucleotide sequence of invasion plasmid antigen gene ipaA from Shigella flexneri 5.

  1990cited by this paper
• A large outbreak of antibiotic-resistant shigellosis at a mass gathering.

  1990cited by this paper
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  1989cited by this paper
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  1989cited by this paper
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  1989cited by this paper
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  1989cited by this paper
• Identification of icsA, a plasmid locus of Shigella flexneri that governs bacterial intra- and intercellular spread through interaction with F-actin.

  1989cited by this paper
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  1989cited by this paper
• Functional organization and nucleotide sequence of virulence Region‐2 on the large virulence plasmid in Shigella flexneri 2a

  1989cited by this paper
• Nucleotide sequence of the invasion plasmid antigen B and C genes (ipaB and ipaC) of Shigella flexneri.

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• Genetic relatedness of the basic replicon of the virulence plasmid in shigellae and enteroinvasive Escherichia coli.

  1988cited by this paper
• Identification of a chromosomal gene controlling temperature-regulated expression of Shigella virulence.

  1988cited by this paper
• Expression of four virulence antigens of Shigella flexneri is positively regulated at the transcriptional level by the 30 kilo Dalton virF protein

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• Characterization of invasion plasmid antigen genes (ipaBCD) from Shigella flexneri.

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• A basic replicon of virulence-associated plasmids of Shigella spp. and enteroinvasive Escherichia coli is homologous with a basic replicon in plasmids of IncF groups

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• Virulence-associated genetic regions comprising 31 kilobases of the 230-kilobase plasmid in Shigella flexneri 2a

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• Cloning of plasmid DNA sequences involved in invasion of HeLa cells by Shigella flexneri

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