New insights into mechanisms of immune glomerular injury.

No abstract is available for this paper.

• Publication year

  1994

• Venue

  Western Journal of Medicine

• Publication date

  Unknown publication date

• Fields of study

  Biology, Medicine

• Identifiers
  PMID 8048228PMCID PMC1022488
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

• Leukocyte adhesion molecules and kidney diseases.

  1994cited by this paper
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• Pathogenesis of glomerulonephritis.

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• Antimyeloperoxidase-associated proliferative glomerulonephritis: an animal model

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• Factors involved in the regulation of mesangial cell proliferation in vitro and in vivo.

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• Role of platelet-derived growth factor in glomerular disease.

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• Role of leukocyte-endothelial cell adhesion molecules in renal inflammation: in vitro and in vivo studies.

  1993cited by this paper
• Cytokines, mesangial cell activation and glomerular injury.

  1993cited by this paper
• Mediation of immune glomerular injury

  1993cited by this paper
• Mechanisms of proteinuria in noninflammatory glomerular diseases.

  1993cited by this paper
• Growth factors in glomerulonephritis.

  1993cited by this paper
• Infusion of platelet-derived growth factor or basic fibroblast growth factor induces selective glomerular mesangial cell proliferation and matrix accumulation in rats.

  1993cited by this paper
• Reactive oxygen species and neutrophil respiratory burst cytochrome b558 are produced by kidney glomerular cells in passive Heymann nephritis.

  1993cited by this paper
• Rat glomerular mesangial cells synthesize basic fibroblast growth factor. Release, upregulated synthesis, and mitogenicity in mesangial proliferative glomerulonephritis.

  1992cited by this paper
• T cells and macrophages and their role in renal injury.

  1992cited by this paper
• Antagonists of transforming growth factor-beta: a novel approach to treatment of glomerulonephritis and prevention of glomerulosclerosis.

  1992cited by this paper
• Platelet-derived growth factor: a potentially important cytokine in glomerular disease.

  1992cited by this paper
• Cell-matrix adhesion receptors: relevance to glomerular pathology.

  1992cited by this paper
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  1992cited by this paper
• Glomerular cells, extracellular matrix accumulation, and the development of glomerulosclerosis in the remnant kidney model.

  1992cited by this paper
• Inhibition of mesangial cell proliferation and matrix expansion in glomerulonephritis in the rat by antibody to platelet-derived growth factor

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• Glomerular epithelial cells secrete a glomerular basement membrane-degrading metalloproteinase.

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• Role of C5b-9 in experimental membranous nephropathy.

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• Altered glomerular extracellular matrix synthesis in experimental membranous nephropathy.

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• Structural characterization of the mesangial cell type IV collagenase and enhanced expression in a model of immune complex-mediated glomerulonephritis.

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• Integrin receptors in the glomerulus: potential role in glomerular injury.

  1992cited by this paper
• The extracellular glycoprotein SPARC interacts with platelet-derived growth factor (PDGF)-AB and -BB and inhibits the binding of PDGF to its receptors.

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• Expression of smooth muscle cell phenotype by rat mesangial cells in immune complex nephritis. Alpha-smooth muscle actin is a marker of mesangial cell proliferation.

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• Platelets in inflammatory glomerular injury.

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• Platelet-complement interactions in mesangial proliferative nephritis in the rat.

  1991cited by this paper
• Current status of the structural and functional basis of glomerular filtration and proteinuria.

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• Platelet-derived growth factor expression in mesangial proliferative glomerulonephritis.

  1991cited by this paper
• Glomerular Injury Induced by the C5b-9 Membrane Attack Complex of Complement

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• Mitogenic effect of platelet‐derived growth factor in human glomerular mesangial cells: modulation and/or suppression by inflammatory cytokines

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• Resident glomerular cells in glomerular injury: mesangial cells.

  1991cited by this paper
• Demonstration of PDGF B-chain mRNA in glomeruli in mesangial proliferative nephritis by in situ hybridization.

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• Platelets mediate glomerular cell proliferation in immune complex nephritis induced by anti-mesangial cell antibodies in the rat.

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• Cytosolic calcium and protein kinase C reduce complement-mediated glomerular epithelial injury.

  1990cited by this paper
• Gelatinase secretion by glomerular epithelial cells.

  1990cited by this paper
• Mediation of immune glomerular injury.

  1990cited by this paper
• The pathogenesis of membranous glomerulonephritis from morphology to molecules

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• Mechanisms and kinetics for platelet and neutrophil localization in immune complex nephritis.

  1989cited by this paper
• Role of reactive oxygen metabolites in experimental glomerular disease

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• Depletion of C6 prevents development of proteinuria in experimental membranous nephropathy in rats.

  1989cited by this paper
• Role of reactive oxygen metabolites in experimental glomerular disease.

  1989cited by this paper
• Complement C5b-9 complex activates phospholipases in glomerular epithelial cells.

  1989cited by this paper
• Transcellular transport and membrane insertion of the C5b-9 membrane attack complex of complement by glomerular epithelial cells in experimental membranous nephropathy.

  1989cited by this paper
• Induction of proteinuria in the rat by a monoclonal antibody against SGP-115/107.

  1988cited by this paper
• Epitope specific induction of proteinuria by monoclonal antibodies.

  1988cited by this paper
• Morphologic correlates of glomerular oxidant injury induced by the myeloperoxidase-hydrogen peroxide-halide system of the neutrophil.

  1988cited by this paper
• Pathogenesis of membranous nephropathy.

  1988cited by this paper
• The human neutrophil serine proteinases, elastase and cathepsin G, can mediate glomerular injury in vivo

  1988cited by this paper
• Evidence suggesting a role for hydroxyl radical in passive Heymann nephritis in rats.

  1988cited by this paper
• Anti-Fx1A produces complement-dependent cytotoxicity of glomerular epithelial cells.

  1988cited by this paper
• Transfer of experimental glomerulonephritis in chickens by mononuclear cells.

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• Platelets mediate neutrophil-dependent immune complex nephritis in the rat.

  1988cited by this paper
• Massive proteinuria induced in rats by a single intravenous injection of a monoclonal antibody.

  1988cited by this paper
• Secretory products of macrophages.

  1987cited by this paper
• New mechanism for glomerular injury. Myeloperoxidase-hydrogen peroxide-halide system.

  1987cited by this paper
• Quantitation and characterization of glomerular procoagulant activity in experimental glomerulonephritis.

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• Participation of the myeloperoxidase-H2O2-halide system in immune complex nephritis.

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• The membrane attack complex in complement-mediated glomerular epithelial cell injury: formation and stability of C5b-9 and C5b-7 in rat membranous nephropathy.

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• Complement membrane attack complex stimulates production of reactive oxygen metabolites by cultured rat mesangial cells.

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• The participation of macrophages, glomerular procoagulant activity, and factor VIII in glomerular fibrin deposition. Studies on anti-GBM antibody-induced glomerulonephritis in rabbits.

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• Direct antiGBM antibody induced alterations in glomerular permselectivity.

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• Influence of antigen distribution on the mediation of immunological glomerular injury.

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• Macrophage-induced glomerular fibrin deposition in experimental glomerulonephritis in the rabbit.

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• Mechanisms of glomerular injury in immune-complex disease.

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