Inducible rodent models of acquired podocyte diseases.

No abstract is available for this paper.

• Publication year

  2009

• Venue

  AJP - Renal Physiology

• Publication date

  2009-02-01

• Fields of study

  Biology, Medicine

• Identifiers
  DOI 10.1152/ajprenal.90421.2008PMID 18784259
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

• Podocytes contribute to the formation of glomerular crescents.

  2008cited by this paper
• Immunohistochemical analyses on serum proteins in nephrons of protein-overload mice by "in vivo cryotechnique".

  2007cited by this paper
• Induction of Passive Heymann Nephritis in Complement Component 6-Deficient PVG Rats1

  2007cited by this paper
• Podocytes in culture: past, present, and future.

  2007cited by this paper
• Darbepoetin alfa protects podocytes from apoptosis in vitro and in vivo.

  2007cited by this paper
• Target antigens and nephritogenic antibodies in membranous nephropathy: of rats and men

  2007cited by this paper
• Effects of Rapamycin on Active Heymann Nephritis

  2007cited by this paper
• Podocyte Damage Resulting in Podocyturia: A Potential Diagnostic Marker to Assess Glomerular Disease Activity

  2007cited by this paper
• The alternative pathway of complement is activated in the glomeruli and tubulointerstitium of mice with adriamycin nephropathy.

  2007cited by this paper
• Minimal change nephropathy and focal segmental glomerulosclerosis

  2007cited by this paper
• Differential Regulation of Toll-Like Receptor 4 Gene Expression in Renal Cells by Angiotensin II: Dependency on AP1 and PU.1 Transcriptional Sites

  2007cited by this paper
• Actin up: regulation of podocyte structure and function by components of the actin cytoskeleton.

  2007cited by this paper
• Disruption of glomerular basement membrane charge through podocyte-specific mutation of agrin does not alter glomerular permselectivity.

  2007cited by this paper
• In mice, proteinuria and renal inflammatory responses to albumin overload are strain-dependent.

  2006cited by this paper
• Advances in transgenic rat production

  2006cited by this paper
• Complement Activation Contributes to Both Glomerular and Tubulointerstitial Damage in Adriamycin Nephropathy in Mice1

  2006cited by this paper
• Differential capacity of anti‐RAP and anti‐megalin antibodies to produce progressive passive Heymann nephritis — implications for the pathogenesis of idiopathic human membranous glomerulonephritis

  2006cited by this paper
• An ancestral haplotype defines susceptibility to doxorubicin nephropathy in the laboratory mouse.

  2006cited by this paper
• Puromycin aminonucleoside induces oxidant-dependent DNA damage in podocytes in vitro and in vivo.

  2006cited by this paper
• Protective effect of lycopene on adriamycin-induced cardiotoxicity and nephrotoxicity.

  2006cited by this paper
• The podocyte's response to injury: role in proteinuria and glomerulosclerosis.

  2006cited by this paper
• Tight junction biology and kidney dysfunction.

  2006cited by this paper
• Induction of glomerular heparanase expression in rats with adriamycin nephropathy is regulated by reactive oxygen species and the renin-angiotensin system.

  2006cited by this paper
• Protective effects of blocking renin-angiotensin system on the progression of renal injury in glomerulosclerosis.

  2005cited by this paper
• Synaptopodin regulates the actin-bundling activity of alpha-actinin in an isoform-specific manner.

  2005cited by this paper
• DNA vaccination with naked DNA encoding MCP-1 and RANTES protects against renal injury in adriamycin nephropathy.

  2005cited by this paper
• Nephrin and podocin dissociate at the onset of proteinuria in experimental membranous nephropathy.

  2005cited by this paper
• The slit diaphragm: a signaling platform to regulate podocyte function

  2005cited by this paper
• Experimental membranous nephropathy redux.

  2005cited by this paper
• Podocyte depletion causes glomerulosclerosis: diphtheria toxin-induced podocyte depletion in rats expressing human diphtheria toxin receptor transgene.

  2005cited by this paper
• Cdc2 and Cdk2 play critical roles in low dose doxorubicin-induced cell death through mitotic catastrophe but not in high dose doxorubicin-induced apoptosis.

