Prolactin and growth hormone signal transduction in lymphohaemopoietic cells

No abstract is available for this paper.

• Publication year

  1998

• Venue

  Cellular and Molecular Life Sciences (CMLS)

• Publication date

  1998-10-01

• Fields of study

  Biology, Medicine

• Identifiers
  DOI 10.1007/s000180050235PMID 9817985PMCID PMC11147239
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

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  1998cited by this paper
• Growth Hormone Preferentially Induces the Rapid, Transient Expression of SOCS-3, a Novel Inhibitor of Cytokine Receptor Signaling*

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• Multistage regulation of Th1-type immune responses by the transcription factor IRF-1.

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• Transcriptional Inhibition by Stat5

  1997cited by this paper
• Mapping of a Cytoplasmic Domain of the Human Growth Hormone Receptor That Regulates Rates of Inactivation of Jak2 and Stat Proteins*

  1997cited by this paper
• Molecular Actions of Prolactin in the Immune System

  1997cited by this paper
• A mammalian model for Laron syndrome produced by targeted disruption of the mouse growth hormone receptor/binding protein gene (the Laron mouse).

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• Macrophages from motheaten and viable motheaten mutant mice show increased proliferative responses to GM-CSF: detection of potential HCP substrates in GM-CSF signal transduction.

  1997cited by this paper
• Tyrosine phosphorylation of the EGF receptor by the kinase Jak2 is induced by growth hormone

  1997cited by this paper
• Multiple prolactin (PRL) receptor cytoplasmic residues and Stat1 mediate PRL signaling to the interferon regulatory factor-1 promoter.

  1997cited by this paper
• The role of GH receptor tyrosine phosphorylation in Stat5 activation.

  1997cited by this paper
• Defective mammopoiesis, but normal hematopoiesis, in mice with a targeted disruption of the prolactin gene

  1997cited by this paper
• A short isoform of the human growth hormone receptor functions as a dominant negative inhibitor of the full-length receptor and generates large amounts of binding protein.

  1997cited by this paper
• Null mutation of the prolactin receptor gene produces multiple reproductive defects in the mouse.

  1997cited by this paper
• Interferon regulatory factor-1 is required for a T helper 1 immune response in vivo.

  1997cited by this paper
• The Role of mGrb10α in Insulin-like Growth Factor I-mediated Growth*

  1997cited by this paper
• Stat5a is mandatory for adult mammary gland development and lactogenesis.

  1997cited by this paper
• Prolactin Activates Tyrosyl Phosphorylation of Insulin Receptor Substrate 1 and Phosphatidylinositol-3-OH Kinase*

  1997cited by this paper
• STATs and gene regulation.

  1997cited by this paper
• Tyrosine Docking Sites of the Rat Prolactin Receptor Required for Association and Activation of Stat5*

  1997cited by this paper
• Signaling molecules involved in coupling growth hormone receptor to mitogen-activated protein kinase activation.

  1997cited by this paper
• Prolactin Stimulates Serine/Tyrosine Phosphorylation and Formation of Heterocomplexes of Multiple Stat5 Isoforms in Nb2 Lymphocytes*

  1997cited by this paper
• Interleukin 3-dependent survival by the Akt protein kinase.

  1997cited by this paper
• Interaction of Growth Hormone-activated STATs with SH2-containing Phosphotyrosine Phosphatase SHP-1 and Nuclear JAK2 Tyrosine Kinase*

  1997cited by this paper
• Nuclear integration of JAK/STAT and Ras/AP-1 signaling by CBP and p300.

  1997cited by this paper
• The p300/CBP family: integrating signals with transcription factors and chromatin.

  1997cited by this paper
• Prolactin and growth hormone receptors

  1996cited by this paper
• Growth Hormone, Interferon-γ, and Leukemia Inhibitory Factor Utilize Insulin Receptor Substrate-2 in Intracellular Signaling*

  1996cited by this paper
• Two contact regions between Stat1 and CBP/p300 in interferon gamma signaling.

  1996cited by this paper
• Alternatively spliced forms in the cytoplasmic domain of the human growth hormone (GH) receptor regulate its ability to generate a soluble GH-binding protein.

  1996cited by this paper
• Two contact regions between Stat1 and CBP/p300 in interferon γ signaling

  1996cited by this paper
• Growth hormone, growth factors and hematopoiesis.

