Splanchnic ischaemia/reperfusion and multiple organ failure.

No abstract is available for this paper.

• Publication year

  1996

• Venue

  British Journal of Anaesthesia

• Publication date

  1996-07-01

• Fields of study

  Medicine

• Identifiers
  DOI 10.1093/BJA/77.1.59PMID 8703631
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

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  1996cited by this paper
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  1995cited by this paper
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  1995cited by this paper
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  1995cited by this paper
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  1995cited by this paper
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  1995cited by this paper
• Efficacy of intraportal injection of anti-ICAM-1 monoclonal antibody against liver cell injury following warm ischemia in the rat.

  1995cited by this paper
• Reperfused gut elaborates PAF that chemoattracts and primes neutrophils.

  1995cited by this paper
• Gut phospholipase A2 mediates neutrophil priming and lung injury after mesenteric ischemia-reperfusion.

  1995cited by this paper
• Tumor necrosis factor-alpha and interleukin-6 selectively regulate neutrophil function in vitro.

  1995cited by this paper
• Gastric mucosal pH and oxygen delivery and oxygen consumption indices in the assessment of adequacy of resuscitation after trauma: a prospective, randomized study.

  1995cited by this paper
• CD11b blockade prevents lung injury despite neutrophil priming after gut ischemia/reperfusion.

  1995cited by this paper
• Increasing splanchnic blood flow in the critically III.

  1995cited by this paper
• Leukocytes contribute to hepatic ischemia/reperfusion injury via intercellular adhesion molecule-1-mediated venular adherence.

  1995cited by this paper
• Inhaled nitric oxide variably improves oxygenation and pulmonary hypertension in patients with acute respiratory distress syndrome.

  1995cited by this paper
• Phospholipase A2 regulates critical inflammatory mediators of multiple organ failure.

  1994cited by this paper
• Contribution of intercellular adhesion molecule 1 (ICAM‐1) to the pathogenesis of splanchnic artery occlusion shock in the rat

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• Leukocyte-endothelial adhesion molecules.

  1994cited by this paper
• Enhanced attachment and elastase-releasing capacity of neutrophils after surgery.

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• Temporal patterns of hemodynamics, oxygen transport, cytokine activity, and complement activity in the development of adult respiratory distress syndrome after severe injury.

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• EVIDENCE FAVORING THE ROLE OF THE GUT AS A CYTOKINE‐GENERATING ORGAN IN RATS SUBJECTED TO HEMORRHAGIC SHOCK

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• Nitric oxide (endothelium-derived relaxing factor) attenuates revascularization-induced lung injury.

  1994cited by this paper
• Effects of antimurine interleukin-6 on bacterial translocation during gut-derived sepsis.

  1994cited by this paper
• Polymorphonuclear leukocyte dysregulation during the systemic inflammatory response syndrome.

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• Immune function in patients undergoing open vs laparoscopic cholecystectomy.

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• Interleukin-6 potentiates neutrophil priming with platelet-activating factor.

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• Oxygen radicals inhibit human plasma acetylhydrolase, the enzyme that catabolizes platelet-activating factor.

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• Blunt injury augments interleukin-6 but not tumor necrosis factor in isolated, perfused rat hindlimbs.

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• Early predictors of postinjury multiple organ failure.

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• Clinical benefits of an immune-enhancing diet for early postinjury enteral feeding.

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• Circulating interleukin‐8 concentrations in patients with multiple organ failure of septic and nonseptic origin

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• Nitric oxide production is inhibited in trauma patients.

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• Translocation. Incidental phenomenon or true pathology?

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• Cytokine Patterns in Patients After Major Vascular Surgery, Hemorrhagic Shock, and Severe Blunt Trauma Relation with Subsequent Adult Respiratory Distress Syndrome and Multiple Organ Failure

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• Assessment of splanchnic oxygenation by gastric tonometry in patients with acute circulatory failure.

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• The Gastrointestinal Tract: The Canary of the Body?

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• Epidemiology of trauma deaths: a reassessment.

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• Free radicals in medicine

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• Lung injury from gut ischemia: insensitivity to portal blood flow diversion.

