Atrial cardiomyocyte calcium signalling.

No abstract is available for this paper.

• Publication year

  2011

• Venue

  Biochimica et Biophysica Acta

• Publication date

  2011-05-01

• Fields of study

  Biology, Medicine

• Identifiers
  DOI 10.1016/j.bbamcr.2011.01.030PMID 21295621
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

• Alterations of atrial Ca(2+) handling as cause and consequence of atrial fibrillation.

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  2010influential reference
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• T-Tubule Remodeling During Transition From Hypertrophy to Heart Failure

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• Elevated InsP3R expression underlies enhanced calcium fluxes and spontaneous extra-systolic calcium release events in hypertrophic cardiac myocytes

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  2009cited by this paper
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• Enhanced Activity of Inositol-1,4,5-Trisphosphate Receptors in Atrial Myocytes of Atrial Fibrillation Patients

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  2008cited by this paper
• Calcium-Handling Abnormalities Underlying Atrial Arrhythmogenesis and Contractile Dysfunction in Dogs With Congestive Heart Failure

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  2007influential reference
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  2006influential reference
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  2005cited by this paper
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  2004influential reference
• Down‐regulation of sarcolipin mRNA expression in chronic atrial fibrillation

  2004cited by this paper
• Modulation of sarcoplasmic reticulum calcium release by calsequestrin in cardiac myocytes.

  2004cited by this paper
• Atrial Fibrillation Is Associated With Increased Spontaneous Calcium Release From the Sarcoplasmic Reticulum in Human Atrial Myocytes

  2004cited by this paper
• beta-adrenergic stimulation restores the Ca transient of ventricular myocytes lacking t-tubules.

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• Inhibitors of SERCA and mitochondrial Ca-uniporter decrease velocity of calcium waves in rat cardiomyocytes

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  2004cited by this paper
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  2002influential reference
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  2001influential reference
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• Regulation of Ryanodine Receptor Opening by Lumenal Ca2+ Underlies Quantal Ca2+ Release in PC12 Cells*

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  1999cited by this paper
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• Calcium concentration and movement in the ventricular cardiac cell during an excitation-contraction cycle.

  1998cited by this paper
• Two-dimensional confocal images of organization, density, and gating of focal Ca2+ release sites in rat cardiac myocytes.

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