  1996cited by this paper
• Multiple stat complexes interact at the interferon regulatory factor-1 interferon-gamma activation sequence in prolactin-stimulated Nb2 T cells.

  1996cited by this paper
• The transcription factor interferon regulatory factor-1 is essential for natural killer cell function in vivo

  1996cited by this paper
• The ubiquitin conjugation system is required for ligand‐induced endocytosis and degradation of the growth hormone receptor.

  1996cited by this paper
• Requirement for phosphoinositide 3-OH kinase in growth hormone signalling to the mitogen-activated protein kinase and p70s6k pathways.

  1996cited by this paper
• The role of the growth hormone (GH) receptor and JAK1 and JAK2 kinases in the activation of Stats 1, 3, and 5 by GH.

  1996cited by this paper
• Cooperation of the tumour suppressors IRF-1 and p53 in response to DNA damage

  1996cited by this paper
• PTP1D is a positive regulator of the prolactin signal leading to beta‐casein promoter activation.

  1996cited by this paper
• Functional interactions between Stat5 and the glucocorticoid receptor

  1996cited by this paper
• Identification of growth hormone receptor (GHR) tyrosine residues required for GHR phosphorylation and JAK2 and STAT5 activation.

  1996cited by this paper
• Differential activation of Stat3 and Stat5 by distinct regions of the growth hormone receptor.

  1996cited by this paper
• Biphasic transcriptional regulation of the interferon regulatory factor-1 gene by prolactin: involvement of gamma-interferon-activated sequence and Stat-related proteins.

  1995cited by this paper
• Maximal activation of transcription by Stat1 and Stat3 requires both tyrosine and serine phosphorylation.

  1995cited by this paper
• Growth Hormone, Interferon-γ, and Leukemia Inhibitory Factor Promoted Tyrosyl Phosphorylation of Insulin Receptor Substrate-1 (*)

  1995cited by this paper
• Amino acids of the human growth hormone receptor that are required for proliferation and Jak-STAT signaling.

  1995cited by this paper
• Growth hormone receptor C-terminal domains required for growth hormone-induced intracellular free Ca2+ oscillations and gene transcription.

  1995cited by this paper
• The membrane proximal region of the cytoplasmic domain of the growth hormone receptor is involved in the activation of Stat 3

  1995cited by this paper
• An IRF-1-dependent pathway of DNA damage-induced apoptosis in mitogen-activated T lymphocytes

  1995cited by this paper
• Transcriptional responses to polypeptide ligands: the JAK-STAT pathway.

  1995cited by this paper
• Proline-rich Sequence-mediated Jak2 Association to the Prolactin Receptor Is Required but Not Sufficient for Signal Transduction (*)

  1995cited by this paper
• Stat1 Depends on Transcriptional Synergy with Sp1 (*)

  1995cited by this paper
• Identification of Phenylalanine 346 in the Rat Growth Hormone Receptor as Being Critical for Ligand-mediated Internalization and Down-regulation (*)

  1995cited by this paper
• The MAPK signaling cascade

  1995cited by this paper
• Transcription Factors Stat3 and Stat5b Are Present in Rat Liver Nuclei Late in an Acute Phase Response and Bind Interleukin-6 Response Elements (*)

  1995cited by this paper
• Intracellular Tyrosine Residues of the Human Growth Hormone Receptor Are Not Required for the Signaling of Proliferation or Jak-STAT Activation (*)

  1995cited by this paper
• Human growth hormone increases cytosolic free calcium in cultured human IM-9 lymphocytes: a novel mechanism of growth hormone transmembrane signalling.

  1994cited by this paper
• Rapid stimulation of mitogen-activated protein kinase of rat liver by prolactin.

  1994cited by this paper
• Tissue distribution and regulation of rat prolactin receptor gene expression. Quantitative analysis by polymerase chain reaction.

  1994cited by this paper
• Activation of protein kinase C family members by the novel polyphosphoinositides PtdIns-3,4-P2 and PtdIns-3,4,5-P3.

  1994cited by this paper
• Growth signaling and JAK2 association mediated by membrane-proximal cytoplasmic regions of prolactin receptors.

  1994cited by this paper
• Growth hormone stimulates tyrosine phosphorylation of insulin receptor substrate-1.

  1994cited by this paper
• Differential expression of surface membrane growth hormone receptor on human peripheral blood lymphocytes detected by dual fluorochrome flow cytometry.

  1994cited by this paper
• Cellular commitment to oncogene-induced transformation or apoptosis is dependent on the transcription factor IRF-1.