  2005cited by this paper
• Nephrin and podocin expression around the onset of puromycin aminonucleoside nephrosis.

  2005cited by this paper
• A Mendelian locus on chromosome 16 determines susceptibility to doxorubicin nephropathy in the mouse.

  2005influential reference
• Cellular response to injury in membranous nephropathy.

  2005cited by this paper
• Alterations of renal phenotype and gene expression profiles due to protein overload in NOD-related mouse strains

  2005cited by this paper
• Induction of B7-1 in podocytes is associated with nephrotic syndrome.

  2004influential reference
• Podocytes populate cellular crescents in a murine model of inflammatory glomerulonephritis.

  2004cited by this paper
• Age-related differences in Adriamycin-induced nephropathy

  2004cited by this paper
• Mitochondrial DNA and its respiratory chain products are defective in doxorubicin nephrosis.

  2004cited by this paper
• CCR1 blockade reduces interstitial inflammation and fibrosis in mice with glomerulosclerosis and nephrotic syndrome.

  2004cited by this paper
• Key molecular events in puromycin aminonucleoside nephrosis rats

  2004cited by this paper
• Glomerular Deposition and Urinary Excretion of Complement Factor H in Idiopathic Membranous Nephropathy

  2004cited by this paper
• Mouse model of membranous nephropathy induced by cationic bovine serum albumin: antigen dose-response relations and strain differences.

  2004cited by this paper
• A podocentric view of nephrology

  2004cited by this paper
• Caspase inhibition switches doxorubicin-induced apoptosis to senescence

  2003cited by this paper
• Populate Cellular Crescents in a Murine Model of Inflammatory Glomerulonephritis

  2003cited by this paper
• Protein overload induces fractalkine upregulation in proximal tubular cells through nuclear factor kappaB- and p38 mitogen-activated protein kinase-dependent pathways.

  2003cited by this paper
• Complement mediates nephrin redistribution and actin dissociation in experimental membranous nephropathy.

  2003cited by this paper
• Podocytes that detach in experimental membranous nephropathy are viable.

  2003cited by this paper
• Hypercholesterolemia is a prerequisite for puromycin inducible damage in mouse kidney.

  2003influential reference
• Cloning of rat homologue of podocin: expression in proteinuric states and in developing glomeruli.

  2003cited by this paper
• C5b-9 and adhesion molecules in human idiopathic membranous nephropathy.

  2002cited by this paper
• Antenatal membranous glomerulonephritis due to anti-neutral endopeptidase antibodies.

  2002cited by this paper
• Altered antioxidant defence in a mouse adriamycin model of glomerulosclerosis.

  2001cited by this paper
• Role of CD8(+) cells in the progression of murine adriamycin nephropathy.

  2001cited by this paper
• Glomerular complement regulation is overwhelmed in passive Heymann nephritis.

  2001cited by this paper
• Renal tubulointerstitial damage caused by persistent proteinuria is attenuated in AT1-deficient mice: role of endothelin-1.

  2001cited by this paper
• Clinical Investigations in Critical CarePredictive Value of Microalbuminuria in Medical ICU Patients: Results of a Pilot Study

  2001cited by this paper
• Depletion of CD4(+) T cells aggravates glomerular and interstitial injury in murine adriamycin nephropathy.

  2001cited by this paper
• Angiotensin-converting enzyme inhibition prevents glomerular-tubule disconnection and atrophy in passive Heymann nephritis, an effect not observed with a calcium antagonist.

  2001cited by this paper
• Podocyte depletion and glomerulosclerosis have a direct relationship in the PAN-treated rat.

  2001cited by this paper
• Rapid and diverse changes of gene expression in the kidneys of protein-overload proteinuria mice detected by microarray analysis.

  2001cited by this paper
• Repulsive charge mechanism serve to maintain lumens and cavities. An histochemical study of rat serosa and kidney.

  2001cited by this paper
• The role of protein traffic in the progression of renal diseases.

  2000cited by this paper
• Interstitial fibrosis in mice with overload proteinuria: deficiency of TIMP-1 is not protective.

