[Anesthetics and nitric oxide. Interactions and their importance for anesthesia].

No abstract is available for this paper.

• Publication year

  1997

• Venue

  Der Anaesthesist

• Publication date

  Unknown publication date

• Fields of study

  Medicine

• Identifiers
  PMID 9382204
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

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• Isoflurane Anesthesia Attenuates Endothelium‐dependent Pulmonary Vasorelaxation by Inhibiting the Synergistic Interaction between Nitric Oxide and Prostacyclin

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• Etomidate and Thiopental Inhibit the Release of Endothelium‐derived Hyperpolarizing Factor in the Human Renal Artery

  1996cited by this paper
• Ketamine inhibits nitric oxide production in mouse-activated macrophage-like cells.

  1996cited by this paper
• Brain Nitric Oxide Synthase Activity Is Decreased by Intravenous Anesthetics

  1996cited by this paper
• Nitrict Oxide Synthase Inhibitors, 7-Nitro Indazole and Nitro sup G -L-Arginine Methyl Ester, Dose Dependently Reduce the Threshold for Isoflurane Anesthesia

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• Cardiac and Regional Hemodynamic Interactions between Halothane and Nitric Oxide Synthase Activity in Dogs

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• Vasomotor Responses of Rat Coronary Arteries to Isoflurane and Halothane Depend on Preexposure Tone and Vessel Size

  1995cited by this paper
• Oxygen-Derived Free Radicals Mediate Isoflurane-Induced Vasoconstriction of Rabbit Coronary Resistance Arteries

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• Inhalation Anesthetics Inhibit the Release of Endothelium-derived Hyperpolarizing Factor in the Rabbit Carotid Artery

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• Propofol-Associated Dilation of Rat Distal Coronary Arteries Is Mediated by Multiple Substances, Including Endothelium-Derived Nitric Oxide

  1995cited by this paper
• Volatile and intravenous anesthetics selectively attenuate the release of endothelium-derived hyperpolarizing factor elicited by bradykinin in the coronary microcirculation

  1995cited by this paper
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  1995cited by this paper
• S‐ETHYLISOTHIOUREA, A NONAMINO ACID INHIBITOR OF NITRIC OXIDE SYNTHASE, REVERSES SEPTIC VASODILATION IN SHEEP

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• Modification of Endothelium-Dependent Relaxation by Propofol, Ketamine, and Midazolam

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• Selective Anesthetic Inhibition of Brain Nitric Oxide Synthase

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• Physiological importance of nitric oxide.

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• Nitric oxide: physiology, pathophysiology, and pharmacology.

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• [Endothelial functions in cardiovascular diseases].

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