Retinoic Acid Potentiates Nitric Oxide-Induced Dedifferentiation through the ERK Pathway in Rabbit Articular Chondrocytes

No abstract is available for this paper.

• Publication year

  2011

• Venue

  Journal of Life Science

• Publication date

  2011-04-30

• Fields of study

  Biology, Medicine, Chemistry

• Identifiers
  DOI 10.5352/JLS.2011.21.4.534
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar

• No claims are published for this paper.

• No concepts are published for this paper.

• Cartilage in normal and osteoarthritis conditions.

  2008cited by this paper
• ERK-1/-2 and p38 Kinase Oppositely Regulate 15-deoxy-Δ12,14-prostaglandinJ2-Induced PPAR-γ Activation That Mediates Dedifferentiation But Not Cyclooxygenase-2 Expression in Articular Chondrocytes

  2007cited by this paper
• Articular cartilage, degenerative process, and repair: current progress.

  2006cited by this paper
• The selective inhibition of mucopolysaccharide synthesis by vitamin a treatment of cultured chick embryo chondrocytes

  2005cited by this paper
• Production of congenital limb defects with retinoic acid: Phenomenological evidence of progressive differentiation during limb morphogenesis

  2004cited by this paper
• Non-steroidal anti-inflammatory drugs (NSAIDs) inhibit nitric oxide-induced apoptosis and dedifferentiation of articular chondrocytes independent of cyclooxygenase activity

  2003cited by this paper
• Actin Cytoskeletal Architecture Regulates Nitric Oxide-induced Apoptosis, Dedifferentiation, and Cyclooxygenase-2 Expression in Articular Chondrocytes via Mitogen-activated Protein Kinase and Protein Kinase C Pathways*

  2003cited by this paper
• Protein kinase Cα and ζ regulate nitric oxide-induced NF-κB activation that mediates cyclooxygenase-2 expression and apoptosis but not dedifferentiation in articular chondrocytes

  2003cited by this paper
• Retinoic acid inhibits chondrogenesis of mesenchymal cells by sustaining expression of N‐cadherin and its associated proteins

  2003cited by this paper
• p38 Kinase-dependent and -independent Inhibition of Protein Kinase C ζ and -α Regulates Nitric Oxide-induced Apoptosis and Dedifferentiation of Articular Chondrocytes*

  2002cited by this paper
• ERK-1/2 and p38 Kinase Oppositely Regulate Nitric Oxide-induced Apoptosis of Chondrocytes in Association with p53, Caspase-3, and Differentiation Status*

  2002cited by this paper
• p38 Kinase Regulates Nitric Oxide-induced Apoptosis of Articular Chondrocytes by Accumulating p53 via NFκB-dependent Transcription and Stabilization by Serine 15 Phosphorylation*

  2002cited by this paper
• Nitric oxide and inflammatory mediators in the perpetuation of osteoarthritis

  2001cited by this paper
• Articular cartilage and changes in Arthritis: Cell biology of osteoarthritis

  2001cited by this paper
• Articular cartilage and changes in Arthritis: Collagen of articular cartilage

  2001cited by this paper
• A Composite Element Binding the Vitamin D Receptor and the Retinoic X Receptor α Mediates the Transforming Growth Factor-β Inhibition of Decorin Gene Expression in Articular Chondrocytes*

  2001cited by this paper
• The Induction of Cell Death in Human Osteoarthritis Chondrocytes by Nitric Oxide Is Related to the Production of Prostaglandin E2 Via the Induction of Cyclooxygenase-2

  2000cited by this paper
• Regulation of Skeletal Progenitor Differentiation by the Bmp and Retinoid Signaling Pathways

  2000cited by this paper
• Cellular interactions and signaling in cartilage development.

  2000cited by this paper
• Developmental patterns of cartilage.

  1999cited by this paper
• Nitric oxide synthase and cyclooxygenases: distribution, regulation, and intervention in arthritis.

  1999cited by this paper
• An introduction to the pathophysiology of osteoarthritis.

  1999cited by this paper
• Retinoids and their receptors in skeletal development

  1998cited by this paper
• The role of nitric oxide in articular cartilage breakdown in osteoarthritis.

  1998cited by this paper
• Nitric oxide inhibits the synthesis of type-II collagen without altering Col2A1 mRNA abundance: prolyl hydroxylase as a possible target.

  1997cited by this paper
• Retinoic Acid Receptor α Function in Vertebrate Limb Skeletogenesis: a Modulator of Chondrogenesis

  1997cited by this paper
• Nitric oxide mediates interleukin-1-induced matrix degradation and basic fibroblast growth factor release in cultured rabbit articular chondrocytes: a possible mechanism of pathological neovascularization in arthritis.

  1996cited by this paper
• 9-cis retinoic acid antagonizes the stimulatory effect of 1,25 dihydroxyvitamin D3 on chondrogenesis of chick limb bud mesenchymal cells: interactions of their receptors.

  1996cited by this paper
• Modulation of limb bud chondrogenesis by retinoic acid and retinoic acid receptors.

  1995cited by this paper
• Nitric oxide mediates suppression of cartilage proteoglycan synthesis by interleukin-1.

  1994cited by this paper
• Interleukin-1 beta-modulated gene expression in immortalized human chondrocytes.

  1994cited by this paper
• Inhibition of chondrogenesis by retinoic acid in limb mesenchymal cells in vitro: effects on PGE2 and cyclic AMP concentrations.

  1988cited by this paper
• Chondrogenesis and cell proliferation in limb bud cell cultures treated with cytosine arabinoside and vitamin A.

  1978cited by this paper

• Destabilising microtubule polymerization regulates chondrocyte dedifferentiation and inflammation via nuclear factor kappa b and β-catenin pathway

  2023cites this paper