  2000cited by this paper
• Cloning of rat nephrin: expression in developing glomeruli and in proteinuric states.

  2000cited by this paper
• Progressive adriamycin nephropathy in mice: sequence of histologic and immunohistochemical events.

  2000influential reference
• Nephrin in experimental glomerular disease.

  2000cited by this paper
• Glomerulosclerosis in Adriamycin-induced nephrosis is accelerated by a lipid-rich diet

  2000cited by this paper
• Pregnancy aggravates proteinuria in subclinical glomerulonephritis in the rat.

  1999cited by this paper
• Renal protective effects of blocking the intrarenal renin-angiotensin system.

  1999cited by this paper
• Podoplanin, a novel 43-kDa membrane protein, controls the shape of podocytes.

  1999cited by this paper
• Effect of flosulide, a selective cyclooxygenase 2 inhibitor, on passive heymann nephritis in the rat.

  1999cited by this paper
• Systemic factors are involved in the pathogenesis of proteinuria-induced glomerulosclerosis in adriamycin nephrotic rats.

  1999cited by this paper
• A critical evaluation of the mechanisms of action proposed for the antitumor effects of the anthracycline antibiotics adriamycin and daunorubicin.

  1999cited by this paper
• Renoprotective effect of contemporary blocking of angiotensin II and endothelin-1 in rats with membranous nephropathy.

  1998cited by this paper
• Angiotensin II and endothelin in chronic glomerulonephritis.

  1998cited by this paper
• Cell Proliferation and Apoptosis of the Glomerular Epithelial Cells in Rats with Puromycin Aminonucleoside Nephrosis

  1998cited by this paper
• Role of intrinsic renal cells versus infiltrating cells in glomerular crescent formation.

  1998influential reference
• Experimental Focal Segmental Glomerulosclerosis in Mice

  1998cited by this paper
• Tissue factor in crescentic glomerulonephritis.

  1997cited by this paper
• Podoplanin, novel 43-kd membrane protein of glomerular epithelial cells, is down-regulated in puromycin nephrosis.

  1997cited by this paper
• Proteoglycan expression in the normal rat kidney.

  1997cited by this paper
• Tissue factor in crescentic glomerulonephritis.

  1997cited by this paper
• Podocyte alpha-actinin induction precedes foot process effacement in experimental nephrotic syndrome.

  1997cited by this paper
• Understanding the nature of renal disease progression.

  1997cited by this paper
• Effect of DP-1904, a thromboxane A2 synthase inhibitor, on passive Heymann nephritis in rats.

  1996cited by this paper
• Sex difference in progression of adriamycin-induced nephropathy in rats.

  1996cited by this paper
• Beneficial effects of systemic immunoglobulin in experimental membranous nephropathy.

  1996influential reference
• Re-evaluation of foot process effacement in acute puromycin aminonucleoside nephrosis.

  1996cited by this paper
• The Heymann nephritis antigenic complex: megalin (gp330) and RAP.

  1995cited by this paper
• Induction of proteinuria by adriamycin or bovine serum albumin in the mouse.

  1995cited by this paper
• Antibodies to glycolipids activate complement and promote proteinuria in passive Heymann nephritis.

  1994cited by this paper
• Modulation of cytoskeletal organization of podocytes during the course of aminonucleoside nephrosis in rats

  1994cited by this paper
• Glomerular size selectivity during protein overload in the rat.

  1993influential reference
• Podocytic cytoskeletal disaggregation and basement-membrane detachment in puromycin aminonucleoside nephrosis.

  1993cited by this paper
• Reactive oxygen species and neutrophil respiratory burst cytochrome b558 are produced by kidney glomerular cells in passive Heymann nephritis.

  1993cited by this paper
• The altered glomerular filtration slits seen in puromycin aminonucleoside nephrosis and protamine sulfate-treated rats contain the tight junction protein ZO-1.

  1992cited by this paper
• Amelioration of glomerular injury in doxorubicin hydrochloride nephrosis by dimethylthiourea.

  1991cited by this paper
• Depletion of C6 prevents development of proteinuria in experimental membranous nephropathy in rats.

