Advanced and High-Throughput Method for Mitochondrial Bioenergetics Evaluation in Neurotrauma.

No abstract is available for this paper.

• Publication year

  2016

• Venue

  Methods in molecular biology

• Publication date

  Unknown publication date

• Fields of study

  Medicine

• Identifiers
  DOI 10.1007/978-1-4939-3816-2_32PMID 27604740
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

• Decreased mitochondrial bioenergetics and calcium buffering capacity in the basal ganglia correlates with motor deficits in a nonhuman primate model of aging.

  2015cited by this paper
• N-acetylcysteine amide confers neuroprotection, improves bioenergetics and behavioral outcome following TBI.

  2014cited by this paper
• N-acetylcysteine amide preserves mitochondrial bioenergetics and improves functional recovery following spinal trauma.

  2014cited by this paper
• A time course of NADPH-oxidase up-regulation and endothelial nitric oxide synthase activation in the hippocampus following neurotrauma.

  2014cited by this paper
• Phenelzine Mitochondrial Functional Preservation and Neuroprotection after Traumatic Brain Injury Related to Scavenging of the Lipid Peroxidation-Derived Aldehyde 4-Hydroxy-2-Nonenal

  2013cited by this paper
• Concentration dependent effect of calcium on brain mitochondrial bioenergetics and oxidative stress parameters

  2013cited by this paper
• nalysis of regional brain mitochondrial bioenergetics and susceptibility to itochondrial inhibition utilizing a microplate based system

  2011cited by this paper
• Post-Injury Administration of the Mitochondrial Permeability Transition Pore Inhibitor, NIM811, Is Neuroprotective and Improves Cognition after Traumatic Brain Injury in Rats

  2011cited by this paper
• High Throughput Microplate Respiratory Measurements Using Minimal Quantities Of Isolated Mitochondria

  2011cited by this paper
• Therapeutic Window Analysis of the Neuroprotective Effects of Cyclosporine A after Traumatic Brain Injury

  2011cited by this paper
• What is the mitochondrial permeability transition pore?

  2009cited by this paper
• Mitochondrial calcium in health and disease.

  2009cited by this paper
• The optimal dosage and window of opportunity to maintain mitochondrial homeostasis following traumatic brain injury using the uncoupler FCCP.

  2009cited by this paper
• Early Mitochondrial Dysfunction after Cortical Contusion Injury

  2009cited by this paper
• Proteomic identification of nitrated brain proteins in traumatic brain‐injured rats treated postinjury with gamma‐glutamylcysteine ethyl ester: Insights into the role of elevation of glutathione as a potential therapeutic strategy for traumatic brain injury

  2009cited by this paper
• Evolution of Post-Traumatic Neurodegeneration after Controlled Cortical Impact Traumatic Brain Injury in Mice and Rats as Assessed by the De Olmos Silver and Fluorojade Staining Methods

  2008cited by this paper
• Neuroprotective Effects of Tempol, a Catalytic Scavenger of Peroxynitrite-Derived Free Radicals, in a Mouse Traumatic Brain Injury Model

  2008cited by this paper
• Attenuation of acute mitochondrial dysfunction after traumatic brain injury in mice by NIM811, a non-immunosuppressive cyclosporin A analog.

  2008cited by this paper
• A Time Course of Contusion-Induced Oxidative Stress and Synaptic Proteins in Cortex in a Rat Model of TBI

  2008cited by this paper
• Peroxynitrite‐mediated oxidative damage to brain mitochondria: Protective effects of peroxynitrite scavengers

  2007cited by this paper
• Temporal relationship of peroxynitrite-induced oxidative damage, calpain-mediated cytoskeletal degradation and neurodegeneration after traumatic brain injury.

  2007cited by this paper
• Proteomic Identification of Oxidized Mitochondrial Proteins following Experimental Traumatic Brain Injury

  2007cited by this paper
• Temporal Characterization of Mitochondrial Bioenergetics after Spinal Cord Injury

  2007cited by this paper
• Post-Injury Administration of Mitochondrial Uncouplers Increases Tissue Sparing and Improves Behavioral Outcome following Traumatic Brain Injury in Rodents

  2007cited by this paper
• Time Course of Post-Traumatic Mitochondrial Oxidative Damage and Dysfunction in a Mouse Model of Focal Traumatic Brain Injury: Implications for Neuroprotective Therapy

  2006cited by this paper
• Relationship of calpain-mediated proteolysis to the expression of axonal and synaptic plasticity markers following traumatic brain injury in mice.