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• Gut ischemia mediates lung injury by a xanthine oxidase-dependent neutrophil mechanism.

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• Elevation of cytokines during open heart surgery with cardiopulmonary bypass: participation of interleukin 8 and 6 in reperfusion injury

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• Portal endotoxin and cytokine responses during abdominal aortic surgery.

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• Ischemia-reperfusion in feline small intestine: a role for nitric oxide.

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• Plasma Cytokines After Thermal Injury and Their Relationship to Infection

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• Interleukin-6 mediates host defense responses induced by abdominal surgery.

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• Platelet-activating factor-induced polymorphonuclear neutrophil priming independent of CD11b adhesion.

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• Increased PMN adherence on endothelial cells after hypoxia: involvement of PAF, CD18/CD11b, and ICAM-1.

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• Plasma cytokines after thermal injury and their relationship to infection.

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• Pneumonia: cause or symptom of postinjury multiple organ failure?

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• Effects of accidental trauma on cytokine and endotoxin production.

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• Gut ischemia/reperfusion produces lung injury independent of endotoxin

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• IL-6 increases endothelial permeability in vitro.

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• The horror autotoxicus and multiple-organ failure.

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• Endotoxin after gut ischemia/reperfusion causes irreversible lung injury.

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• Selective gut decontamination reduces nosocomial infections and length of stay but not mortality or organ failure in surgical intensive care unit patients.

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• Anoxia/reoxygenation-induced neutrophil adherence to cultured endothelial cells.

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• Systemic cytokine response after major surgery

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• Cytokine serum level during severe sepsis in human IL-6 as a marker of severity.

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• Neutrophil (PMN) phagocytosis and chemotaxis after reperfusion injury.

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• Reperfusion injury.

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• The effects of ischemia and ischemia-reperfusion on bacterial translocation, lipid peroxidation, and gut histology: studies on hemorrhagic shock in pigs.

  1992cited by this paper
• Pathophysiology of mesenteric ischemia.

  1992cited by this paper
• Possible induction of intercellular adhesion molecule-1 (ICAM-1) expression on endothelial cells by platelet-activating factor (PAF).

  1992cited by this paper
• Hypoxia induces endothelial cells to increase their adherence for neutrophils: role of PAF.

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• Simultaneous liver and lung injury following gut ischemia is mediated by xanthine oxidase.

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• Definitions for sepsis and organ failure and guidelines for the use of innovative therapies in sepsis. The ACCP/SCCM Consensus Conference Committee. American College of Chest Physicians/Society of Critical Care Medicine.

  1992cited by this paper
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  1992cited by this paper
• Liver injury is a reversible neutrophil-mediated event following gut ischemia.

  1992cited by this paper
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  1992cited by this paper
• Incommensurate oxygen consumption in response to maximal oxygen availability predicts postinjury multiple organ failure.

  1991cited by this paper
• Does selective decontamination of the gastrointestinal tract prevent multiple organ failure? An experimental study.

  1991cited by this paper
• Bacterial translocation in trauma patients.

  1991cited by this paper
• Gastric mucosal pH as a prognostic index of mortality in critically ill patients

  1991cited by this paper
• Leukocyte activation in ischemia-reperfusion injury of skeletal muscle.

  1991cited by this paper
• Selective and early increase of IL‐1 inhibitors, IL‐6 and cortisol after elective surgery

  1991cited by this paper
• Hypovolemic shock promotes neutrophil sequestration in lungs by a xanthine oxidase-related mechanism.

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• Gut bacterial translocation via the portal vein: a clinical perspective with major torso trauma.

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• Ischemia activates neutrophils but inhibits their local and remote diapedesis.

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• Leukocyte adherence to venular endothelium during ischemia-reperfusion.

  1989cited by this paper
• Multiple systems organ failure: epidemiology and prognosis.

  1989cited by this paper
• Hypoxic reperfusion attenuates postischemic microvascular injury.

  1989cited by this paper
• Superoxide mediates reperfusion-induced leukocyte-endothelial cell interactions.

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• Evidence for neutrophil-related acute lung injury after intestinal ischemia-reperfusion.

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