  1994cited by this paper
• Prolactin-induced proliferation of Nb2 cells involves tyrosine phosphorylation of the prolactin receptor and its associated tyrosine kinase JAK2.

  1994cited by this paper
• Growth hormone and erythropoietin differentially activate DNA-binding proteins by tyrosine phosphorylation

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• Distinct cytoplasmic regions of the growth hormone receptor are required for activation of JAK2, mitogen-activated protein kinase, and transcription.

  1994cited by this paper
• The protein tyrosine kinase P59fyn is associated with prolactin (PRL) receptor and is activated by PRL stimulation of T-lymphocytes.

  1994cited by this paper
• Phosphatidylinositol 3-kinase activation is required for insulin stimulation of pp70 S6 kinase, DNA synthesis, and glucose transporter translocation

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• JAK protein tyrosine kinases: their role in cytokine signalling.

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• Induction by interleukin‐6 of interferon regulatory factor 1 (IRF‐1) gene expression through the palindromic interferon response element pIRE and cell type‐dependent control of IRF‐1 binding to DNA.

  1994cited by this paper
• Growth hormone induces tyrosine phosphorylation but does not alter insulin-like growth factor-I gene expression in human IM9 lymphocytes.

  1994cited by this paper
• Cytoplasmic sequences of the growth hormone receptor necessary for signal transduction.

  1994cited by this paper
• Multiple prolactin-responsive elements mediate G1 and S phase expression of the interferon regulatory factor-1 gene.

  1994cited by this paper
• Evidence for generation of the growth hormone-binding protein through proteolysis of the growth hormone membrane receptor.

  1993cited by this paper
• Expression of prolactin receptors in murine lymphoid cells in normal and autoimmune situations.

  1993cited by this paper
• SH2 and SH3 domains.

  1993cited by this paper
• Mutational analysis of the intracellular domain of the human growth hormone receptor.

  1993cited by this paper
• Prolactin and autoimmune disease.

  1993cited by this paper
• Stimulation by growth hormone of MAP kinase activity in 3T3-F442A fibroblasts.

  1992cited by this paper
• Hyperprolactinemia in systemic lupus erythematosus: association with disease activity.

  1992cited by this paper
• Human thymocytes express a prolactin-like messenger ribonucleic acid and synthesize bioactive prolactin-like proteins.

  1992cited by this paper
• Prolactin gene expression in human thymocytes.

  1992cited by this paper
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• A role for protein kinases in the growth hormone regulation of cytochrome P4502C12 and insulin-like growth factor-I messenger RNA expression in primary adult rat hepatocytes.

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  1990cited by this paper
• Haemopoietic receptors and helical cytokines.

  1990cited by this paper
• Prolactin stimulates transcription of growth-related genes in Nb2 T lymphoma cells.

  1990cited by this paper
• Growth hormone stimulates the formation of sn-1,2-diacylglycerol in rat hepatocytes.

  1990cited by this paper
• Interferon-regulatory factor 1 is an immediate-early gene under transcriptional regulation by prolactin in Nb2 T cells

  1990cited by this paper
• The phenotypic and molecular characterization of Nb2 lymphoma cells activated with IL-2 and human growth hormone.

  1989cited by this paper
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  1989cited by this paper
• The growth hormone-binding protein in rat serum is an alternatively spliced form of the rat growth hormone receptor.

  1989cited by this paper
• Growth hormone stimulates c-fos gene expression by means of protein kinase C without increasing inositol lipid turnover.

  1989cited by this paper
• Mouse serum growth hormone (GH) binding protein has GH receptor extracellular and substituted transmembrane domains.

  1989cited by this paper
• Suppression of macrophage activation and T-lymphocyte function in hypoprolactinemic mice.

  1988cited by this paper
• Rapid activation of protein kinase C in isolated rat liver nuclei by prolactin, a known hepatic mitogen.

  1988cited by this paper
• Immunomodulation by bromocriptine.

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• Prolactin and autoimmunity.

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• Prolactin regulates antitumor immune response through induction of tumoricidal macrophages and release of IL‐12

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• Multiple internalization motifs differentially used by prolactin receptor isoforms mediate similar endocytic pathways.

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• Prolactin influences proliferation and apoptosis of a human IgE secreting myeloma cell line, U266.

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• In Vitro Effects of Prolactin on the Lympho-Hemopoietic System

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