  2006cited by this paper
• Mitochondrial permeability transition in CNS trauma: Cause or effect of neuronal cell death?

  2005cited by this paper
• Mitochondrial damage and dysfunction in traumatic brain injury.

  2004cited by this paper
• The ketogenic diet increases mitochondrial uncoupling protein levels and activity

  2004cited by this paper
• Mitochondrial Uncoupling as a Therapeutic Target Following Neuronal Injury

  2004cited by this paper
• Mitochondrial uncoupling protein‐2 protects the immature brain from excitotoxic neuronal death

  2003cited by this paper
• Biochemistry 5th ed

  2002cited by this paper
• Dietary supplement creatine protects against traumatic brain injury

  2000cited by this paper
• Dose-response curve and optimal dosing regimen of cyclosporin A after traumatic brain injury in rats.

  2000cited by this paper
• Continuous infusion of cyclosporin A postinjury significantly ameliorates cortical damage following traumatic brain injury.

  2000cited by this paper
• Cyclosporin A attenuates acute mitochondrial dysfunction following traumatic brain injury.

  1999cited by this paper
• The mitochondrial permeability transition pore and its role in cell death.

  1999cited by this paper
• Cyclosporin A Significantly Ameliorates Cortical Damage Following Experimental Traumatic Brain Injury in Rodents

  1999cited by this paper
• Mitochondrial Dysfunction and Calcium Perturbation Induced by Traumatic Brain Injury

  1997cited by this paper
• The respiratory chain and oxidative phosphorylation.

  1956cited by this paper
• Polarographic measurement of oxygen tension in whole blood and tissues during total by-pass of the heart.

  1953cited by this paper
• Continuous recording of blood oxygen tensions by polarography.

  1953cited by this paper

• Increased Prolylcarboxypeptidase Expression Can Serve as a Biomarker of Senescence in Culture

  2024cites this paper
• Time-Course Evaluation of Brain Regional Mitochondrial Bioenergetics in a Pre-Clinical Model of Severe Penetrating Traumatic Brain Injury

  2021cites this paper
• Age- and Organ-Specific Differences in Mitochondrial Bioenergetics in Brown Norway Rats

  2020cites this paper
• Bioenergetic restoration and neuroprotection after therapeutic targeting of mitoNEET: New mechanism of pioglitazone following traumatic brain injury.

  2020influential citation
• Formoterol, a β2-adrenoreceptor agonist, induces mitochondrial biogenesis and promotes cognitive recovery after traumatic brain injury.

  2020cites this paper
• A method for assessing tissue respiration in anatomically defined brain regions

  2020cites this paper
• Temporal changes in inflammatory mitochondria-enriched microRNAs following traumatic brain injury and effects of miR-146a nanoparticle delivery

  2020cites this paper
• Developing Successful Neuroprotective Treatments for TBI: Translational Approaches, Novel Directions, Opportunities and Challenges

  2020cites this paper
• Acute Mitochondrial Impairment Underlies Prolonged Cellular Dysfunction after Repeated Mild Traumatic Brain Injuries

  2019cites this paper
• Comprehensive Profile of Acute Mitochondrial Dysfunction in a Preclinical Model of Severe Penetrating TBI

  2019cites this paper
• An in vivo study into the metabolic reprogramming of hepatocellular carcinoma

  2018cites this paper
• Mitochondrial uncoupling prodrug improves tissue sparing, cognitive outcome, and mitochondrial bioenergetics after traumatic brain injury in male mice

  2018cites this paper
• Combination Drug Therapy of Pioglitazone and D-cycloserine Attenuates Chronic Orofacial Neuropathic Pain and Anxiety by Improving Mitochondrial Function Following Trigeminal Nerve Injury

  2017cites this paper
• Targeting mitochondrial dysfunction in CNS injury using Methylene Blue; still a magic bullet?

  2017cites this paper