Functional interaction of nuclear factors EF-C, HNF-4, and RXR alpha with hepatitis B virus enhancer I

No abstract is available for this paper.

• Publication year

  1993

• Venue

  Journal of Virology

• Publication date

  1993-07-01

• Fields of study

  Biology, Medicine

• Identifiers
  DOI 10.1128/jvi.67.7.3940-3950.1993PMID 8389913PMCID PMC237761
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

• Differentiated liver cell specificity of the second enhancer of hepatitis B virus

  1993cited by this paper
• Multiplicity generates diversity in the retinoic acid signalling pathways.

  1992cited by this paper
• Antagonism between apolipoprotein AI regulatory protein 1, Ear3/COUP-TF, and hepatocyte nuclear factor 4 modulates apolipoprotein CIII gene expression in liver and intestinal cells

  1992cited by this paper
• Repression by ARP-1 sensitizes apolipoprotein AI gene responsiveness to RXR alpha and retinoic acid

  1992cited by this paper
• Retinoid X receptor RXR alpha binds to and trans-activates the hepatitis B virus enhancer.

  1992cited by this paper
• Retinoid X receptor is an auxiliary protein for thyroid hormone and retinoic acid receptors

  1992cited by this paper
• Homodimer formation of retinoid X receptor induced by 9-cis retinoic acid

  1992cited by this paper
• Transcriptional regulation of precore and pregenomic RNAs of hepatitis B virus

  1992cited by this paper
• Retinoid X receptor interacts with nuclear receptors in retinoic acid, thyroid hormone and vitamin D3 signalling

  1992cited by this paper
• Chicken ovalbumin upstream promoter transcription factor (COUP-TF) dimers bind to different GGTCA response elements, allowing COUP-TF to repress hormonal induction of the vitamin D3, thyroid hormone, and retinoic acid receptors

  1992cited by this paper
• Purification, cloning, and RXR identity of the HeLa cell factor with which RAR or TR heterodimerizes to bind target sequences efficiently.

  1992cited by this paper
• Convergence of 9-cis retinoic acid and peroxisome proliferator signalling pathways through heterodimer formation of their receptors

  1992cited by this paper
• Orphan receptors: in search of a unifying hypothesis for activation.

  1992cited by this paper
• 9-cis retinoic acid is a high affinity ligand for the retinoid X receptor.

  1992cited by this paper
• Retinoid X receptor-COUP-TF interactions modulate retinoic acid signaling.

  1992cited by this paper
• Different binding site requirements for binding and activation for the bipartite enhancer factor EF-1A.

  1992cited by this paper
• Characterization of three RXR genes that mediate the action of 9-cis retinoic acid.

  1992cited by this paper
• COUP orphan receptors are negative regulators of retinoic acid response pathways

  1992cited by this paper
• 9-Cis retinoic acid stereoisomer binds and activates the nuclear receptor RXRα

  1992cited by this paper
• The COUP-TFs compose a family of functionally related transcription factors.

  1991cited by this paper
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  1991cited by this paper
• Functional analysis of a liver-specific enhancer of the hepatitis B virus.

  1991cited by this paper
• Interaction of nuclear factor EF-1A with the polyomavirus enhancer region

  1991cited by this paper
• Characterization of the hepatitis B virus EnhI enhancer and X promoter complex

  1991cited by this paper
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  1991cited by this paper
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  1991cited by this paper
• A single element within the hepatitis B virus enhancer binds multiple proteins and responds to multiple stimuli

  1990cited by this paper
• Functional organization of the hepatitis B virus enhancer

  1990cited by this paper
• Nuclear receptor that identifies a novel retinoic acid response pathway

  1990cited by this paper
• Liver-enriched transcription factor HNF-4 is a novel member of the steroid hormone receptor superfamily.

  1990cited by this paper
• Characterization of an autoregulated response element in the mouse retinoic acid receptor type beta gene.

  1990cited by this paper
• A new enhancer element, ENII, identified in the X gene of hepatitis B virus

  1990cited by this paper
• The genome of hepatitis B virus contains a second enhancer: cooperation of two elements within this enhancer is required for its function

  1990cited by this paper
• Hierarchic and cooperative binding of the rat liver nuclear protein C/EBP at the hepatitis B virus enhancer

  1990cited by this paper
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  1989cited by this paper
• Binding of nuclear factor EF-C to a functional domain of the hepatitis B virus enhancer region

  1989cited by this paper
• COUP transcription factor is a member of the steroid receptor superfamily

  1989cited by this paper
• Cellular factors that interact with the hepatitis B virus enhancer

  1989cited by this paper
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  1989cited by this paper
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  1989cited by this paper
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  1989cited by this paper
• The SV40 enhancer contains two distinct levels of organization

  1988cited by this paper
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  1988cited by this paper
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  1988cited by this paper
• Cooperativity and hierarchical levels of functional organization in the SV40 enhancer.

  1988cited by this paper
• Properties of the human hepatitis B virus enhancer: position effects and cell-type nonspecificity

  1988cited by this paper
• A High-Efficiency HeLa Cell Nuclear Transcription Extract

  1988cited by this paper
• Purification and characterization of chicken ovalbumin upstream promoter transcription factor from HeLa cells.

  1987cited by this paper
• Broad specificity of the hepatitis B enhancer function.

  1987cited by this paper
• The molecular biology of the hepatitis B viruses.

  1987cited by this paper
• Multiple nuclear proteins in liver cells are bound to hepatitis B virus enhancer element and its upstream sequences.

  1987cited by this paper
• Identification of a promoter element located upstream from the hepatitis B virus X gene

  1987cited by this paper
• Interactions between a DNA-binding transcription factor (COUP) and a non-DNA binding factor (S300-II).

  1987cited by this paper
• Gene regulation by steroid hormones.

  1987cited by this paper
• The human hepatitis B virus enhancer requires trans-acting cellular factor(s) for activity

  1986cited by this paper
• Interaction of a nuclear factor with the polyomavirus enhancer region.

  1986cited by this paper
• Rapid and efficient site-specific mutagenesis without phenotypic selection.

  1985cited by this paper
• A human hepatitis B viral enhancer element.

  1985cited by this paper
• A novel expression selection approach allows precise mapping of the hepatitis B virus enhancer.

  1985cited by this paper
• Recombinant genomes which express chloramphenicol acetyltransferase in mammalian cells

  1982cited by this paper
• Controlled synthesis of HBsAg in a differentiated human liver carcinoma-derived cell line

  1979cited by this paper

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• Strategies to Inhibit Hepatitis B Virus Gene Transcription

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  2009cites this paper
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