  1989influential reference

• Endothelial cell-released CD93 contributes to podocyte injury in idiopathic nephrotic syndrome.

  2026cites this paper
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  2026cites this paper
• Voclosporin ameliorates proteinuria in a model of non-inflammatory glomerular disease.

  2026cites this paper
• Framework for precision medicine in focal segmental glomerulosclerosis: Translation of sparsentan-responsive genes in a rat model to kidney disease associated proteins in biofluids

  2025cites this paper
• T2‐Weighted Imaging and Intravoxel Incoherent Motion for Monitoring Glomerulonephritis

  2025cites this paper
• RNA Alternative Splicing and Polyadenylation and Regulation of the Glomerular Filtration Barrier

  2025cites this paper
• Effects of Prothrombin on Podocytopathy and Proteinuria in Glomerular Disease.

  2025cites this paper
• The Life of a Kidney Podocyte

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• The role of B lymphocyte subsets in nephrotic syndrome: functions, mechanisms, clinical significance and future perspectives

  2025cites this paper
• Model Systems of Chronic Kidney Disease: A Detailed Overview and Recent Advances

  2025cites this paper
• Revisiting nephrin signaling and its specialized effects on the uniquely adaptable podocyte

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• Hypoalbuminemia Increases Fibrin Clot Density and Impairs Fibrinolysis

  2025cites this paper
• Fbxo11 maintains mitochondrial function and prevents podocyte injury in adriamycin-induced nephropathy by mediating the ubiquitin degradation of Fosl2.

  2024cites this paper
• Subsequent Transition from Minimal Change Disease to Secondary form of Focal Segmental Glomerulosclerosis: Not a Sampling Error yet Still Amenable to Immunosuppressive Therapy

  2024cites this paper
• Role of the Ste20‐like kinase SLK in podocyte adhesion

  2024influential citation
• Pharmacological investigation of indole alkaloids from Alstonia scholaris against chronic glomerulonephritis.

  2023cites this paper
• Rodent models to study type 1 and type 2 diabetes induced human diabetic nephropathy

  2023cites this paper
• Podocyte-specific deletion of ubiquitin carboxyl-terminal hydrolase L1 causes podocyte injury by inducing endoplasmic reticulum stress

  2023cites this paper
• Association of serum thymosin β4 with malnutrition-inflammation-atherosclerosis syndrome in peritoneal dialysis patients: a cross-sectional study

  2023cites this paper
• Prothrombin Knockdown Protects Podocytes and Reduces Proteinuria in Glomerular Disease

  2023cites this paper
• Podocyte Injury at Young Age Causes Premature Senescence and Worsens Glomerular Aging.

  2023cites this paper
• Capsazepine (CPZ) Inhibits TRPC6 Conductance and Is Protective in Adriamycin-Induced Nephropathy and Diabetic Glomerulopathy

  2023cites this paper
• The Pathophysiological Role of Thymosin β4 in the Kidney Glomerulus

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• Macrophages polarization in renal inflammation and fibrosis animal models (Review)

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• Prothrombin Knockdown Protects Podocytes and Reduces Proteinuria in Glomerular Disease Running Headline: Prothrombin Modulates Podocyte Health

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  2023influential citation
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  2022cites this paper
• Transition from minimal change disease to focal segmental glomerulosclerosis related to occupational exposure: A case report

  2022cites this paper
• Role of detached podocytes in differentiating between minimal change disease and early focal segmental glomerulosclerosis, can we rely on routine light microscopy?

  2022cites this paper
• Complement induces podocyte pyroptosis in membranous nephropathy by mediating mitochondrial dysfunction

  2022cites this paper
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  2022cites this paper
• The Effects of TRPC6 Knockout in Animal Models of Kidney Disease

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• Ion channels and channelopathies in glomeruli.

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• Complement induces podocyte pyroptosis in membranous nephropathy by mediating mitochondrial dysfunction

  2022cites this paper
• Gain-of-function, focal segmental glomerulosclerosis Trpc6 mutation minimally affects susceptibility to renal injury in several mouse models

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  2022cites this paper
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